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Complement contributes to inflammatory tissue destruction in a mouse model of Ross River virus-induced diseaseResidue 82 of the Chikungunya virus E2 attachment protein modulates viral dissemination and arthritis in miceHigh rates of o'nyong nyong and Chikungunya virus transmission in coastal Kenyaγδ T Cells Play a Protective Role in Chikungunya Virus-Induced DiseaseDevelopment of a highly protective combination monoclonal antibody therapy against Chikungunya virusModeling host genetic regulation of influenza pathogenesis in the collaborative crossDifferential Induction of Type I Interferon Responses in Myeloid Dendritic Cells by Mosquito and Mammalian-Cell-Derived AlphavirusesModulation of type I IFN induction by a virulence determinant within the alphavirus nsP1 proteinIdentifying protective host gene expression signatures within the spleen during West Nile virus infection in the collaborative cross modelVaccine efficacy in senescent mice challenged with recombinant SARS-CoV bearing epidemic and zoonotic spike variantsA Reverse Genetics Platform That Spans the Zika Virus Family TreeAnimal Models of Chikungunya Virus Infection and Disease.A mouse model for Betacoronavirus subgroup 2c using a bat coronavirus strain HKU5 variantA protective role for complement C3 protein during pandemic 2009 H1N1 and H5N1 influenza A virus infectionA mouse-adapted SARS-coronavirus causes disease and mortality in BALB/c mice.MyD88 is required for protection from lethal infection with a mouse-adapted SARS-CoV.SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.Vaccination with DNA plasmids expressing Gn coupled to C3d or alphavirus replicons expressing gn protects mice against Rift Valley fever virus.Identification of adult mouse neurovirulence determinants of the Sindbis virus strain AR86.Regulation of the hepatitis C virus RNA replicase by endogenous lipid peroxidation.Genomic profiling of collaborative cross founder mice infected with respiratory viruses reveals novel transcripts and infection-related strain-specific gene and isoform expression.Mannose binding lectin is required for alphavirus-induced arthritis/myositis.Unique signatures of long noncoding RNA expression in response to virus infection and altered innate immune signaling.Chikungunya: a re-emerging virus.Annotation of long non-coding RNAs expressed in collaborative cross founder mice in response to respiratory virus infection reveals a new class of interferon-stimulated transcriptsCharacterization of Ross River virus tropism and virus-induced inflammation in a mouse model of viral arthritis and myositisHost genetic diversity enables Ebola hemorrhagic fever pathogenesis and resistance.Successful vaccination strategies that protect aged mice from lethal challenge from influenza virus and heterologous severe acute respiratory syndrome coronavirus.A gnotobiotic mouse model demonstrates that dietary fiber protects against colorectal tumorigenesis in a microbiota- and butyrate-dependent manner.NLRX1 is a regulator of mitochondrial antiviral immunity.A mouse model of chikungunya virus-induced musculoskeletal inflammatory disease: evidence of arthritis, tenosynovitis, myositis, and persistence.Acute and chronic B cell depletion disrupts CD4+ and CD8+ T cell homeostasis and expansion during acute viral infection in miceGenetic analysis of complex traits in the emerging Collaborative Cross.Natural resistance-associated macrophage protein is a cellular receptor for sindbis virus in both insect and mammalian hosts.Chemokines and viruses: friends or foes?A double-inactivated severe acute respiratory syndrome coronavirus vaccine provides incomplete protection in mice and induces increased eosinophilic proinflammatory pulmonary response upon challengeIntegrative deep sequencing of the mouse lung transcriptome reveals differential expression of diverse classes of small RNAs in response to respiratory virus infectionToll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus InfectionExpression quantitative trait Loci for extreme host response to influenza a in pre-collaborative cross mice.Genome Wide Identification of SARS-CoV Susceptibility Loci Using the Collaborative Cross.
P50
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P50
name
Mark T Heise
@en
type
label
Mark T Heise
@en
prefLabel
Mark T Heise
@en