Human Langerhans cells induce distinct IL-22-producing CD4+ T cells lacking IL-17 production. - Wikidata - awgldk - TriplyDB

Human Langerhans cells induce distinct IL-22-producing CD4+ T cells lacking IL-17 production.

Human Langerhans cells induce distinct IL-22-producing CD4+ T cells lacking IL-17 production.

http://www.wikidata.org/entity/Q33564666

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Molecular Mechanisms of Cutaneous Inflammatory Disorder: Atopic Dermatitis The Newly Identified T Helper 22 Cells Lodge in Leukemia Regulation of Dendritic Cell Function in Inflammation Distinct functional programs in fetal T and myeloid lineages T Helper Subsets, Peripheral Plasticity, and the Acute Phase Protein, α1-Antitrypsin The known unknowns of the human dendritic cell network Immunology of psoriasis Understanding dendritic cells and their role in cutaneous carcinoma and cancer immunotherapy Th22 cells in allergic disease T Helper Lymphocyte Subsets and Plasticity in Autoimmunity and Cancer: An Overview Antigen-Presenting Human γδ T Cells Promote Intestinal CD4+ T Cell Expression of IL-22 and Mucosal Release of Calprotectin New insights in the immunologic basis of psoriasis.Th17 cells and activated dendritic cells are increased in vitiligo lesions The pathophysiological role of dendritic cell subsets in psoriasis.Reversal of atopic dermatitis with narrow-band UVB phototherapy and biomarkers for therapeutic response.Cytokine requirements for the differentiation and expansion of IL-17A- and IL-22-producing human Vgamma2Vdelta2 T cells.Computational modeling of heterogeneity and function of CD4+ T cells Tailored immunity by skin antigen-presenting cells.Role of interleukin-22 in inflammatory bowel disease Differential capacity of human skin dendritic cells to polarize CD4+ T cells into IL-17, IL-21 and IL-22 producing cells Transcriptional fingerprints of antigen-presenting cell subsets in the human vaginal mucosa and skin reflect tissue-specific immune microenvironments.Effector CD4+ T cells, the cytokines they generate, and GVHD: something old and something new.Atopic dermatitis results in intrinsic barrier and immune abnormalities: implications for contact dermatitis.IL-10 conditioning of human skin affects the distribution of migratory dendritic cell subsets and functional T cell differentiation.Nonlesional atopic dermatitis skin is characterized by broad terminal differentiation defects and variable immune abnormalities.Identification of novel immune and barrier genes in atopic dermatitis by means of laser capture microdissection.Vitamin D regulates cytokine patterns secreted by dendritic cells to promote differentiation of IL-22-producing T cells.Langerhans cell precursors acquire RANK/CD265 in prenatal human skin.Emerging Role of Interleukin 22 in Hepatitis B Virus Infection: a Double-edged Sword.Contrasting Roles of IL-22 and IL-17 in Murine Genital Tract Infection by Neisseria gonorrhoeae.Pathogen-specific T cell depletion and reactivation of opportunistic pathogens in HIV infection.A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.Existence of Th22 in children and evaluation of IL-22 + CD4 + T, Th17, and other T cell effector subsets from healthy children compared to adults Functional diversity of human vaginal APC subsets in directing T-cell responses.Calcitonin Gene-Related Peptide-Exposed Endothelial Cells Bias Antigen Presentation to CD4+ T Cells toward a Th17 Response.Dendritic cells and vaccine design for sexually-transmitted diseases.Pituitary adenylate cyclase-activating peptide and vasoactive intestinal polypeptide bias Langerhans cell Ag presentation toward Th17 cells.IL-23 induces IL-22 and IL-17 production in response to Chlamydia muridarum genital tract infection, but the absence of these cytokines does not influence disease pathogenesis.Interleukin 22 inhibits intracellular growth of Mycobacterium tuberculosis by enhancing calgranulin A expression.The Th17 pathway and inflammatory diseases of the intestines, lungs, and skin

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Human Langerhans cells induce distinct IL-22-producing CD4+ T cells lacking IL-17 production.

http://www.wikidata.org/entity/Q33564666

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Human Langerhans cells induce ...... ells lacking IL-17 production.

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Human Langerhans cells induce ...... ells lacking IL-17 production.

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Human Langerhans cells induce ...... ells lacking IL-17 production.

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type

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Human Langerhans cells induce ...... ells lacking IL-17 production.

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Human Langerhans cells induce ...... ells lacking IL-17 production.

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Human Langerhans cells induce ...... ells lacking IL-17 production.

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PNAS.0911472106

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Human Langerhans cells induce ...... ells lacking IL-17 production.

@en

P2093

Hideki Fujita

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James G Krueger

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Juana Gonzalez

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Kristine E Nograles

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Toyoko Kikuchi

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P2860

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

The biological functions of T helper 17 cell effector cytokines in inflammation

Memory IL-22-producing CD4+ T cells specific for Candida albicans are present in humans

A human natural killer cell subset provides an innate source of IL-22 for mucosal immunity

Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

IL-22-producing "T22" T cells account for upregulated IL-22 in atopic dermatitis despite reduced IL-17-producing TH17 T cells.

Cytokine-producing dendritic cells in the pathogenesis of inflammatory skin diseases.

Cutting edge: immune cells as sources and targets of the IL-10 family members?

Origin, homeostasis and function of Langerhans cells and other langerin-expressing dendritic cells.

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21795-21800

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106

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19996179

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2799849

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