Nitric oxide induces airway smooth muscle cell relaxation by decreasing the frequency of agonist-induced Ca2+ oscillations.
about
Ca2+ oscillations, Ca2+ sensitization, and contraction activated by protein kinase C in small airway smooth muscle.Treatment of experimental asthma using a single small molecule with anti-inflammatory and BK channel-activating properties.β-Agonist-mediated relaxation of airway smooth muscle is protein kinase A-dependent.Local small airway epithelial injury induces global smooth muscle contraction and airway constrictionThe relaxant effect of Nigella sativa on smooth muscles, its possible mechanisms and clinical applicationsRelaxin regulates myofibroblast contractility and protects against lung fibrosisEmerging role of PKA/eNOS pathway in therapeutic angiogenesis for ischaemic tissue diseasesSoluble guanylate cyclase modulators blunt hyperoxia effects on calcium responses of developing human airway smooth muscle.Evidence that NO/cGMP/PKG signalling cascade mediates endothelium dependent inhibition of IP₃R mediated Ca²⁺ oscillations in myocytes and pericytes of ureteric microvascular network in situBronchodilatory effect of hydrogen sulfide in rat.Regulation of retinal angiogenesis by endothelial nitric oxide synthase signaling pathway.Ion channel regulation of intracellular calcium and airway smooth muscle function.Generation of nitric oxide gradients in microfluidic devices for cell culture using spatially controlled chemical reactions.Phosphodiesterase V inhibition reduces airway responsiveness, but does not improve the beneficial effect of deep inspiration.Hydraulic conductivity of endothelial cell-initiated arterial cocultures.Bitter tasting compounds dilate airways by inhibiting airway smooth muscle calcium oscillations and calcium sensitivity.Exploring lung physiology in health and disease with lung slicesPrecision cut lung slices as an efficient tool for in vitro lung physio-pharmacotoxicology studies.Augmentation of S-Nitrosoglutathione Controls Cigarette Smoke-Induced Inflammatory-Oxidative Stress and Chronic Obstructive Pulmonary Disease-Emphysema Pathogenesis by Restoring Cystic Fibrosis Transmembrane Conductance Regulator Function.Heparin responses in vascular smooth muscle cells involve cGMP-dependent protein kinase (PKG)A multi-scale approach to airway hyperresponsiveness: from molecule to organ.Role of acetylcholine and calcium ions in three vascular contraction models: Angiotensin II, phenylephrine and caffeine.Muscarinic drugs regulate the PKG-II-dependent phosphorylation of M3 muscarinic acetylcholine receptors at plasma membranes from airway smooth muscle.Effects of Hyperoxia on the Developing Airway and Pulmonary Vasculature.NMDA receptors mediate contractile responses in human airway smooth muscle cells.Hydrogen sulphide inhibits Ca2+ release through InsP3 receptors and relaxes airway smooth muscle.
P2860
Q30533330-9C17C894-DD95-42EA-8A0D-4714CBB2D8E8Q30557489-75E7F64B-A1E5-466F-A6E4-BA2CAC4EB06EQ34044729-17D0009E-A51E-4EB5-9089-F8A773E2B7D6Q34082614-FC6F47AB-C57C-481D-8F1D-028B2BEEE7F2Q35288613-AF48B8A0-5A97-42CD-8084-CFFD2A8702B6Q35679329-9A279D08-411E-4DC8-B611-F8D072EE3A98Q36042713-0898A068-F0B6-4088-8FD7-8E430ECB8A14Q36066461-0F04BEFC-41BF-402C-B5A0-82BA76A0A96DQ36306865-D3A63203-BFB6-4FF7-A9BC-D8CEFABC2044Q36652619-995F5A0C-9FB7-47B2-A08B-7FE39FAC7DE1Q37239797-BB26A0C7-71A0-4885-BB5B-BB65D762FE16Q37325179-3BA7CD18-2D53-420D-BB36-9EEC7B5FFC3DQ37337084-933F5583-89AF-4E66-A4E9-6767CEE4803CQ37466878-CF9D9E02-B1C7-4D4A-884C-C7EB3CDD53F1Q37637277-7EFA5545-7FA3-497B-9E5E-C6E604C99902Q37671289-06E1A507-E13C-4DBC-975D-1936CA0B25B2Q37877938-96A63EAF-4EC1-4293-AA84-0B31EA6EAD01Q38048927-A2173175-07FD-4606-A238-B209AB59EF87Q39071592-4ED78A77-B3B9-432E-8134-D35B6DCB8E3AQ41767067-3A29ED7B-166D-4594-8A17-C8B01AB740EDQ42135142-5EE457AF-E438-4F14-9C2E-775E4D3F3203Q42324607-27E3FF9C-2B06-444C-87FB-AB9FE121B2BBQ42478037-CEEF773A-6409-479F-BB42-4B3DB0D4B86CQ47431365-0B2A175B-3DB8-42E3-ADD9-69E4596E33B8Q51639133-39AB2020-EAFA-416D-98D3-FC7186D19307Q54719091-7AFBCB20-9BA0-4DED-920B-FC3389FCDDC7
P2860
Nitric oxide induces airway smooth muscle cell relaxation by decreasing the frequency of agonist-induced Ca2+ oscillations.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Nitric oxide induces airway sm ...... ist-induced Ca2+ oscillations.
@ast
Nitric oxide induces airway sm ...... ist-induced Ca2+ oscillations.
@en
type
label
Nitric oxide induces airway sm ...... ist-induced Ca2+ oscillations.
@ast
Nitric oxide induces airway sm ...... ist-induced Ca2+ oscillations.
@en
prefLabel
Nitric oxide induces airway sm ...... ist-induced Ca2+ oscillations.
@ast
Nitric oxide induces airway sm ...... ist-induced Ca2+ oscillations.
@en
P2860
P356
P1476
Nitric oxide induces airway sm ...... nist-induced Ca2+ oscillations
@en
P2093
Michael J Sanderson
P2860
P304
P356
10.1085/JGP.200910365
P577
2010-03-01T00:00:00Z