No contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure overload hypertrophy.
about
Calcineurin-NFATc regulates type 2 inositol 1,4,5-trisphosphate receptor (InsP3R2) expression during cardiac remodeling.Cardiac inositol 1,4,5-trisphosphate receptors.Loss of IP3 Receptor-Mediated Ca2+ Release in Mouse B Cells Results in Abnormal B Cell Development and Function.Functionally redundant control of cardiac hypertrophic signaling by inositol 1,4,5-trisphosphate receptors.
P2860
No contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure overload hypertrophy.
description
2012 nî lūn-bûn
@nan
2012 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
No contribution of IP3-R(2) to ...... pressure overload hypertrophy.
@ast
No contribution of IP3-R(2) to ...... pressure overload hypertrophy.
@en
type
label
No contribution of IP3-R(2) to ...... pressure overload hypertrophy.
@ast
No contribution of IP3-R(2) to ...... pressure overload hypertrophy.
@en
prefLabel
No contribution of IP3-R(2) to ...... pressure overload hypertrophy.
@ast
No contribution of IP3-R(2) to ...... pressure overload hypertrophy.
@en
P2093
P2860
P1476
No contribution of IP3-R(2) to ...... pressure overload hypertrophy
@en
P2093
Elizabeth A Woodcock
Farah Sheikh
Helen Kiriazis
Julie R McMullen
Kunfu Ouyang
Nicola Cooley
Xiao-Jun Du
P2860
P304
P356
10.1161/CIRCHEARTFAILURE.112.972158
P50
P577
2012-12-20T00:00:00Z