Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response. - Wikidata - awgldk - TriplyDB

Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

http://www.wikidata.org/entity/Q33432736

about

Abnormal PfEMP1/knob display on Plasmodium falciparum-infected erythrocytes containing hemoglobin variants: fresh insights into malaria pathogenesis and protection Malaria parasite type 4 equilibrative nucleoside transporters (ENT4) are purine transporters with distinct substrate specificity A Dormant Microbial Component in the Development of Preeclampsia Incidence of Severe Malaria Syndromes and Status of Immune Responses among Khat Chewer Malaria Patients in Ethiopia A surprising role for uric acid: the inflammatory malaria response.Dual effect of Plasmodium-infected erythrocytes on dendritic cell maturation.Dual engagement of the NLRP3 and AIM2 inflammasomes by plasmodium-derived hemozoin and DNA during malaria Analysis of innate defences against Plasmodium falciparum in immunodeficient mice.Neutralization of malaria glycosylphosphatidylinositol in vitro by serum IgG from malaria-exposed individuals.Inflammatory Flt3l is essential to mobilize dendritic cells and for T cell responses during Plasmodium infection.Plasma uric acid levels correlate with inflammation and disease severity in Malian children with Plasmodium falciparum malaria A potential role for plasma uric acid in the endothelial pathology of Plasmodium falciparum malaria.Plasmodium falciparum-derived uric acid precipitates induce maturation of dendritic cells.Xanthine oxidoreductase regulates macrophage IL1β secretion upon NLRP3 inflammasome activation.Evolutionary context for the association of γ-globin, serum uric acid, and hypertension in African Americans.Lipid metabolites of the phospholipase A2 pathway and inflammatory cytokines are associated with brain volume in paediatric cerebral malaria Inflammatory cytokine and humoral responses to Plasmodium falciparum glycosylphosphatidylinositols correlates with malaria immunity and pathogenesis.Hyperuricaemia in the Pacific: why the elevated serum urate levels?The immunological balance between host and parasite in malaria.Pure Hemozoin is inflammatory in vivo and activates the NALP3 inflammasome via release of uric acid Experimental cerebral malaria progresses independently of the Nlrp3 inflammasome.Longitudinal transition trajectory of gouty arthritis and its comorbidities: a population-based study.Atypical activation of dendritic cells by Plasmodium falciparum.The Emerging Role of Pattern Recognition Receptors in the Pathogenesis of Malaria.

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P2860

Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

http://www.wikidata.org/entity/Q33432736

description

2009 nî lūn-bûn

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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2009 թվականի ապրիլին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

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Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

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type

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Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

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Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

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Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

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Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

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JOURNAL.PONE.0005194

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Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.

@en

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Aleksandra Leliwa-Sytek

http://www.w3.org/2001/XMLSchema#string

Barbara Evans

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Diana van de Hoef

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James E Evans

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Jamie Marie Orengo

http://www.w3.org/2001/XMLSchema#string

Marian Nyako

http://www.w3.org/2001/XMLSchema#string

P2860

Human malarial disease: a consequence of inflammatory cytokine release

Interleukin-10 and the interleukin-10 receptor

The global distribution of clinical episodes of Plasmodium falciparum malaria

Gout-associated uric acid crystals activate the NALP3 inflammasome

Xanthine oxidoreductase is central to the evolution and function of the innate immune system

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity

Severe falciparum malaria. World Health Organization, Communicable Diseases Cluster

Molecular identification of a danger signal that alerts the immune system to dying cells

Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration.

Plasmodium-induced inflammation by uric acid

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10.1371/JOURNAL.PONE.0005194

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Plasmodium falciparum

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