Upregulation of early growth response factor-1 by bile acids requires mitogen-activated protein kinase signaling.
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Bile acid stimulates hepatocyte polarization through a cAMP-Epac-MEK-LKB1-AMPK pathway.Toxicogenomic analysis reveals profibrogenic effects of trichloroethylene in autoimmune-mediated cholangitis in miceCigarette smoke-induced pulmonary inflammatory responses are mediated by EGR-1/GGPPS/MAPK signaling.Fibrinogen deficiency increases liver injury and early growth response-1 (Egr-1) expression in a model of chronic xenobiotic-induced cholestasis.Bile acids induce inflammatory genes in hepatocytes: a novel mechanism of inflammation during obstructive cholestasis.Increased activation of the Wnt/β-catenin pathway in spontaneous hepatocellular carcinoma observed in farnesoid X receptor knockout mice.Diurnal regulation of the early growth response 1 (Egr-1) protein expression by hepatocyte nuclear factor 4alpha (HNF4alpha) and small heterodimer partner (SHP) cross-talk in liver fibrosis.Early growth response factor-1 limits biliary fibrosis in a model of xenobiotic-induced cholestasis in mice.Differential Feedback Regulation of Δ4-3-Oxosteroid 5β-Reductase Expression by Bile Acids.Novel insight into mechanisms of cholestatic liver injury.Therapeutic targets for cholestatic liver injuryPharmacology of bile acid receptors: Evolution of bile acids from simple detergents to complex signaling molecules.Bile acids promote diethylnitrosamine-induced hepatocellular carcinoma via increased inflammatory signaling.Osteopontin is an initial mediator of inflammation and liver injury during obstructive cholestasis after bile duct ligation in mice.Mechanisms of STAT3 activation in the liver of FXR knockout mice.Trovafloxacin potentiation of lipopolysaccharide-induced tumor necrosis factor release from RAW 264.7 cells requires extracellular signal-regulated kinase and c-Jun N-Terminal Kinase.Interactions of flavan-3-ols and procyanidins with membranes: mechanisms and the physiological relevance.Mechanisms of bile acid mediated inflammation in the liver.The FXR Agonist, Obeticholic Acid, Suppresses HCC Proliferation & Metastasis: Role of IL-6/STAT3 Signalling Pathway.E2F1 is a novel fibrogenic gene that regulates cholestatic liver fibrosis through the Egr-1/SHP/EID1 network.IL-17A synergistically enhances bile acid-induced inflammation during obstructive cholestasis.Chenodeoxycholic acid increases the induction of CYP1A1 in HepG2 and H4IIE cells.GW4064 attenuates lipopolysaccharide‑induced hepatic inflammation and apoptosis through inhibition of the Toll‑like receptor 4‑mediated p38 mitogen‑activated protein kinase signaling pathway in mice.
P2860
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P2860
Upregulation of early growth response factor-1 by bile acids requires mitogen-activated protein kinase signaling.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Upregulation of early growth r ...... ated protein kinase signaling.
@ast
Upregulation of early growth r ...... ated protein kinase signaling.
@en
type
label
Upregulation of early growth r ...... ated protein kinase signaling.
@ast
Upregulation of early growth r ...... ated protein kinase signaling.
@en
prefLabel
Upregulation of early growth r ...... ated protein kinase signaling.
@ast
Upregulation of early growth r ...... ated protein kinase signaling.
@en
P2093
P2860
P1476
Upregulation of early growth r ...... ated protein kinase signaling.
@en
P2093
Bryan L Copple
Jeon-Ok Moon
Katryn Allen
Nam Deuk Kim
P2860
P356
10.1016/J.TAAP.2009.11.013
P577
2009-11-17T00:00:00Z