The pronounced Th17 profile in systemic sclerosis (SSc) together with intracellular expression of TGFbeta and IFNgamma distinguishes SSc phenotypes. - Wikidata - awgldk - TriplyDB

The pronounced Th17 profile in systemic sclerosis (SSc) together with intracellular expression of TGFbeta and IFNgamma distinguishes SSc phenotypes.

The pronounced Th17 profile in systemic sclerosis (SSc) together with intracellular expression of TGFbeta and IFNgamma distinguishes SSc phenotypes.

http://www.wikidata.org/entity/Q33470222

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Challenges and Current Efforts in the Development of Biomarkers for Chronic Inflammatory and Remodeling Conditions of the Lungs Spotlight on tocilizumab and its potential in the treatment of systemic sclerosis Gene expression profiling offers insights into the role of innate immune signaling in SSc Serum protein profiling of systemic lupus erythematosus and systemic sclerosis using recombinant antibody microarrays SOCS3 regulates graft-versus-host disease.High IL-17E and low IL-17C dermal expression identifies a fibrosis-specific motif common to morphea and systemic sclerosis.Elevated levels of CD4(+)CD25(+)FoxP3(+) T cells in systemic sclerosis patients contribute to the secretion of IL-17 and immunosuppression dysfunction.Interleukin 13- and interleukin 17A-induced pulmonary hypertension phenotype due to inhalation of antigen and fine particles from air pollution Characterization of HCV-specific CD4+Th17 immunity in recurrent hepatitis C-induced liver allograft fibrosis.Experimentally-derived fibroblast gene signatures identify molecular pathways associated with distinct subsets of systemic sclerosis patients in three independent cohorts.Interleukin-17A promotes functional activation of systemic sclerosis patient-derived dermal vascular smooth muscle cells by extracellular-regulated protein kinases signalling pathway.IL-17A induces endothelial inflammation in systemic sclerosis via the ERK signaling pathway TLR7, IFNγ, and T-bet: their roles in the development of ABCs in female-biased autoimmunity.Vascular involvement in systemic sclerosis (scleroderma).Tanshinone IIA attenuates interleukin-17A-induced systemic sclerosis patient-derived dermal vascular smooth muscle cell activation via inhibition of the extracellular signal-regulated kinase signaling pathway Pathogenesis of Systemic Sclerosis.Increased frequency of circulating Th22 in addition to Th17 and Th2 lymphocytes in systemic sclerosis: association with interstitial lung disease.The Role of IL-17 and Th17 Lymphocytes in Autoimmune Diseases IL-21 Promotes Pulmonary Fibrosis through the Induction of Profibrotic CD8+ T Cells.Interleukin-17A and interleukin-23 in morphea.Cytokine profiles in localized scleroderma and relationship to clinical features.Frequency of circulating topoisomerase-I-specific CD4 T cells predicts presence and progression of interstitial lung disease in scleroderma Phenotypic Alterations Involved in CD8+ Treg Impairment in Systemic Sclerosis Interferon-γ and systemic autoimmunity.Th17 cytokines and arthritis Th17 cells favor inflammatory responses while inhibiting type I collagen deposition by dermal fibroblasts: differential effects in healthy and systemic sclerosis fibroblasts.Increased frequency of Th17 cells in systemic sclerosis is related to disease activity and collagen overproduction.Autologous haemopoietic stem cell transplantation for autoimmune diseases.Deciphering the genetic background of systemic sclerosis.Immunotherapeutic implication of IL-6 blockade.Circulating biomarkers of interstitial lung disease in systemic sclerosis.Is CD69 an effective brake to control inflammatory diseases?Transforming growth factor β--at the centre of systemic sclerosis.CD4+ Group 1 Innate Lymphoid Cells (ILC) Form a Functionally Distinct ILC Subset That Is Increased in Systemic Sclerosis.Photopheresis: Advances and Use in Systemic Sclerosis.Aberrant immune response with consequent vascular and connective tissue remodeling - causal to scleroderma and associated syndromes such as Raynaud phenomenon and other fibrosing syndromes?IL-17, IL-6 and IFN-γ in Systemic Sclerosis Patients.CD69: from activation marker to metabolic gatekeeper.Potential roles of interleukin-17A in the development of skin fibrosis in mice.Crucial role for CD69 in allergic inflammatory responses: CD69-Myl9 system in the pathogenesis of airway inflammation.

P2860

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P2860

The pronounced Th17 profile in systemic sclerosis (SSc) together with intracellular expression of TGFbeta and IFNgamma distinguishes SSc phenotypes.

http://www.wikidata.org/entity/Q33470222

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

The pronounced Th17 profile in ...... distinguishes SSc phenotypes.

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The pronounced Th17 profile in ...... distinguishes SSc phenotypes.

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The pronounced Th17 profile in ...... distinguishes SSc phenotypes.

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The pronounced Th17 profile in ...... distinguishes SSc phenotypes.

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Anila Hussiani

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Dirk M Wuttge

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Erik Lubberts

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Jasper Broen

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Lenny van Bon

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Robbert Simms

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Roger Hesselstrand

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Timothy R D J Radstake

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Yanhui Deng

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P2860

Toll-like receptor signalling

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

Development, cytokine profile and function of human interleukin 17-producing helper T cells

Phenotypic and functional features of human Th17 cells

Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis

Increased expression of interleukin 17 in inflammatory bowel disease

Increase in activated T cells and reduction in suppressor inducer T cells in systemic sclerosis.

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6

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26301207

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