SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism. - Wikidata - awgldk - TriplyDB

SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.

SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.

http://www.wikidata.org/entity/Q33553351

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Animal models for SARS and MERS coronaviruses Molecular pathology of emerging coronavirus infections Inborn errors of human STAT1: allelic heterogeneity governs the diversity of immunological and infectious phenotypes Lack of innate interferon responses during SARS coronavirus infection in a vaccination and reinfection ferret model A decade after SARS: strategies for controlling emerging coronaviruses The role of epidermal growth factor receptor (EGFR) signaling in SARS coronavirus-induced pulmonary fibrosis.Severe acute respiratory syndrome coronavirus envelope protein ion channel activity promotes virus fitness and pathogenesis The PDZ-binding motif of severe acute respiratory syndrome coronavirus envelope protein is a determinant of viral pathogenesis.Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.Transcriptomic analysis reveals a mechanism for a prefibrotic phenotype in STAT1 knockout mice during severe acute respiratory syndrome coronavirus infection.Unique signatures of long noncoding RNA expression in response to virus infection and altered innate immune signaling.Successful vaccination strategies that protect aged mice from lethal challenge from influenza virus and heterologous severe acute respiratory syndrome coronavirus.Coronavirus non-structural protein 16: evasion, attenuation, and possible treatments.Toll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus Infection Molecular determinants of severe acute respiratory syndrome coronavirus pathogenesis and virulence in young and aged mouse models of human disease.Genome Wide Identification of SARS-CoV Susceptibility Loci Using the Collaborative Cross.Into the eye of the cytokine storm.Recent progress in studies of arterivirus- and coronavirus-host interactions The effect of inhibition of PP1 and TNFα signaling on pathogenesis of SARS coronavirus Critical role of phospholipase A2 group IID in age-related susceptibility to severe acute respiratory syndrome-CoV infection.Identification of the Mechanisms Causing Reversion to Virulence in an Attenuated SARS-CoV for the Design of a Genetically Stable Vaccine.Intranasal treatment with poly(I•C) protects aged mice from lethal respiratory virus infections.Induction of alternatively activated macrophages enhances pathogenesis during severe acute respiratory syndrome coronavirus infection A live, impaired-fidelity coronavirus vaccine protects in an aged, immunocompromised mouse model of lethal disease.Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice.Development of animal models against emerging coronaviruses: From SARS to MERS coronavirus Release of severe acute respiratory syndrome coronavirus nuclear import block enhances host transcription in human lung cells.Type I interferons in viral control and immune regulation Mechanisms of severe acute respiratory syndrome coronavirus-induced acute lung injury.Wild-type and innate immune-deficient mice are not susceptible to the Middle East respiratory syndrome coronavirus.CXCL10-CXCR3 enhances the development of neutrophil-mediated fulminant lung injury of viral and nonviral origin.Epigenetic Landscape during Coronavirus Infection A molecular arms race between host innate antiviral response and emerging human coronaviruses.Middle East respiratory syndrome and severe acute respiratory syndrome.Overactive Epidermal Growth Factor Receptor Signaling Leads to Increased Fibrosis after Severe Acute Respiratory Syndrome Coronavirus Infection.Pathogenic human coronavirus infections: causes and consequences of cytokine storm and immunopathology.SARS-CoV nucleocapsid protein interacts with cellular pyruvate kinase protein and inhibits its activity.Efficient Reverse Genetic Systems for Rapid Genetic Manipulation of Emergent and Preemergent Infectious Coronaviruses.Airway Memory CD4(+) T Cells Mediate Protective Immunity against Emerging Respiratory Coronaviruses.Murine norovirus inhibits B cell development in the bone marrow of STAT1-deficient mice.

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SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.

http://www.wikidata.org/entity/Q33553351

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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SARS-CoV pathogenesis is regul ...... eceptor independent mechanism.

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SARS-CoV pathogenesis is regul ...... eceptor independent mechanism.

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SARS-CoV pathogenesis is regul ...... eceptor independent mechanism.

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SARS-CoV pathogenesis is regul ...... eceptor independent mechanism.

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SARS-CoV pathogenesis is regul ...... eceptor independent mechanism.

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SARS-CoV pathogenesis is regul ...... eceptor independent mechanism.

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Alan Whitmore

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Elaine W Lamirande

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Jerrold M Ward

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Jun Chen

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Kanta Subbarao

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Thomas E Morrison

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William Funkhouser

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P2860

IFN-lambdas mediate antiviral protection through a distinct class II cytokine receptor complex

Influenza B virus NS1 protein inhibits conjugation of the interferon (IFN)-induced ubiquitin-like ISG15 protein

Time course and cellular localization of SARS-CoV nucleoprotein and RNA in lungs from fatal cases of SARS.

Resistance to Alpha/Beta Interferon Is a Determinant of West Nile Virus Replication Fitness and Virulence

Viruses and interferon: a fight for supremacy

Pathogenesis of severe acute respiratory syndrome

Signal transducers and activators of transcription (STATs): Novel targets of chemopreventive and chemotherapeutic drugs

Identification of a novel coronavirus in patients with severe acute respiratory syndrome

Targeted disruption of the mouse Stat1 gene results in compromised innate immunity to viral disease

Transcriptional responses to polypeptide ligands: the JAK-STAT pathway

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Anjeanette Roberts

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severe acute respiratory syndrome-related coronavirus

severe acute respiratory syndrome coronavirus

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2851658

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