Oncogene-induced reactive oxygen species fuel hyperproliferation and DNA damage response activation
about
Reactive Oxygen Species and Targeted Therapy for Pancreatic CancerDNA repair and aging: the impact of the p53 familyMolecular pathways: targeting the dependence of mutant RAS cancers on the DNA damage responsePerspectives on the combination of radiotherapy and targeted therapy with DNA repair inhibitors in the treatment of pancreatic cancermtDNA germ line variation mediated ROS generates retrograde signaling and induces pro-cancerous metabolic featuresStable cellular senescence is associated with persistent DDR activationGenetic Evidence for XPC-KRAS Interactions During Lung Cancer DevelopmentRescue from replication stress during mitosis.AMPKα1 deficiency promotes cellular proliferation and DNA damage via p21 reduction in mouse embryonic fibroblasts.Emerging roles of the p38 MAPK and PI3K/AKT/mTOR pathways in oncogene-induced senescence.Stress-induced DNA damage biomarkers: applications and limitations.Structural and elemental changes in glioblastoma cells in situ: complementary imaging with high resolution visible light- and X-ray microscopy.Biobehavioral and neuroendocrine correlates of antioxidant enzyme activity in ovarian carcinoma.Effects of Low-Dose Bisphenol A on DNA Damage and Proliferation of Breast Cells: The Role of c-MycZNF32 protects against oxidative stress-induced apoptosis by modulating C1QBP transcription.Disulfiram anti-cancer efficacy without copper overload is enhanced by extracellular H2O2 generation: antagonism by tetrathiomolybdate.Identification of linc-NeD125, a novel long non coding RNA that hosts miR-125b-1 and negatively controls proliferation of human neuroblastoma cells.Uric acid and transforming growth factor in fructose-induced production of reactive oxygen species in skeletal muscleCancer prevention and therapy through the modulation of the tumor microenvironment.Metabolic pathways regulated by TAp73 in response to oxidative stressLoss of ARID1A expression leads to sensitivity to ROS-inducing agent elesclomol in gynecologic cancer cells.KRas, ROS and the initiation of pancreatic cancerDual oxidase 2 and pancreatic adenocarcinoma: IFN-γ-mediated dual oxidase 2 overexpression results in H2O2-induced, ERK-associated up-regulation of HIF-1α and VEGF-A.NOX4 downregulation leads to senescence of human vascular smooth muscle cells.Inhibition of cancer antioxidant defense by natural compounds.DNA damage accumulation and repair defects in acute myeloid leukemia: implications for pathogenesis, disease progression, and chemotherapy resistance.The unfolded protein response and cellular senescence. A review in the theme: cellular mechanisms of endoplasmic reticulum stress signaling in health and disease.The human Nox4: gene, structure, physiological function and pathological significance.K-ras Mutations as the Earliest Driving Force in a Subset of Colorectal Carcinomas.Physical interaction of estrogen receptor with MnSOD: implication in mitochondrial O2.- upregulation and mTORC2 potentiation in estrogen-responsive breast cancer cells.Clinical relevance of thyroid cell models in redox research.Reactive oxygen species mediate oridonin-induced apoptosis through DNA damage response and activation of JNK pathway in diffuse large B cell lymphoma.NADPH oxidases: new actors in thyroid cancer?Autophagy-mediated degradation of nuclear envelope proteins during oncogene-induced senescence.LMP1 Increases Expression of NADPH Oxidase (NOX) and Its Regulatory Subunit p22 in NP69 Nasopharyngeal Cells and Makes Them Sensitive to a Treatment by a NOX Inhibitor.IFNγ induces oxidative stress, DNA damage and tumor cell senescence via TGFβ/SMAD signaling-dependent induction of Nox4 and suppression of ANT2.Transplantation and Damage-Associated Molecular Patterns (DAMPs).Inhibition of succinate dehydrogenase by the mitochondrial chaperone TRAP1 has anti-oxidant and anti-apoptotic effects on tumor cells.Prolonged inflammatory microenvironment is crucial for pro-neoplastic growth and genome instability: a detailed review.Targeting reactive oxygen species in development and progression of pancreatic cancer.
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P2860
Oncogene-induced reactive oxygen species fuel hyperproliferation and DNA damage response activation
description
2014 nî lūn-bûn
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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年學術文章
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name
Oncogene-induced reactive oxyg ...... DNA damage response activation
@ast
Oncogene-induced reactive oxyg ...... DNA damage response activation
@en
Oncogene-induced reactive oxyg ...... NA damage response activation.
@nl
type
label
Oncogene-induced reactive oxyg ...... DNA damage response activation
@ast
Oncogene-induced reactive oxyg ...... DNA damage response activation
@en
Oncogene-induced reactive oxyg ...... NA damage response activation.
@nl
prefLabel
Oncogene-induced reactive oxyg ...... DNA damage response activation
@ast
Oncogene-induced reactive oxyg ...... DNA damage response activation
@en
Oncogene-induced reactive oxyg ...... NA damage response activation.
@nl
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Oncogene-induced reactive oxyg ...... DNA damage response activation
@en
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10.1038/CDD.2014.16
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2014-02-28T00:00:00Z