A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis. - Wikidata - awgldk - TriplyDB

A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

http://www.wikidata.org/entity/Q36005402

about

Myeloid-Derived Suppressor Cells in Bacterial Infections Paving the Road to Tumor Development and Spreading: Myeloid-Derived Suppressor Cells are Ruling the Fate Differential inflammatory response to Helicobacter pylori infection: etiology and clinical outcomes IL-22 Restrains Tapeworm-Mediated Protection against Experimental Colitis via Regulation of IL-25 Expression Antigen-Presenting Human γδ T Cells Promote Intestinal CD4+ T Cell Expression of IL-22 and Mucosal Release of Calprotectin Interleukin-7 produced by intestinal epithelial cells in response to Citrobacter rodentium infection plays a major role in innate immunity against this pathogen.IL-17a and IL-22 Induce Expression of Antimicrobials in Gastrointestinal Epithelial Cells and May Contribute to Epithelial Cell Defense against Helicobacter pylori Systems-wide analyses of mucosal immune responses to Helicobacter pylori at the interface between pathogenicity and symbiosis Role of Inflammatory Monocytes in Vaccine-Induced Reduction of Helicobacter felis Infection Loss of gastrokine-2 drives premalignant gastric inflammation and tumor progression.Schlafen 4-expressing myeloid-derived suppressor cells are induced during murine gastric metaplasia.Treatment with Cestode Parasite Antigens Results in Recruitment of CCR2+ Myeloid Cells, the Adoptive Transfer of Which Ameliorates Colitis.STAT3: a critical component in the response to Helicobacter pylori infection.Inflammation, immunity, and vaccines for Helicobacter pylori infection.Cytokines IL-17 and IL-22 in the host response to infection.Cellular and Molecular Dynamics of Th17 Differentiation and its Developmental Plasticity in the Intestinal Immune Response Helicobacter: Inflammation, immunology and vaccines.Nongenetically modified Lactococcus lactis-adjuvanted vaccination enhanced innate immunity against Helicobacter pylori.The importance of TH22 and TC22 cells in the pathogenesis of Helicobacter pylori-associated gastric diseases.Generating a Tolerogenic Cell Therapy Knowledge Graph from Literature.Monocytic Myeloid-Derived Suppressor Cells in Chronic Infections.Helicobacter pylori-induced IL-33 modulates mast cell responses, benefits bacterial growth, and contributes to gastritis.Accumulation of T-helper 22 cells, interleukin-22 and myeloid-derived suppressor cells promotes gastric cancer progression in elderly patients.Helicobacter pylori-infected C57BL/6 mice with different gastrointestinal microbiota have contrasting gastric pathology, microbial and host immune responses.The Interleukin-20 Cytokines in Intestinal Diseases.

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A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

http://www.wikidata.org/entity/Q36005402

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

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A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

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type

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A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

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A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

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A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

@ast

A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

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A pro-inflammatory role for Th22 cells in Helicobacter pylori-associated gastritis.

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Bo-sheng Li

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Fang-yuan Mao

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Gang Guo

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Hong Guo

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Hui-jie Yang

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Liu-sheng Peng

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Liu-yang Yang

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Ming Zeng

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Na Chen

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Ping Cheng

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P2860

Proinflammatory S100 proteins regulate the accumulation of myeloid-derived suppressor cells

Myeloid-derived suppressor cells as regulators of the immune system

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

Human Langerhans cells induce distinct IL-22-producing CD4+ T cells lacking IL-17 production.

Interleukin (IL)-23 mediates Toxoplasma gondii-induced immunopathology in the gut via matrixmetalloproteinase-2 and IL-22 but independent of IL-17.

Helicobacter pylori immune escape is mediated by dendritic cell-induced Treg skewing and Th17 suppression in mice.

IL-22+ CD4+ T cells are associated with therapeutic trichuris trichiura infection in an ulcerative colitis patient.

Nod1 responds to peptidoglycan delivered by the Helicobacter pylori cag pathogenicity island.

IL-22 signaling contributes to West Nile encephalitis pathogenesis.

Human TH17 lymphocytes promote blood-brain barrier disruption and central nervous system inflammation

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Helicobacter pylori

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