about
52-kDa Ro/SSA epitopes preferentially recognized by antibodies from mothers of children with neonatal lupus and congenital heart blockStructural organization and Zn2+-dependent subdomain interactions involving autoantigenic epitopes in the Ring-B-box-coiled-coil (RBCC) region of Ro52Self protection from anti-viral responses--Ro52 promotes degradation of the transcription factor IRF7 downstream of the viral Toll-Like receptors.The E3 ubiquitin ligase Ro52 negatively regulates IFN-beta production post-pathogen recognition by polyubiquitin-mediated degradation of IRF3Extraordinary antigenicity of the human Ro52 autoantigen.Key sequences involved in the spreading of the systemic autoimmune response to spliceosomal proteins.Autoantibodies in primary Sjögren's syndrome: new insights into mechanisms of autoantibody diversification and disease pathogenesis.Epitope spreading in systemic lupus erythematosus: identification of triggering peptide sequences.Anti-Ro52 autoantibodies from patients with Sjögren's syndrome inhibit the Ro52 E3 ligase activity by blocking the E3/E2 interface.Clinical, immunological, and immunogenetic aspects of autoantibody production against Ro/SSA, La/SSB and their linear epitopes in primary Sjögren's syndrome (pSS): a European multicentre study.Sensitive and robust luminescent profiling of anti-La and other autoantibodies in Sjogren's syndrome.Ro/SSA autoantibodies directly bind cardiomyocytes, disturb calcium homeostasis, and mediate congenital heart block.Autoantibodies to intracellular autoantigens and their B-cell epitopes: molecular probes to study the autoimmune response.Sjögren's syndrome associated with systemic lupus erythematosus.Rapid serological detection of autoantibodies associated with Sjögren's syndrome.Immunoglobulin variable genes and epitope recognition of human monoclonal anti-Ro 52-kd in primary Sjögren's syndrome.H1N1 vaccination in Sjögren's syndrome triggers polyclonal B cell activation and promotes autoantibody production.The 52 000 MW Ro/SS-A autoantigen in Sjögren's syndrome/systemic lupus erythematosus (Ro52) is an interferon-gamma inducible tripartite motif protein associated with membrane proximal structures.Structurally derived mutations define congenital heart block-related epitopes within the 200-239 amino acid stretch of the Ro52 protein.Cellular basis of ectopic germinal center formation and autoantibody production in the target organ of patients with Sjögren's syndrome.Molecular signature of a public clonotypic autoantibody in primary Sjögren's syndrome: a "forbidden" clone in systemic autoimmunity.Reactivity with dichotomous determinants of Ro 60 stratifies autoantibody responses in lupus and primary Sjögren's syndrome
P2860
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P2860
description
1999 nî lūn-bûn
@nan
1999 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@ast
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@en
type
label
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@ast
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@en
prefLabel
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@ast
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@en
P1433
P1476
Analysis of B-cell epitopes of the Ro/SS-A autoantigen.
@en
P2093
P304
P356
10.1016/S0167-5699(99)01458-9
P577
1999-05-01T00:00:00Z