Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle. - Wikidata - awgldk - TriplyDB

Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle.

Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle.

http://www.wikidata.org/entity/Q33474804

about

Understanding the interplay between host immunity and Epstein-Barr virus in NPC patients Therapeutic implications of Epstein-Barr virus infection for the treatment of nasopharyngeal carcinoma Defining immune engagement thresholds for in vivo control of virus-driven lymphoproliferation Viral inhibition of the transporter associated with antigen processing (TAP): a striking example of functional convergent evolution A negative feedback modulator of antigen processing evolved from a frameshift in the cowpox virus genome.Identification of Epstein-Barr Virus Replication Proteins in Burkitt's Lymphoma Cells.Kaposi's sarcoma-associated herpesvirus K3 and K5 ubiquitin E3 ligases have stage-specific immune evasion roles during lytic replication Epstein-Barr virus evades CD4+ T cell responses in lytic cycle through BZLF1-mediated downregulation of CD74 and the cooperation of vBcl-2.The EBV immunoevasins vIL-10 and BNLF2a protect newly infected B cells from immune recognition and elimination Unexpected patterns of Epstein-Barr virus transcription revealed by a high throughput PCR array for absolute quantification of viral mRNA.Persistent infection drives the development of CD8+ T cells specific for late lytic infection antigens in lymphocryptovirus-infected macaques and Epstein-Barr virus-infected humans.Epstein-Barr virus isolates retain their capacity to evade T cell immunity through BNLF2a despite extensive sequence variation.Latent Expression of the Epstein-Barr Virus (EBV)-Encoded Major Histocompatibility Complex Class I TAP Inhibitor, BNLF2a, in EBV-Positive Gastric Carcinomas Human herpesviridae methods of natural killer cell evasion An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Hiding lipid presentation: viral interference with CD1d-restricted invariant natural killer T (iNKT) cell activation EBV BILF1 evolved to downregulate cell surface display of a wide range of HLA class I molecules through their cytoplasmic tail.Epstein-Barr virus microRNAs reduce immune surveillance by virus-specific CD8+ T cells.Immune responses to Epstein-Barr virus: molecular interactions in the virus evasion of CD8+ T cell immunity Epstein-Barr virus genetics: talking about the BAC generation.Evasion of adaptive and innate immune response mechanisms by γ-herpesviruses.Modes of infection and oncogenesis by the Epstein-Barr virus.Epstein- Barr Virus: Clinical and Epidemiological Revisits and Genetic Basis of Oncogenesis The Missing Link in Epstein-Barr Virus Immune Evasion: the BDLF3 Gene Induces Ubiquitination and Downregulation of Major Histocompatibility Complex Class I (MHC-I) and MHC-II p53 and Sp1 cooperate to regulate the expression of Epstein-Barr viral Zta protein.The Epstein-Barr virus-encoded BILF1 protein modulates immune recognition of endogenously processed antigen by targeting major histocompatibility complex class I molecules trafficking on both the exocytic and endocytic pathways Cloning of the Epstein-Barr virus-related rhesus lymphocryptovirus as a bacterial artificial chromosome: a loss-of-function mutation of the rhBARF1 immune evasion gene.An Epstein-Barr Virus MicroRNA Blocks Interleukin-1 (IL-1) Signaling by Targeting IL-1 Receptor 1.Sequence analysis of EBV immune evasion gene BNLF2a in EBV associated tumors and healthy individuals from nasopharyngeal carcinoma endemic and non-endemic regions of China.CD8+ T cell responses to lytic EBV infection: late antigen specificities as subdominant components of the total response.Cooperation between Epstein-Barr virus immune evasion proteins spreads protection from CD8+ T cell recognition across all three phases of the lytic cycle.Upregulation of interleukin 7 receptor alpha and programmed death 1 marks an epitope-specific CD8+ T-cell response that disappears following primary Epstein-Barr virus infection.Cytotoxic CD4+ T cell responses to EBV contrast with CD8 responses in breadth of lytic cycle antigen choice and in lytic cycle recognition Innate immune modulation in EBV infection.Mechanism of activation of the BNLF2a immune evasion gene of Epstein-Barr virus by Zta.Herpesviruses: Harmonious Pathogens but Relevant Cofactors in Other Diseases?Proteome-wide analysis of CD8+ T cell responses to EBV reveals differences between primary and persistent infection

