Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes. - Wikidata - awgldk - TriplyDB

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

http://www.wikidata.org/entity/Q33906288

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STIM proteins: dynamic calcium signal transducers Store-operated Ca2+ entry in muscle physiology and diseases Store-operated calcium entry and diabetic complications Orai1 deficiency leads to heart failure and skeletal myopathy in zebrafish High glucose and diabetes enhanced store-operated Ca(2+) entry and increased expression of its signaling proteins in mesangial cells.Gastrodin Inhibits Store-Operated Ca2+ Entry and Alleviates Cardiac Hypertrophy.No contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure overload hypertrophy.The calcium release-activated calcium channel Orai1 represents a crucial component in hypertrophic compensation and the development of dilated cardiomyopathy.Protein O-GlcNAcylation and cardiovascular (patho)physiology.Expression and localization of TRPC proteins in rat ventricular myocytes at various developmental stages.STIM1 and Orai1: novel targets for vascular diseases?Stim and Orai proteins in neuronal Ca(2+) signaling and excitability STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophy.A store-operated calcium channel inhibitor attenuates collagen-induced arthritis.Hypertrophy in skeletal myotubes induced by junctophilin-2 mutant, Y141H, involves an increase in store-operated Ca2+ entry via Orai1.STIM1 enhances SR Ca2+ content through binding phospholamban in rat ventricular myocytes.STIM1-Ca2+ signaling modulates automaticity of the mouse sinoatrial node.Critical role for stromal interaction molecule 1 in cardiac hypertrophy Store-Operated Ca2+ Channels in Mesangial Cells Inhibit Matrix Protein Expression.STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytes Differential roles of the C and N termini of Orai1 protein in interacting with stromal interaction molecule 1 (STIM1) for Ca2+ release-activated Ca2+ (CRAC) channel activation Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling.Altered sarcoplasmic reticulum calcium cycling--targets for heart failure therapy.STIM1-dependent Ca(2+) microdomains are required for myofilament remodeling and signaling in the heart.Transient Receptor Potential Canonical (TRPC)/Orai1-dependent Store-operated Ca2+ Channels: NEW TARGETS OF ALDOSTERONE IN CARDIOMYOCYTES.STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function.Enhanced store-operated Ca2+ influx and ORAI1 expression in ventricular fibroblasts from human failing heart.Transcriptional mechanisms regulating Ca(2+) homeostasis.Roles for the sarco-/endoplasmic reticulum in cardiac myocyte contraction, protein synthesis, and protein quality control.What role for store-operated Ca²⁺ entry in muscle?Emerging roles for native Orai Ca2+ channels in cardiovascular disease ADP-Induced Ca2+ Signaling and Proliferation of Rat Ventricular Myofibroblasts Depend on Phospholipase C-Linked TRP Channels Activation Within Lipid Rafts.Calcium and Excitation-Contraction Coupling in the Heart Store-operated calcium entry is present in HL-1 cardiomyocytes and contributes to resting calcium.Differential role for stromal interacting molecule 1 in the regulation of vascular function.Essential Role of Smooth Muscle STIM1 in Hypertension and Cardiovascular Dysfunction.Long-term administration of pyridostigmine attenuates pressure overload-induced cardiac hypertrophy by inhibiting calcineurin signalling.Emergence of Orai3 activity during cardiac hypertrophy.

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P2860

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

http://www.wikidata.org/entity/Q33906288

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@ast

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@en

type

label

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@ast

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@en

prefLabel

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@ast

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@en

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J.YJMCC.2010.01.020

P1433

Journal of Molecular and Cellular Cardiology

P1476

Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.

@en

P2093

Derk Frank

http://www.w3.org/2001/XMLSchema#string

Hugo A Katus

http://www.w3.org/2001/XMLSchema#string

Mareen Salz

http://www.w3.org/2001/XMLSchema#string

Mark A Sussman

http://www.w3.org/2001/XMLSchema#string

Mirko Voelkers

http://www.w3.org/2001/XMLSchema#string

Natalie Gude

http://www.w3.org/2001/XMLSchema#string

Nicole Herzog

http://www.w3.org/2001/XMLSchema#string

Nima Dolatabadi

http://www.w3.org/2001/XMLSchema#string

Norbert Frey

http://www.w3.org/2001/XMLSchema#string

Oliver Friedrich

http://www.w3.org/2001/XMLSchema#string

P2860

Orai1 is an essential pore subunit of the CRAC channel

Extracellular S100A1 protein inhibits apoptosis in ventricular cardiomyocytes via activation of the extracellular signal-regulated protein kinase 1/2 (ERK1/2)

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study

Cardiac hypertrophy: the good, the bad, and the ugly

Hair loss and defective T- and B-cell function in mice lacking ORAI1

STIMulating store-operated Ca(2+) entry.

TRPC1 channels are critical for hypertrophic signaling in the heart.

Cardiac calcium signalling.

Roles of TRP channels in the development of cardiac hypertrophy.

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1329-1334

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scholarly article

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10.1016/J.YJMCC.2010.01.020

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6

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48

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2010-02-06T00:00:00Z

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41413400

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20138887

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5511312

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