MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis. - Wikidata - awgldk - TriplyDB

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

http://www.wikidata.org/entity/Q33731819

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Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and Apoptosis Endothelial to Mesenchymal Transition (EndoMT) in the Pathogenesis of Human Fibrotic Diseases Vascular Remodelling and Mesenchymal Transition in Systemic Sclerosis The fibrosis-cell death axis in heart failure MicroRNA Expression Signature in Human Calcific Aortic Valve Disease.Endothelial to mesenchymal transition contributes to arsenic-trioxide-induced cardiac fibrosis.The Living Scar--Cardiac Fibroblasts and the Injured Heart.Nutrition has a pervasive impact on cardiac microRNA expression in isogenic mice.Nogo-C regulates cardiomyocyte apoptosis during mouse myocardial infarction Reduction in miRNA-125b-5p levels is associated with obstructive renal injury.The epigenetic landscape related to reactive oxygen species formation in the cardiovascular system.MicroRNA-9 inhibits high glucose-induced proliferation, differentiation and collagen accumulation of cardiac fibroblasts by down-regulation of TGFBR2.MicroRNA as a Therapeutic Target in Cardiac Remodeling.Plasminogen Activator Inhibitor Type I Controls Cardiomyocyte Transforming Growth Factor-β and Cardiac Fibrosis.Analysis of primary microRNA loci from nascent transcriptomes reveals regulatory domains governed by chromatin architecture.Exacerbation of diabetic cardiac hypertrophy in OVE26 mice by angiotensin II is associated with JNK/c-Jun/miR-221-mediated autophagy inhibition.A carvedilol-responsive microRNA, miR-125b-5p protects the heart from acute myocardial infarction by repressing pro-apoptotic bak1 and klf13 in cardiomyocytes.MicroRNAs in Heart Failure, Cardiac Transplantation, and Myocardial Recovery: Biomarkers with Therapeutic Potential.Interleukin-10 Inhibits Bone Marrow Fibroblast Progenitor Cell-Mediated Cardiac Fibrosis in Pressure-Overloaded Myocardium.A novel acetyltransferase p300 inhibitor ameliorates hypertension-associated cardio-renal fibrosis.Rosmarinic acid attenuates cardiac fibrosis following long-term pressure overload via AMPKα/Smad3 signaling.CYP2J2 metabolites, epoxyeicosatrienoic acids, attenuate Ang II-induced cardiac fibrotic response by targeting Gα12/13.Recombinant Adeno-Associated Virus-Mediated Delivery of MicroRNA-21-3p Lowers Hypertension.A Potential Link Between Oxidative Stress and Endothelial-to-Mesenchymal Transition in Systemic Sclerosis Cancer driver mutations in endometriosis: Variations on the major theme of fibrogenesis

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MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

http://www.wikidata.org/entity/Q33731819

description

2015 nî lūn-bûn

@nan

2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

@wuu

name

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

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MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

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type

label

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

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MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

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prefLabel

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

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MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

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CIRCULATIONAHA.115.018174

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MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.

@en

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Aaron T Place

http://www.w3.org/2001/XMLSchema#string

Asish K Ghosh

http://www.w3.org/2001/XMLSchema#string

Douglas E Vaughan

http://www.w3.org/2001/XMLSchema#string

Rahul Rai

http://www.w3.org/2001/XMLSchema#string

Sheila B Murphy

http://www.w3.org/2001/XMLSchema#string

Suresh K Verma

http://www.w3.org/2001/XMLSchema#string

Varun Nagpal

http://www.w3.org/2001/XMLSchema#string

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A human gene that shows identity with the gene encoding the angiotensin receptor is located on chromosome 11

Control of cell cycle progression by c-Jun is p53 dependent

MicroRNAs: target recognition and regulatory functions

Apelin signaling antagonizes Ang II effects in mouse models of atherosclerosis

A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure

Myofibroblasts: trust your heart and let fate decide.

A cardiac microRNA governs systemic energy homeostasis by regulation of MED13

MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

Control of stress-dependent cardiac growth and gene expression by a microRNA

MicroRNA-133 controls cardiac hypertrophy

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291-301

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10.1161/CIRCULATIONAHA.115.018174

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