Candida albicans biofilms do not trigger reactive oxygen species and evade neutrophil killing. - Wikidata - awgldk - TriplyDB

Candida albicans biofilms do not trigger reactive oxygen species and evade neutrophil killing.

Candida albicans biofilms do not trigger reactive oxygen species and evade neutrophil killing.

http://www.wikidata.org/entity/Q36412239

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Plasticity of Candida albicans Biofilms The Host's Reply to Candida Biofilm Bacterial Vaginosis Biofilms: Challenges to Current Therapies and Emerging Solutions Integration of Posttranscriptional Gene Networks into Metabolic Adaptation and Biofilm Maturation in Candida albicans The Extracellular Matrix of Candida albicans Biofilms Impairs Formation of Neutrophil Extracellular Traps The Candida albicans Biofilm Matrix: Composition, Structure and Function Streptococcus gordonii comCDE (competence) operon modulates biofilm formation with Candida albicans Fungal Biofilms: In Vivo Models for Discovery of Anti-Biofilm Drugs Fungal Super Glue: The Biofilm Matrix and Its Composition, Assembly, and Functions.Host contributions to construction of three device-associated Candida albicans biofilms Control of Candida albicans metabolism and biofilm formation by Pseudomonas aeruginosa phenazines.Candida albicans pathogenicity mechanisms.Candida albicans biofilms: development, regulation, and molecular mechanisms.Biofilm Exopolysaccharides of Pathogenic Fungi: Lessons from Bacteria.Streptococcal co-infection augments Candida pathogenicity by amplifying the mucosal inflammatory response.Vaginal Heparan Sulfate Linked to Neutrophil Dysfunction in the Acute Inflammatory Response Associated with Experimental Vulvovaginal Candidiasis.Thriving within the host: Candida spp. interactions with phagocytic cells.Recent insights into Candida albicans biofilm resistance mechanisms Characterization of biofilm formation and the role of BCR1 in clinical isolates of Candida parapsilosis.Automation of serum (1→3)-beta-D-glucan testing allows reliable and rapid discrimination of patients with and without candidemia.Microbial Biofilms in Pulmonary and Critical Care Diseases.Conserved Inhibition of Neutrophil Extracellular Trap Release by Clinical Candida albicans Biofilms.Mechanisms involved in the triggering of neutrophil extracellular traps (NETs) by Candida glabrata during planktonic and biofilm growth.Protocol for Identifying Natural Agents That Selectively Affect Adhesion, Thickness, Architecture, Cellular Phenotypes, Extracellular Matrix, and Human White Blood Cell Impenetrability of Candida albicans Biofilms.Inactivation of genes TEC1 and EFG1 in Candida albicans influences extracellular matrix composition and biofilm morphology Development and regulation of single- and multi-species Candida albicans biofilms.Oxidative and nitrosative stress responses during macrophage-Candida albicans biofilm interaction.Fungal Strategies to Evade the Host Immune Recognition.Immune Recognition of Fungal Polysaccharides.Negative regulation of filamentous growth in Candida albicans by Dig1p.The Interface between Fungal Biofilms and Innate Immunity.Macrophage Migration Is Impaired within Candida albicans Biofilms.Chemotherapy-induced oral mucositis and associated infections in a novel organotypic model.Neutrophil extracellular traps in fungal infection.Impact of oxidative and osmotic stresses on Candida albicans biofilm formation.Community Development between Porphyromonas gingivalis and Candida albicans Mediated by InlJ and Als3.

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P2860

Candida albicans biofilms do not trigger reactive oxygen species and evade neutrophil killing.

http://www.wikidata.org/entity/Q36412239

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2012 nî lūn-bûn

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2012年学术文章

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Candida albicans biofilms do n ...... and evade neutrophil killing.

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Candida albicans biofilms do n ...... and evade neutrophil killing.

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Candida albicans biofilms do n ...... and evade neutrophil killing.

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Candida albicans biofilms do n ...... and evade neutrophil killing.

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The Journal of Infectious Diseases

P1476

Candida albicans biofilms do n ...... and evade neutrophil killing.

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P2093

Angela Thompson

http://www.w3.org/2001/XMLSchema#string

Anna Dongari-Bagtzoglou

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Helena Kashleva

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Hongbin Xu

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John Vasilakos

http://www.w3.org/2001/XMLSchema#string

Takanori Sobue

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Zhihong Xie

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P2860

Prostaglandin production during growth of Candida albicans biofilms

Dectin-1 is a major beta-glucan receptor on macrophages

Characterization of mucosal Candida albicans biofilms

Interaction of Candida albicans with adherent human peripheral blood mononuclear cells increases C. albicans biofilm formation and results in differential expression of pro- and anti-inflammatory cytokines.

Compromised host defense on Pseudomonas aeruginosa biofilms: characterization of neutrophil and biofilm interactions.

Host defence against Staphylococcus aureus biofilms infection: phagocytosis of biofilms by polymorphonuclear neutrophils (PMN).

Goa1p of Candida albicans localizes to the mitochondria during stress and is required for mitochondrial function and virulence.

Susceptibility of germfree phagocyte oxidase- and nitric oxide synthase 2-deficient mice, defective in the production of reactive metabolites of both oxygen and nitrogen, to mucosal and systemic candidiasis of endogenous origin.

A quantitative real-time RT-PCR assay for mature C. albicans biofilms.

Genetic basis of Candida biofilm resistance due to drug-sequestering matrix glucan.

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1936-1945

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10.1093/INFDIS/JIS607

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12

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206

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2012-10-02T00:00:00Z

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231863881

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23033146

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3502374

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