The contribution of inositol 1,4,5-trisphosphate and ryanodine receptors to agonist-induced Ca(2+) signaling of airway smooth muscle cells.
about
Ca2+ oscillations, Ca2+ sensitization, and contraction activated by protein kinase C in small airway smooth muscle.Nitric oxide induces airway smooth muscle cell relaxation by decreasing the frequency of agonist-induced Ca2+ oscillations.Human airway contraction and formoterol-induced relaxation is determined by Ca2+ oscillations and Ca2+ sensitivity.Acetylcholine promotes Ca2+ and NO-oscillations in adipocytes implicating Ca2+→NO→cGMP→cADP-ribose→Ca2+ positive feedback loop--modulatory effects of norepinephrine and atrial natriuretic peptideActivation of store-operated calcium entry in airway smooth muscle cells: insight from a mathematical model.A deterministic model predicts the properties of stochastic calcium oscillations in airway smooth muscle cells.Interaction between endoplasmic/sarcoplasmic reticulum stress (ER/SR stress), mitochondrial signaling and Ca(2+) regulation in airway smooth muscle (ASM)Bitter tasting compounds dilate airways by inhibiting airway smooth muscle calcium oscillations and calcium sensitivity.Minimally invasive imaging method based on second harmonic generation and multiphoton excitation fluorescence in translational respiratory research.Exploring lung physiology in health and disease with lung slicesMitochondrial regulation of cytosolic Ca²⁺ signals in smooth muscle.Subplasma membrane Ca2+ signals.The role of caveolae in the pathophysiology of lung diseases.A mathematical analysis of agonist- and KCl-induced Ca(2+) oscillations in mouse airway smooth muscle cells.Silencing MaxiK activity in corporal smooth muscle cells initiates compensatory mechanisms to maintain calcium homeostasis.Agonist-evoked Ca(2+) wave progression requires Ca(2+) and IP(3)A multi-scale approach to airway hyperresponsiveness: from molecule to organ.At the cross-point of connexins, calcium, and ATP: blocking hemichannels inhibits vasoconstriction of rat small mesenteric arteries.Selective inhibition of histamine-evoked Ca2+ signals by compartmentalized cAMP in human bronchial airway smooth muscle cells.Hydrogen sulphide inhibits Ca2+ release through InsP3 receptors and relaxes airway smooth muscle.
P2860
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P2860
The contribution of inositol 1,4,5-trisphosphate and ryanodine receptors to agonist-induced Ca(2+) signaling of airway smooth muscle cells.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
The contribution of inositol 1 ...... of airway smooth muscle cells.
@ast
The contribution of inositol 1 ...... of airway smooth muscle cells.
@en
type
label
The contribution of inositol 1 ...... of airway smooth muscle cells.
@ast
The contribution of inositol 1 ...... of airway smooth muscle cells.
@en
prefLabel
The contribution of inositol 1 ...... of airway smooth muscle cells.
@ast
The contribution of inositol 1 ...... of airway smooth muscle cells.
@en
P2093
P2860
P1476
The contribution of inositol 1 ...... of airway smooth muscle cells.
@en
P2093
Martin Edelmann
Michael J Sanderson
P2860
P304
P356
10.1152/AJPLUNG.90559.2008
P577
2009-05-22T00:00:00Z