The Pla protease of Yersinia pestis degrades fas ligand to manipulate host cell death and inflammation
about
Temporal Progression of Pneumonic Plague in Blood of Nonhuman Primate: A Transcriptomic AnalysisThe Yersinia Type III secretion effector YopM Is an E3 ubiquitin ligase that induced necrotic cell death by targeting NLRP3Fibrinolytic and procoagulant activities of Yersinia pestis and Salmonella enterica.Yersinia adhesins: An arsenal for infection.Production of outer membrane vesicles by the plague pathogen Yersinia pestisCaspase-3 mediates the pathogenic effect of Yersinia pestis YopM in liver of C57BL/6 mice and contributes to YopM's function in spleen.Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways.Combinational deletion of three membrane protein-encoding genes highly attenuates yersinia pestis while retaining immunogenicity in a mouse model of pneumonic plague.Disruption of fas-fas ligand signaling, apoptosis, and innate immunity by bacterial pathogensCircumventing Y. pestis Virulence by Early Recruitment of Neutrophils to the Lungs during Pneumonic Plague.Spatially distinct neutrophil responses within the inflammatory lesions of pneumonic plagueDissociation of Tissue Destruction and Bacterial Expansion during Bubonic Plague.Early emergence of Yersinia pestis as a severe respiratory pathogenOn the in vivo significance of bacterial resistance to antimicrobial peptides.Two Isoforms of Yersinia pestis Plasminogen Activator Pla: Intraspecies Distribution, Intrinsic Disorder Propensity, and Contribution to Virulence.Impact of the Pla protease substrate α2-antiplasmin on the progression of primary pneumonic plague.Inactivation of Peroxiredoxin 6 by the Pla Protease of Yersinia pestis.Yersinia versus host immunity: how a pathogen evades or triggers a protective responseManipulation of Interleukin-1β and Interleukin-18 Production by Yersinia pestis Effectors YopJ and YopM and Redundant Impact on Virulence.Activation and Evasion of Inflammasomes by Yersinia.Cell-Extrinsic TNF Collaborates with TRIF Signaling To Promote Yersinia-Induced Apoptosis.Proteolysis of plasminogen activator inhibitor-1 by Yersinia pestis remodulates the host environment to promote virulenceAntimicrobial Peptide Conformation as a Structural Determinant of Omptin Protease SpecificityInhibition of outer membrane proteases of the omptin family by aprotinin.Uropathogenic Escherichia coli Epigenetically Manipulate Host Cell Death Pathways.Depletion of Glucose Activates Catabolite Repression during Pneumonic Plague.
P2860
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P2860
The Pla protease of Yersinia pestis degrades fas ligand to manipulate host cell death and inflammation
description
2014 nî lūn-bûn
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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2014 թվականի ապրիլին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
The Pla protease of Yersinia p ...... st cell death and inflammation
@ast
The Pla protease of Yersinia p ...... st cell death and inflammation
@en
type
label
The Pla protease of Yersinia p ...... st cell death and inflammation
@ast
The Pla protease of Yersinia p ...... st cell death and inflammation
@en
prefLabel
The Pla protease of Yersinia p ...... st cell death and inflammation
@ast
The Pla protease of Yersinia p ...... st cell death and inflammation
@en
P2093
P2860
P1433
P1476
The Pla protease of Yersinia p ...... st cell death and inflammation
@en
P2093
Adam J Caulfield
Lindsay M Gielda
Margaret E Walker
P2860
P304
P356
10.1016/J.CHOM.2014.03.005
P577
2014-04-01T00:00:00Z