P2860

Q26823898-8E73FF7B-F3E9-4DF2-893C-0049305AD08E Q27016002-3614FF0B-2732-4AD3-9703-727681C15131 Q27324746-C10EB2F1-086E-4EC3-ADD5-8EE809C97D96 Q28083232-C5B39C96-178A-4192-A722-F91EA7DC503A Q30422739-1049F197-4F9D-47B7-B38E-510658BFE566 Q30488509-CA6BC76B-AFB5-49A7-B792-7083D0BD7974 Q34059252-479E8BB9-8D4A-436C-A7F3-64A8864E7504 Q34116845-5461620B-1D88-433C-B44E-C5B9CFFE3002 Q34277034-888CD3BC-F512-40DC-8E44-BF181D3AAC57 Q34703035-5E50088E-F545-41A1-B5C9-DF646E2FBFB6 Q35531669-89742D60-2D0E-43AE-9BB3-ED7EC6CA1E05 Q35665981-798DF704-1396-45E8-B5DA-4FE5A4978F3D Q36080810-529D274C-2045-4DA3-9409-99083BC217DB Q36102137-2F7CA3A2-464C-468D-BB2C-1DA94ED30A84 Q36171933-F64FAC60-4659-4AB4-8965-D6CF9911B6AA Q36395876-3BEB6FA0-5B33-47E9-A11F-3172A69FF044 Q36590231-2201F175-9854-444C-AF9E-44A7D7557E8F Q37369220-9612F75A-7EBE-4024-AFC6-021B5F1337DC Q37652046-B91DB794-707E-4B8D-8A33-FFE86921EF22 Q37856451-D23BF3C7-08BF-48EB-9EF0-E65CD0DE6043 Q38111863-A51995CA-FCAB-4316-8406-753C651BEE38 Q38191876-32417780-E520-4B4A-9A0E-A549F9F05B13 Q38257974-CD1BFED4-9A0E-43F7-833C-8752BDFF1622 Q38827812-106C33E4-F8B4-4C37-8C8C-7DE1DC7C369D Q39328814-E044BEED-6B1E-48D8-BA76-40A716E6B7CA Q39625133-F959B18A-7265-49DE-9D4A-AFF6D7F3A8C1 Q39630573-0B98AAA2-2120-4764-BC2C-E3ACC0ED9934 Q40088720-2AC27B45-2D29-41A9-A9E8-30EF06FBF768 Q40955418-6A37746F-1BB1-47B4-8DA2-2A182C5E5D7C Q41591290-2467B321-F55C-4A3D-830B-85B15A7D7C8F Q41807836-0A84CD81-9332-42C6-93EE-F5F2D919DC4D Q41951746-D47A13BB-1F1A-4B26-89EC-877AF11A7664 Q42066029-E2A76C11-D1D7-4ED7-AE4D-6A657CCE503A Q42702857-28295899-A3EF-49CE-A484-F56E9C83C40F Q54248129-9D40B1FB-6042-4737-BBAB-8B38DDFF3FCF Q55397851-A90A9759-92FC-4C67-867E-2737A8B9935B Q58699977-D203D07B-8273-4179-B591-DFC349330C2A

P2860

Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle.

