Induction of the SOS response increases the efficiency of global nucleotide excision repair of cyclobutane pyrimidine dimers, but not 6-4 photoproducts, in UV-irradiated Escherichia coli.
about
INO80 chromatin remodeling complex promotes the removal of UV lesions by the nucleotide excision repair pathwayAnswering the Call: Coping with DNA Damage at the Most Inopportune TimeRepair activity of base and nucleotide excision repair enzymes for guanine lesions induced by nitrosative stress.Comparative gene expression profiles following UV exposure in wild-type and SOS-deficient Escherichia coliUV-induced mutagenesis in Escherichia coli SOS response: a quantitative model.Nucleotide excision repair genes are expressed at low levels and are not detectably inducible in Caenorhabditis elegans somatic tissues, but their function is required for normal adult life after UVC exposureRecovery of DNA replication in UV-irradiated Escherichia coli requires both excision repair and recF protein function.A cut above: discovery of an alternative excision repair pathway in bacteria.Participation of recombination proteins in rescue of arrested replication forks in UV-irradiated Escherichia coli need not involve recombination.Mfd is required for rapid recovery of transcription following UV-induced DNA damage but not oxidative DNA damage in Escherichia coli.Inefficient replication reduces RecA-mediated repair of UV-damaged plasmids introduced into competent Escherichia coliHigh-throughput identification and rational design of synergistic small-molecule pairs for combating and bypassing antibiotic resistance.Photobiological Origins of the Field of Genomic Maintenance.Transcription coupled nucleotide excision repair in Escherichia coli can be affected by changing the arginine at position 529 of the beta subunit of RNA polymerase.UvrD helicase: an old dog with a new trick: how one step backward leads to many steps forward.The uvrA, uvrB and uvrC genes are required for repair of ultraviolet light induced DNA photoproducts in Halobacterium sp. NRC-1.Prioritizing the repair of DNA damage that is encountered by RNA polymerase.Historical perspective on the DNA damage response.Antagonism of ultraviolet-light mutagenesis by the methyl-directed mismatch-repair system of Escherichia coli.Simulating the temporal modulation of inducible DNA damage response in Escherichia coli.Recruitment of UvrBC complexes to UV-induced damage in the absence of UvrA increases cell survival.
P2860
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P2860
Induction of the SOS response increases the efficiency of global nucleotide excision repair of cyclobutane pyrimidine dimers, but not 6-4 photoproducts, in UV-irradiated Escherichia coli.
description
1998 nî lūn-bûn
@nan
1998 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Induction of the SOS response ...... V-irradiated Escherichia coli.
@ast
Induction of the SOS response ...... V-irradiated Escherichia coli.
@en
type
label
Induction of the SOS response ...... V-irradiated Escherichia coli.
@ast
Induction of the SOS response ...... V-irradiated Escherichia coli.
@en
prefLabel
Induction of the SOS response ...... V-irradiated Escherichia coli.
@ast
Induction of the SOS response ...... V-irradiated Escherichia coli.
@en
P2860
P1476
Induction of the SOS response ...... UV-irradiated Escherichia coli
@en
P2093
D J Crowley
P C Hanawalt
P2860
P304
P407
P577
1998-07-01T00:00:00Z