Prevention of steatosis by hepatic JNK1 - Wikidata - awgldk - TriplyDB

Prevention of steatosis by hepatic JNK1

Prevention of steatosis by hepatic JNK1

http://www.wikidata.org/entity/Q33580974

about

JNK Signaling: Regulation and Functions Based on Complex Protein-Protein Partnerships Stress kinases in the modulation of metabolism and energy balance Neuroinflammatory basis of metabolic syndrome Energy metabolism in the liver Neuroinflammation in overnutrition-induced diseases Involvement of the IRE1α-XBP1 pathway and XBP1s-dependent transcriptional reprogramming in metabolic diseases Implication of inflammatory signaling pathways in obesity-induced insulin resistance A liver full of JNK: signaling in regulation of cell function and disease pathogenesis, and clinical approaches Liver Patt1 deficiency protects male mice from age-associated but not high-fat diet-induced hepatic steatosis Mechanisms for insulin resistance: common threads and missing links Role of muscle c-Jun NH2-terminal kinase 1 in obesity-induced insulin resistance.Role of the hypothalamic-pituitary-thyroid axis in metabolic regulation by JNK1 PPAR/RXR Regulation of Fatty Acid Metabolism and Fatty Acid omega-Hydroxylase (CYP4) Isozymes: Implications for Prevention of Lipotoxicity in Fatty Liver Disease An ErChen and YinChen Decoction Ameliorates High-Fat-Induced Nonalcoholic Steatohepatitis in Rats by Regulating JNK1 Signaling Pathway.Inflammation and insulin resistance: an old story with new ideas.Reactive oxygen and nitrogen species in steatotic hepatocytes: a molecular perspective on the pathophysiology of ischemia-reperfusion injury in the fatty liver.The PPARα-FGF21 hormone axis contributes to metabolic regulation by the hepatic JNK signaling pathway.Requirement of JIP1-mediated c-Jun N-terminal kinase activation for obesity-induced insulin resistance.MAPK signalling in cellular metabolism: stress or wellness?cJun NH2-terminal kinase 1 (JNK1): roles in metabolic regulation of insulin resistance JNK regulation of hepatic manifestations of the metabolic syndrome.Hepatic mitogen-activated protein kinase phosphatase 1 selectively regulates glucose metabolism and energy homeostasis.Female Nur77-deficient mice show increased susceptibility to diet-induced obesity.Alteration of JNK-1 signaling in skeletal muscle fails to affect glucose homeostasis and obesity-associated insulin resistance in mice Endoplasmic reticulum stress and the unfolded protein response in nonalcoholic fatty liver disease.Loss of mitogen-activated protein kinase phosphatase-1 protects from hepatic steatosis by repression of cell death-inducing DNA fragmentation factor A (DFFA)-like effector C (CIDEC)/fat-specific protein 27.Sirtuin 1 (SIRT1) protein degradation in response to persistent c-Jun N-terminal kinase 1 (JNK1) activation contributes to hepatic steatosis in obesity.The metabolically benign and malignant fatty liver.Inflammatory concepts of obesity.New insights into insulin resistance in the diabetic heart.Hypothalamic inflammation: a double-edged sword to nutritional diseases.Dissociation of inositol-requiring enzyme (IRE1α)-mediated c-Jun N-terminal kinase activation from hepatic insulin resistance in conditional X-box-binding protein-1 (XBP1) knock-out mice.Mechanisms Linking Inflammation to Insulin Resistance Cytochrome P4502E1, oxidative stress, JNK, and autophagy in acute alcohol-induced fatty liver.Hepatic cannabinoid receptor-1 mediates diet-induced insulin resistance via inhibition of insulin signaling and clearance in mice.Adipose tissue dysfunction in humans: a potential role for the transmembrane protein ENPP1.Hepatic apoptosis postburn is mediated by c-Jun N-terminal kinase 2.p38γ and p38δ reprogram liver metabolism by modulating neutrophil infiltration.Skeletal muscle-specific overproduction of constitutively activated c-Jun N-terminal kinase (JNK) induces insulin resistance in mice Central melanin-concentrating hormone influences liver and adipose metabolism via specific hypothalamic nuclei and efferent autonomic/JNK1 pathways.

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Prevention of steatosis by hepatic JNK1

http://www.wikidata.org/entity/Q33580974

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Prevention of steatosis by hepatic JNK1

@ast

Prevention of steatosis by hepatic JNK1

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Prevention of steatosis by hepatic JNK1.

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type

label

Prevention of steatosis by hepatic JNK1

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Prevention of steatosis by hepatic JNK1

@en

Prevention of steatosis by hepatic JNK1.

@nl

prefLabel

Prevention of steatosis by hepatic JNK1

@ast

Prevention of steatosis by hepatic JNK1

@en

Prevention of steatosis by hepatic JNK1.

@nl

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J.CMET.2009.09.007

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Cell Metabolism

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Prevention of steatosis by hepatic JNK1

@en

P2093

Dae Young Jung

http://www.w3.org/2001/XMLSchema#string

Hwi Jin Ko

http://www.w3.org/2001/XMLSchema#string

Jason K Kim

http://www.w3.org/2001/XMLSchema#string

John Y Jun

http://www.w3.org/2001/XMLSchema#string

Julie Cavanagh-Kyros

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Roger J Davis

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Susan Gray

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P2860

A nucleoprotein complex containing Sp1, C/EBP beta, and HMGI-Y controls human insulin receptor gene transcription

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

CEACAM1 regulates insulin clearance in liver

Control of hepatic gluconeogenesis through the transcriptional coactivator PGC-1

Insulin degradation: progress and potential

Insulin/IGF-1 and TNF-alpha stimulate phosphorylation of IRS-1 at inhibitory Ser307 via distinct pathways

Dual roles for glucokinase in glucose homeostasis as determined by liver and pancreatic beta cell-specific gene knock-outs using Cre recombinase

A central role for JNK in obesity and insulin resistance

Insulin-regulated hepatic gluconeogenesis through FOXO1-PGC-1alpha interaction

Cell-permeable peptide inhibitors of JNK: novel blockers of beta-cell death.

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491-498

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10.1016/J.CMET.2009.09.007

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6

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10

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Guadalupe Sabio

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40038543

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19945406

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2804105

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