Inhibition of osteoclast bone resorption by disrupting vacuolar H+-ATPase a3-B2 subunit interaction.
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Probing subunit-subunit interactions in the yeast vacuolar ATPase by peptide arraysHuman genetics in rheumatoid arthritis guides a high-throughput drug screen of the CD40 signaling pathwayThe diabetes susceptibility gene Clec16a regulates mitophagyLuteolin inhibition of V-ATPase a3-d2 interaction decreases osteoclast resorptive activity.The Sirt1 Activators SRT2183 and SRT3025 Inhibit RANKL-Induced Osteoclastogenesis in Bone Marrow-Derived Macrophages and Down-Regulate Sirt3 in Sirt1 Null CellsPerturbation of the Vacuolar ATPase: A NOVEL CONSEQUENCE OF INOSITOL DEPLETIONInside job: ligand-receptor pharmacology beneath the plasma membrane.Targeting reversible disassembly as a mechanism of controlling V-ATPase activityNovel techniques in the development of osteoporosis drug therapy: the osteoclast ruffled-border vacuolar H(+)-ATPase as an emerging target.The Cdc42 guanine nucleotide exchange factor FGD6 coordinates cell polarity and endosomal membrane recycling in osteoclasts.Effect of Allium cepa L. on Lipopolysaccharide-Stimulated Osteoclast Precursor Cell Viability, Count, and Morphology Using 4',6-Diamidino-2-phenylindole-Staining.V-ATPase promotes transforming growth factor-β-induced epithelial-mesenchymal transition of rat proximal tubular epithelial cells.N-Linked Glycosylation Is Required for Vacuolar H+ -ATPase (V-ATPase) a4 Subunit Stability, Assembly, and Cell Surface Expression.Allium cepa L. and Quercetin Inhibit RANKL/Porphyromonas gingivalis LPS-Induced Osteoclastogenesis by Downregulating NF-κB Signaling Pathway.Interferon-inducible transmembrane proteins of the innate immune response act as membrane organizers by influencing clathrin and v-ATPase localization and function.Ammonia-independent sodium uptake mediated by Na+ channels and NHEs in the freshwater ribbon leech Nephelopsis obscura.SNX10 Plays a Critical Role in MMP9 Secretion via JNK-p38-ERK Signaling Pathway.IL-15 and RANKL Play a Synergistically Important Role in Osteoclastogenesis.Topology, glycosylation and conformational changes in the membrane domain of the vacuolar H+-ATPase a subunit.Deficiency of sorting nexin 10 prevents bone erosion in collagen-induced mouse arthritis through promoting NFATc1 degradation.Vacuolar H+-ATPase: An Essential Multitasking Enzyme in Physiology and Pathophysiology
P2860
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P2860
Inhibition of osteoclast bone resorption by disrupting vacuolar H+-ATPase a3-B2 subunit interaction.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Inhibition of osteoclast bone ...... ase a3-B2 subunit interaction.
@ast
Inhibition of osteoclast bone ...... ase a3-B2 subunit interaction.
@en
type
label
Inhibition of osteoclast bone ...... ase a3-B2 subunit interaction.
@ast
Inhibition of osteoclast bone ...... ase a3-B2 subunit interaction.
@en
prefLabel
Inhibition of osteoclast bone ...... ase a3-B2 subunit interaction.
@ast
Inhibition of osteoclast bone ...... ase a3-B2 subunit interaction.
@en
P2093
P2860
P356
P1476
Inhibition of osteoclast bone ...... Pase a3-B2 subunit interaction
@en
P2093
Gazelle J Crasto
Morris F Manolson
Norbert Kartner
P2860
P304
37476-37490
P356
10.1074/JBC.M110.123281
P407
P577
2010-09-13T00:00:00Z