about
The Pla protease of Yersinia pestis degrades fas ligand to manipulate host cell death and inflammationProduction of outer membrane vesicles by the plague pathogen Yersinia pestisProgression of primary pneumonic plague: a mouse model of infection, pathology, and bacterial transcriptional activityA plasminogen-activating protease specifically controls the development of primary pneumonic plague.RovA, a global regulator of Yersinia pestis, specifically required for bubonic plague.Disruption of fas-fas ligand signaling, apoptosis, and innate immunity by bacterial pathogensEarly emergence of Yersinia pestis as a severe respiratory pathogenImpact of the Pla protease substrate α2-antiplasmin on the progression of primary pneumonic plague.Hfq-dependent, co-ordinate control of cyclic diguanylate synthesis and catabolism in the plague pathogen Yersinia pestisInactivation of Peroxiredoxin 6 by the Pla Protease of Yersinia pestis.Draft Genome Sequence of a Multidrug-Resistant Klebsiella quasipneumoniae subsp. similipneumoniae Isolate from a Clinical Source.Posttranscriptional regulation of the Yersinia pestis cyclic AMP receptor protein Crp and impact on virulence.Proteolysis of plasminogen activator inhibitor-1 by Yersinia pestis remodulates the host environment to promote virulenceStcE, a metalloprotease secreted by Escherichia coli O157:H7, specifically cleaves C1 esterase inhibitor.Acquisition of stcE, a C1 esterase inhibitor-specific metalloprotease, during the evolution of Escherichia coli O157:H7.Depletion of Glucose Activates Catabolite Repression during Pneumonic Plague.
P50
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P50
description
researcher
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wetenschapper
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հետազոտող
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
@nl
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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Wyndham W Lathem
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