http://www.wikidata.org/entity/Q33474804

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@ast

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@en

type

label

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@ast

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@en

prefLabel

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@ast

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@en

P1433

Q33474804-3D2F6E44-2DBD-434A-8DCC-C26CA060C3D2

P1476

Q33474804-8E40D44B-85FA-4539-A21F-6C64F3E5AB3C

P2093

Q33474804-10EC0E0C-F36E-4AAC-B3CF-6F68E5301A0E

Q33474804-55DCC4F8-98C1-48E8-ACB3-EA8746936169

Q33474804-67F68287-A3CC-4FB3-88AC-E437A099AB57

Q33474804-7355B28F-1903-4E2B-8F55-15439B702ABC

Q33474804-94CAE370-C74D-4619-BA79-E8BD771FD9AA

Q33474804-9C16D2A8-4BDA-4D99-8981-FED02469831E

Q33474804-9D246CCE-6904-473C-B4BA-9414877A12D9

P2860

Q33474804-03EE593F-0FFE-413E-9BB9-30E85B4A771E

Q33474804-043EE00C-2B68-4E81-A490-D80A236391A4

Q33474804-07FE374B-3D7C-4448-A69D-715EB14751E3

Q33474804-0EE90F30-DC29-40FB-B3AB-2707D429278B

Q33474804-13F930EA-C212-4001-B009-1822ED883700

Q33474804-1A81BD70-583D-41B3-837D-A55DBB471707

Q33474804-24F224F8-EA7D-4EDF-AA4C-17E26DA44278

Q33474804-2E39F8C8-181A-4168-A7D4-BF14D196E411

Q33474804-333B20DD-1E23-48E4-9D26-A2B235BA7F2B

Q33474804-350FBF20-53E6-4388-A080-994AA1763EA2

P304

Q33474804-9AF03AAC-46B3-462C-AD2E-326B766FC8FC

P31

Q33474804-2C5E365A-0432-49C3-A0F3-14A15D7FE5AE

P356

Q33474804-84D0E07F-0E76-496A-BA12-04263ED8822D

P433

Q33474804-70994544-B12C-43D6-815E-F24B10D2654D

P478

Q33474804-E6B09538-06B8-4B08-AA00-CF8129E2EC0D

P50

Q33474804-0586083D-90F4-4DBA-B3C6-B1E49DA99627

Q33474804-1877F6DE-B0BB-40EE-A573-4BA574D91FDB

Q33474804-D9C5B7BF-7D13-4B0C-BADA-242105DF3EEB

Q33474804-E3C452BD-BE9F-4F82-B4FC-6FC27F3628BA

P577

Q33474804-02C86E71-5E1B-4979-82A8-28B7A25477F8

P5875

Q33474804-77084753-E8F1-4B5F-ACFD-D6A0D241B940

P698

Q33474804-A8EC2B67-77B8-4A47-BE6E-3DAA125CDDC5

P921

Q33474804-7E4EF641-5089-40A0-B8F8-DC6D2E57B6D3

P932

Q33474804-A1A1E971-B9A9-4398-AC3A-7854AE04FCFA

P356

JOURNAL.PPAT.1000490

P1433

P1476

Stage-specific inhibition of M ...... tein during virus lytic cycle.

@en

P2093

Alan B Rickinson

http://www.w3.org/2001/XMLSchema#string

Andrew D Hislop

http://www.w3.org/2001/XMLSchema#string

Claire Shannon-Lowe

http://www.w3.org/2001/XMLSchema#string

Daniëlle Horst

http://www.w3.org/2001/XMLSchema#string

Elisabeth Kremmer

http://www.w3.org/2001/XMLSchema#string

Emmanuel J H J Wiertz

http://www.w3.org/2001/XMLSchema#string

Maaike E Ressing

http://www.w3.org/2001/XMLSchema#string

P2860

Complete nucleotide sequence of the rhesus lymphocryptovirus: genetic validation for an Epstein-Barr virus animal model.

Immediate early and early lytic cycle proteins are frequent targets of the Epstein-Barr virus-induced cytotoxic T cell response

The Epstein-Barr virus G-protein-coupled receptor contributes to immune evasion by targeting MHC class I molecules for degradation

Human cytomegalovirus US3 impairs transport and maturation of major histocompatibility complex class I heavy chains.

Virus and cell RNAs expressed during Epstein-Barr virus replication

Viral immune evasion: a masterpiece of evolution.

The Epstein-Barr virus (EBV) early protein EB2 is a posttranscriptional activator expressed under the control of EBV transcription factors EB1 and R.

Host shutoff during productive Epstein-Barr virus infection is mediated by BGLF5 and may contribute to immune evasion.

The role of the secretory immunological synapse in killing by CD8+ CTL.

The Epstein-Barr virus lytic program is controlled by the co-operative functions of two transactivators.

P304

e1000490

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PPAT.1000490

http://www.w3.org/2001/XMLSchema#string

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

5

http://www.w3.org/2001/XMLSchema#string

P50

Nathan P. Croft

Jaap M Middeldorp

P577

2009-06-26T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

26321418

http://www.w3.org/2001/XMLSchema#string

P698

19557156

http://www.w3.org/2001/XMLSchema#string

P921

Epstein–Barr virus

P932

2695766

http://www.w3.org/2001/XMLSchema#string