Susceptibility of germfree phagocyte oxidase- and nitric oxide synthase 2-deficient mice, defective in the production of reactive metabolites of both oxygen and nitrogen, to mucosal and systemic candidiasis of endogenous origin.
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NADPH oxidase-driven phagocyte recruitment controls Candida albicans filamentous growth and prevents mortalityLive imaging of disseminated candidiasis in zebrafish reveals role of phagocyte oxidase in limiting filamentous growth.Candida albicans induces arginine biosynthetic genes in response to host-derived reactive oxygen species.Transcriptional response of Candida albicans to nitric oxide and the role of the YHB1 gene in nitrosative stress and virulence.Chemokine receptor Ccr1 drives neutrophil-mediated kidney immunopathology and mortality in invasive candidiasisAbsence of phagocyte NADPH oxidase 2 leads to severe inflammatory response in lungs of mice infected with Coccidioides.Candida albicans Pathogenesis: Fitting within the Host-Microbe Damage Response Framework.Variation in Candida albicans EFG1 expression enables host-dependent changes in colonizing fungal populations.Candida albicans biofilms do not trigger reactive oxygen species and evade neutrophil killing.Normal adaptation of Candida albicans to the murine gastrointestinal tract requires Efg1p-dependent regulation of metabolic and host defense genes.The game theory of Candida albicans colonization dynamics reveals host status-responsive gene expression.Live Candida albicans suppresses production of reactive oxygen species in phagocytes.Effect of Antifungal Treatment in a Diet-Based Murine Model of Disseminated Candidiasis Acquired via the Gastrointestinal Tract.Innate antifungal immunity: the key role of phagocytes.Innate and adaptive antifungal immune responses: partners on an equal footing.Oxidative and nitrosative stress on phagocytes' function: from effective defense to immunity evasion mechanisms.Murine models of Candida gastrointestinal colonization and dissemination.Beyond Candida albicans: Mechanisms of immunity to non-albicans Candida speciesPhagocytosis and nitric oxide production by peritoneal adherent cells in response to Candida albicans in aging: a collaboration to elucidate the pathogenesis of denture stomatitis.Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii.Glutathione levels in and total antioxidant capacity of Candida sp. cells exposed to oxidative stress caused by hydrogen peroxide.
P2860
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P2860
Susceptibility of germfree phagocyte oxidase- and nitric oxide synthase 2-deficient mice, defective in the production of reactive metabolites of both oxygen and nitrogen, to mucosal and systemic candidiasis of endogenous origin.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@ast
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@en
type
label
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@ast
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@en
prefLabel
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@ast
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@en
P2093
P2860
P1476
Susceptibility of germfree pha ...... didiasis of endogenous origin.
@en
P2093
Caroline Westwater
David A Schofield
Edward Balish
Emily E Paulling
Peter J Nicholas
Thomas F Warner
P2860
P304
P356
10.1128/IAI.73.3.1313-1320.2005
P407
P577
2005-03-01T00:00:00Z