Id2, Id3 and Id4 overcome a Smad7-mediated block in tumorigenesis, generating TGF-β-independent melanoma. - Wikidata - awgldk - TriplyDB

Id2, Id3 and Id4 overcome a Smad7-mediated block in tumorigenesis, generating TGF-β-independent melanoma.

Id2, Id3 and Id4 overcome a Smad7-mediated block in tumorigenesis, generating TGF-β-independent melanoma.

http://www.wikidata.org/entity/Q33652300

about

Inhibitor of DNA Binding 4 (ID4) is highly expressed in human melanoma tissues and may function to restrict normal differentiation of melanoma cells Id3 induces an Elk-1-caspase-8-dependent apoptotic pathway in squamous carcinoma cells Inhibitor of differentiation-4 (Id4) stimulates pigmentation in melanoma leading to histiocyte infiltration.Alpha-1-antitrypsin functions as a protective factor in preeclampsia through activating Smad2 and inhibitor of DNA binding 4.Id2 Collaborates with Id3 To Suppress Invariant NKT and Innate-like Tumors.New susceptibility loci for cutaneous melanoma risk and progression revealed using a porcine model.Hyaluronic acid-CD44 interactions promote BMP4/7-dependent Id1/3 expression in melanoma cells

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Id2, Id3 and Id4 overcome a Smad7-mediated block in tumorigenesis, generating TGF-β-independent melanoma.

http://www.wikidata.org/entity/Q33652300

description

2013 nî lūn-bûn

@nan

2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

@ast

Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

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type

label

Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

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Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

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prefLabel

Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

@ast

Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

@en

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Id2, Id3 and Id4 overcome a Sm ...... ng TGF-β-independent melanoma.

@en

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Cynthia M Simbulan-Rosenthal

http://www.w3.org/2001/XMLSchema#string

Dean S Rosenthal

http://www.w3.org/2001/XMLSchema#string

Kyle A DiVito

http://www.w3.org/2001/XMLSchema#string

Valerie A Trabosh

http://www.w3.org/2001/XMLSchema#string

You-Shin Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts

Regulation of transforming growth factor-beta signaling by protein inhibitor of activated STAT, PIASy through Smad3

Id proteins in development, cell cycle and cancer

The Id proteins and angiogenesis

Id genes and proteins as promising targets in cancer therapy

Interactions between ID and OLIG proteins mediate the inhibitory effects of BMP4 on oligodendroglial differentiation

Inhibitor of differentiation 4 (Id4) is a potential tumor suppressor in prostate cancer.

Targeting Id1 and Id3 by a specific peptide aptamer induces E-box promoter activity, cell cycle arrest, and apoptosis in breast cancer cells.

Id2 is a retinoblastoma protein target and mediates signalling by Myc oncoproteins.

A model for melanosome biogenesis based on the purification and analysis of early melanosomes

P304

951-958

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scholarly article

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10.1093/CARCIN/BGT479

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4

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35

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259349905

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24343358

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4031572

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