Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
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Targeting gastrointestinal stromal tumors: the role of regorafenibFunctional Relationship between Tumor-Associated Macrophages and Macrophage Colony-Stimulating Factor as Contributors to Cancer ProgressionEliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology.Colony-stimulating factor 1 receptor blockade prevents fractionated whole-brain irradiation-induced memory deficitsColony-stimulating factor 1 receptor inhibition prevents microglial plaque association and improves cognition in 3xTg-AD mice.CCR 20th Anniversary Commentary: A Genetic Mechanism of Imatinib Resistance in Gastrointestinal Stromal Tumor-Where Are We a Decade Later?Targeting cells of the myeloid lineage attenuates pain and disease progression in a prostate model of bone cancerTargeting myeloid-derived suppressor cells with colony stimulating factor-1 receptor blockade can reverse immune resistance to immunotherapy in indoleamine 2,3-dioxygenase-expressing tumorsVHL-deficient renal cancer cells gain resistance to mitochondria-activating apoptosis inducers by activating AKT through the IGF1R-PI3K pathway.Pharmacological Inhibition of KIT Activates MET Signaling in Gastrointestinal Stromal Tumors.Combined inhibition of MAP kinase and KIT signaling synergistically destabilizes ETV1 and suppresses GIST tumor growth.Clinical development landscape in GIST: from novel agents that target accessory pathways to revisiting non-targeted therapies.Nanomedicine Strategies to Target Tumor-Associated Macrophages.Novel Insights into the Treatment of Imatinib-Resistant Gastrointestinal Stromal Tumors.Tumor-Associated Macrophages as Target for Antitumor Therapy.Emerging drugs for the treatment of gastrointestinal stromal tumour.CSF-1R Inhibitor Development: Current Clinical Status.PD-1/PD-L1 Blockade Enhances T-cell Activity and Antitumor Efficacy of Imatinib in Gastrointestinal Stromal Tumors.Wnt/β-catenin Signaling Contributes to Tumor Malignancy and Is Targetable in Gastrointestinal Stromal Tumor.Microglia prevent peripheral immune cell invasion and promote an anti-inflammatory environment in the brain of APP-PS1 transgenic mice
P2860
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P2860
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@ast
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@en
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor.
@nl
type
label
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@ast
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@en
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor.
@nl
prefLabel
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@ast
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@en
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor.
@nl
P2093
P2860
P50
P1476
Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor
@en
P2093
Anson T Ku
Benjamin L Green
Cristina R Antonescu
Darren R Veach
Eric C Sorenson
Ferdinand Rossi
Jonathan B Greer
Megan H Crawley
Michael J Cavnar
Peter Besmer
P2860
P304
P356
10.1158/1078-0432.CCR-13-3033
P407
P577
2014-02-28T00:00:00Z