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Oxidants and antioxidants in sulfur mustard-induced injury

Oxidants and antioxidants in sulfur mustard-induced injury

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Silibinin attenuates sulfur mustard analog-induced skin injury by targeting multiple pathways connecting oxidative stress and inflammation Pathogenesis and treatment of skin lesions caused by sulfur mustard.Comparative transcriptional and translational analysis of heme oxygenase expression in response to sulfur mustard.Myeloperoxidase deficiency attenuates nitrogen mustard-induced skin injuries.Oxidative stress resulting from exposure of a human salivary gland cells to paraoxon: an in vitro model for organophosphate oral exposure.Catalytic antioxidant AEOL 10150 treatment ameliorates sulfur mustard analog 2-chloroethyl ethyl sulfide-associated cutaneous toxic effects.Protective effects of erythropoietin and N-acetylcysteine on methotrexate-induced lung injury in rats.Structural changes in hair follicles and sebaceous glands of hairless mice following exposure to sulfur mustard.The generation of 4-hydroxynonenal, an electrophilic lipid peroxidation end product, in rabbit cornea organ cultures treated with UVB light and nitrogen mustard.Modulation of keratinocyte expression of antioxidants by 4-hydroxynonenal, a lipid peroxidation end product.Expression of proliferative and inflammatory markers in a full-thickness human skin equivalent following exposure to the model sulfur mustard vesicant, 2-chloroethyl ethyl sulfide Pentoxifylline attenuates nitrogen mustard-induced acute lung injury, oxidative stress and inflammation.Antioxidant effects of the ethanol extract from flower of Camellia japonica via scavenging of reactive oxygen species and induction of antioxidant enzymes.Sulfur mustard analog, 2-chloroethyl ethyl sulfide-induced skin injury involves DNA damage and induction of inflammatory mediators, in part via oxidative stress, in SKH-1 hairless mouse skin.Mechanism underlying acute lung injury due to sulfur mustard exposure in rats.Flavanone silibinin treatment attenuates nitrogen mustard-induced toxic effects in mouse skin Nitrogen mustard exposure of murine skin induces DNA damage, oxidative stress and activation of MAPK/Akt-AP1 pathway leading to induction of inflammatory and proteolytic mediators.Coenzyme Q(1) as a probe for mitochondrial complex I activity in the intact perfused hyperoxia-exposed wild-type and Nqo1-null mouse lung.The hepatoprotective activity of olive oil and Nigella sativa oil against CCl4 induced hepatotoxicity in male rats.The Mixture of Salvianolic Acids from Salvia miltiorrhiza and Total Flavonoids from Anemarrhena asphodeloides Attenuate Sulfur Mustard-Induced Injury Attenuation of Nitrogen Mustard-Induced Pulmonary Injury and Fibrosis by Anti-Tumor Necrosis Factor-α Antibody.Protective role of spleen-derived macrophages in lung inflammation, injury, and fibrosis induced by nitrogen mustard.Nitrogen Mustard-Induced Corneal Injury Involves DNA Damage and Pathways Related to Inflammation, Epithelial-Stromal Separation, and Neovascularization.Attenuation of acute nitrogen mustard-induced lung injury, inflammation and fibrogenesis by a nitric oxide synthase inhibitor Role of TNFR1 in lung injury and altered lung function induced by the model sulfur mustard vesicant, 2-chloroethyl ethyl sulfide.Ablation of ceramide synthase 2 causes chronic oxidative stress due to disruption of the mitochondrial respiratory chain Regulation of Hsp27 and Hsp70 expression in human and mouse skin construct models by caveolae following exposure to the model sulfur mustard vesicant, 2-chloroethyl ethyl sulfide Mechanisms of sulfur mustard analog 2-chloroethyl ethyl sulfide-induced DNA damage in skin epidermal cells and fibroblasts Mustard vesicating agent-induced toxicity in the skin tissue and silibinin as a potential countermeasure.Investigation of anticholinergic and non-steroidal anti-inflammatory prodrugs which reduce chemically induced skin inflammation.Emerging targets for treating sulfur mustard-induced injuries.Macrophages and inflammatory mediators in pulmonary injury induced by mustard vesicants.Management of sulfur mustard-induced chronic pruritus: a review of clinical trials.Sulfur mustard and respiratory diseases.Treatment for sulfur mustard lung injuries; new therapeutic approaches from acute to chronic phase A clinicopathological approach to sulfur mustard-induced organ complications: a major review.Mustard Gas Inhalation Injury: Therapeutic Strategy.Role of oxidative stress in sulfur mustard-induced pulmonary injury and antioxidant protection.The footprint of TGF-β in airway remodeling of the mustard lung.Mustard vesicant-induced lung injury: Advances in therapy.

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Oxidants and antioxidants in sulfur mustard-induced injury

http://www.wikidata.org/entity/Q33621786

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2010 nî lūn-bûn

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2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2010 թվականի օգոստոսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Oxidants and antioxidants in sulfur mustard-induced injury

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Oxidants and antioxidants in sulfur mustard-induced injury

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Oxidants and antioxidants in sulfur mustard-induced injury

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J.1749-6632.2010.05605.X

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Annals of the New York Academy of Sciences

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Oxidants and antioxidants in sulfur mustard-induced injury

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P2093

Adrienne T Black

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Debra L Laskin

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Diane E Heck

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Jeffrey D Laskin

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Ned D Heindel

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Vasanthi Sunil

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Yi-Hua Jan

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P2860

Selective targeting of selenocysteine in thioredoxin reductase by the half mustard 2-chloroethyl ethyl sulfide in lung epithelial cells

Inhibition of inducible Nitric Oxide Synthase by a mustard gas analog in murine macrophages.

Sulfur mustard analog induces oxidative stress and activates signaling cascades in the skin of SKH-1 hairless mice.

Mechanisms mediating the vesicant actions of sulfur mustard after cutaneous exposure

Prooxidant and antioxidant functions of nitric oxide in liver toxicity.

Nitric oxide and peroxynitrite in ozone-induced lung injury.

Toxicology and pharmacology of the chemical warfare agent sulfur mustard.

Protection by antioxidants against toxicity and apoptosis induced by the sulphur mustard analog 2-chloroethylethyl sulphide (CEES) in Jurkat T cells and normal human lymphocytes.

Inflammatory mediators and modulators released in organ culture from rabbit skin lesions produced in vivo by sulfur mustard. I. Quantitative histopathology; PMN, basophil, and mononuclear cell survival; and unbound (serum) protein content.

Inhibition of NADPH cytochrome P450 reductase by the model sulfur mustard vesicant 2-chloroethyl ethyl sulfide is associated with increased production of reactive oxygen species.

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92-100

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10.1111/J.1749-6632.2010.05605.X

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1203

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Patrick J Sinko

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45694318

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