Differential role for c-Rel and C/EBPbeta/delta in TLR-mediated induction of proinflammatory cytokines. - Wikidata - awgldk - TriplyDB

Differential role for c-Rel and C/EBPbeta/delta in TLR-mediated induction of proinflammatory cytokines.

Differential role for c-Rel and C/EBPbeta/delta in TLR-mediated induction of proinflammatory cytokines.

http://www.wikidata.org/entity/Q33588112

about

A key role for NF-κB transcription factor c-Rel in T-lymphocyte-differentiation and effector functions C/EBPδ deficiency sensitizes mice to ionizing radiation-induced hematopoietic and intestinal injury The IL-17 pathway as a major therapeutic target in autoimmune diseases A20-binding inhibitor of NF-κB (ABIN1) controls Toll-like receptor-mediated CCAAT/enhancer-binding protein β activation and protects from inflammatory disease Elevated CO2 selectively inhibits interleukin-6 and tumor necrosis factor expression and decreases phagocytosis in the macrophage.The transcription factor C/EBP delta has anti-apoptotic and anti-inflammatory roles in pancreatic beta cells CCAAT/enhancer-binding protein β and NF-κB mediate high level expression of chemokine genes CCL3 and CCL4 by human chondrocytes in response to IL-1β.Molecular mechanism underlying persistent induction of LCN2 by lipopolysaccharide in kidney fibroblasts Suppressor of cytokine signaling 3 inhibits LPS-induced IL-6 expression in osteoblasts by suppressing CCAAT/enhancer-binding protein {beta} activity Role of macrophage CCAAT/enhancer binding protein delta in the pathogenesis of rheumatoid arthritis in collagen-induced arthritic mice.Resistin stimulates expression of chemokine genes in chondrocytes via combinatorial regulation of C/EBPβ and NF-κB Molecular mechanisms that influence the macrophage m1-m2 polarization balance.Transcription of Tnfaip3 is regulated by NF-κB and p38 via C/EBPβ in activated macrophages.Biological roles of CCAAT/Enhancer-binding protein delta during inflammation.Expression of toll-like receptors on breast tumors: taking a toll on tumor microenvironment.Arc of a vicious circle: pathways activated by Mycobacterium tuberculosis that target the HIV-1 long terminal repeat.C5a-regulated CCAAT/enhancer-binding proteins β and δ are essential in Fcγ receptor-mediated inflammatory cytokine and chemokine production in macrophages.CCAAT/enhancer binding protein-δ expression by dendritic cells regulates CNS autoimmune inflammatory disease.Critical role for CCAAT/enhancer-binding protein β in immune complex-induced acute lung injury.CCAAT/enhancer-binding protein β (C/EBPβ) expression regulates dietary-induced inflammation in macrophages and adipose tissue in mice TRAF3: a novel tumor suppressor gene in macrophages.Prognostic impact of c-Rel nuclear expression and REL amplification and crosstalk between c-Rel and the p53 pathway in diffuse large B-cell lymphoma.Targeting chemotherapy-induced PTX3 in tumor stroma to prevent the progression of drug-resistant cancers.Targeting activation of specific NF-κB subunits prevents stress-dependent atherothrombotic gene expression.CCAAT/enhancer-binding protein δ is a critical mediator of lipopolysaccharide-induced acute lung injury.Links between Toll-like receptor 4 and breast cancer γ-Tocotrienol inhibits lipopolysaccharide-induced interlukin-6 and granulocyte colony-stimulating factor by suppressing C/EBPβ and NF-κB in macrophages FBXW7α attenuates inflammatory signalling by downregulating C/EBPδ and its target gene Tlr4.Idiopathic inflammatory myopathies: pathogenic mechanisms of muscle weakness Myeloid depletion of SOCS3 enhances LPS-induced acute lung injury through CCAAT/enhancer binding protein δ pathway.Independent, parallel pathways to CXCL10 induction in HCV-infected hepatocytes.The many faces of C/EBPδ and their relevance for inflammation and cancer.Suppressors of cytokine signaling (SOCS) in T cell differentiation, maturation, and function.IRAK-1 contributes to lipopolysaccharide-induced reactive oxygen species generation in macrophages by inducing NOX-1 transcription and Rac1 activation and suppressing the expression of antioxidative enzymes.Direct, interferon-independent activation of the CXCL10 promoter by NF-κB and interferon regulatory factor 3 during hepatitis C virus infection.Transcriptional control of the TNF gene C/EBP{delta} and STAT-1 are required for TLR8 transcriptional activity.Regulation of the Expression of Heparan Sulfate 3-O-Sulfotransferase 3B (HS3ST3B) by Inflammatory Stimuli in Human Monocytes.Epigenetic and Transcriptional Regulation of IRAK-M Expression in Macrophages.Feasibility of implementing cell-based pathway reporter assays in early high-throughput screening assay cascades for antibody drug discovery.

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Differential role for c-Rel and C/EBPbeta/delta in TLR-mediated induction of proinflammatory cytokines.

http://www.wikidata.org/entity/Q33588112

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Differential role for c-Rel an ...... of proinflammatory cytokines.

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Differential role for c-Rel an ...... of proinflammatory cytokines.

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Differential role for c-Rel an ...... of proinflammatory cytokines.

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Differential role for c-Rel an ...... of proinflammatory cytokines.

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Differential role for c-Rel an ...... of proinflammatory cytokines.

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Differential role for c-Rel an ...... of proinflammatory cytokines.

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JIMMUNOL.0802971

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Journal of Immunology

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Differential role for c-Rel an ...... of proinflammatory cytokines.

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Elizabeth Lye

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Fang Shen

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Ira Kim

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Nobutaka Suzuki

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Sarah L Gaffen

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Shinobu Suzuki

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Shizuo Akira

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Steve Gerondakis

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Wen-Chen Yeh

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P2860

A nuclear factor for IL-6 expression (NF-IL6) is a member of a C/EBP family

Enhanced TLR-mediated NF-IL6 dependent gene expression by Trib1 deficiency

Defective adipocyte differentiation in mice lacking the C/EBPbeta and/or C/EBPdelta gene.

CCAAT/enhancer-binding proteins: structure, function and regulation

A member of the C/EBP family, NF-IL6 beta, forms a heterodimer and transcriptionally synergizes with NF-IL6

Innate immune recognition

Pathogen recognition and innate immunity

NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

NF-kappaB regulation in the immune system

Severe impairment of interleukin-1 and Toll-like receptor signalling in mice lacking IRAK-4

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7212-7221

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10.4049/JIMMUNOL.0802971

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11

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182

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Pamela S Ohashi

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2009-06-01T00:00:00Z

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24439383

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19454718

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Toll-like receptor

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2805849

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