Glucocorticoid enhancement of memory storage involves noradrenergic activation in the basolateral amygdala
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Biomarkers of Psychological Stress in Health Disparities ResearchTargeting abnormal neural circuits in mood and anxiety disorders: from the laboratory to the clinicThe Role of Lactate-Mediated Metabolic Coupling between Astrocytes and Neurons in Long-Term Memory FormationModulation of Fear Extinction by Stress, Stress Hormones and Estradiol: A Review.Role of Glia in Stress-Induced Enhancement and Impairment of MemoryInteroception and stress.Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disordersThe brain on stress: Insight from studies using the Visible Burrow SystemShort environmental enrichment in adulthood reverses anxiety and basolateral amygdala hypertrophy induced by maternal separation.Peripheral and central effects of circulating catecholaminesPredator Stress-Induced CRF Release Causes Enduring Sensitization of Basolateral Amygdala Norepinephrine Systems that Promote PTSD-Like Startle Abnormalities.Interacting brain systems modulate memory consolidation.Interactions between epinephrine, ascending vagal fibers, and central noradrenergic systems in modulating memory for emotionally arousing events.Activation of the basolateral amygdala induces long-term enhancement of specific memory representations in the cerebral cortexImpaired conditioned fear response and startle reactivity in epinephrine-deficient miceContextual exploration previous to an aversive event predicts long-term emotional consequences of severe stress.Post-exposure sleep deprivation facilitates correctly timed interactions between glucocorticoid and adrenergic systems, which attenuate traumatic stress responses.The basolateral amygdala modulates specific sensory memory representations in the cerebral cortex.Memory modulation.Emotion-induced retrograde amnesia varies as a function of noradrenergic-glucocorticoid activityInvolvement of basolateral amygdala alpha2-adrenoceptors in modulating consolidation of inhibitory avoidance memory.Corticotropin-releasing factor in the basolateral amygdala enhances memory consolidation via an interaction with the beta-adrenoceptor-cAMP pathway: dependence on glucocorticoid receptor activation.Oral administration of a corticotropin-releasing hormone receptor antagonist significantly attenuates behavioral, neuroendocrine, and autonomic responses to stress in primatesCognitive neuroscience of emotional memory.Glucocorticoid effects on memory consolidation depend on functional interactions between the medial prefrontal cortex and basolateral amygdala.Membrane-associated glucocorticoid activity is necessary for modulation of long-term memory via chromatin modification.Maternal care, gene expression, and the development of individual differences in stress reactivity.Memory-influencing intra-basolateral amygdala drug infusions modulate expression of Arc protein in the hippocampusLesions of the nucleus basalis magnocellularis induced by 192 IgG-saporin block memory enhancement with posttraining norepinephrine in the basolateral amygdala.Memory enhancement induced by post-training intrabasolateral amygdala infusions of beta-adrenergic or muscarinic agonists requires activation of dopamine receptors: Involvement of right, but not left, basolateral amygdalaGlucocorticoids in the prefrontal cortex enhance memory consolidation and impair working memory by a common neural mechanismInvolvement of stress-released corticotropin-releasing hormone in the basolateral amygdala in regulating memory consolidation.Stress-induced enhancement of mouse amygdalar synaptic plasticity depends on glucocorticoid and ß-adrenergic activity.THE RELATIONSHIP BETWEEN ACUTE GLUCOCORTICOID LEVELS AND HIPPOCAMPAL FUNCTION DEPENDS UPON TASK AVERSIVENESS AND MEMORY PROCESSING STAGE.Neuropeptide S enhances memory during the consolidation phase and interacts with noradrenergic systems in the brain.Glucocorticoid enhancement of memory requires arousal-induced noradrenergic activation in the basolateral amygdalaStress enhances retrieval of drug-related memories in abstinent heroin addictsThe influence of acute stress on the regulation of conditioned fear.Involvement of basolateral amygdala GABAA receptors in the effect of dexamethasone on memory in rats.Glucocorticoid effects on object recognition memory require training-associated emotional arousal.
P2860
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P2860
Glucocorticoid enhancement of memory storage involves noradrenergic activation in the basolateral amygdala
description
1997 nî lūn-bûn
@nan
1997 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@ast
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@en
type
label
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@ast
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@en
prefLabel
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@ast
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@en
P2093
P2860
P356
P1476
Glucocorticoid enhancement of ...... on in the basolateral amygdala
@en
P2093
B Roozendaal
G L Quirarte
J L McGaugh
P2860
P304
14048-14053
P356
10.1073/PNAS.94.25.14048
P407
P577
1997-12-01T00:00:00Z