Glucocorticoid enhancement of memory requires arousal-induced noradrenergic activation in the basolateral amygdala - Wikidata - awgldk - TriplyDB

Glucocorticoid enhancement of memory requires arousal-induced noradrenergic activation in the basolateral amygdala

Glucocorticoid enhancement of memory requires arousal-induced noradrenergic activation in the basolateral amygdala

http://www.wikidata.org/entity/Q34598037

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Regulation of excitatory synapses and fearful memories by stress hormones The temporal dynamics model of emotional memory processing: a synthesis on the neurobiological basis of stress-induced amnesia, flashbulb and traumatic memories, and the Yerkes-Dodson law Local amplification of glucocorticoids in the aging brain and impaired spatial memory Stress Effects on Multiple Memory System Interactions Linking Mitochondria to Synapses: New Insights for Stress-Related Neuropsychiatric Disorders Modulation of Fear Extinction by Stress, Stress Hormones and Estradiol: A Review.Overexpression of Mineralocorticoid Receptors Partially Prevents Chronic Stress-Induced Reductions in Hippocampal Memory and Structural Plasticity Memory reconsolidation, emotional arousal, and the process of change in psychotherapy: New insights from brain science Differential Activation in Amygdala and Plasma Noradrenaline during Colorectal Distention by Administration of Corticotropin-Releasing Hormone between Healthy Individuals and Patients with Irritable Bowel Syndrome Dopaminergic modulation of the persistence of one-trial hippocampus-dependent memory Stress-induced obesity and the emotional nervous system Acute Stress Suppresses Synaptic Inhibition and Increases Anxiety via Endocannabinoid Release in the Basolateral Amygdala.Revisiting propranolol and PTSD: Memory erasure or extinction enhancement?Predator Stress-Induced CRF Release Causes Enduring Sensitization of Basolateral Amygdala Norepinephrine Systems that Promote PTSD-Like Startle Abnormalities.Enhancing offspring hypothalamic-pituitary-adrenal (HPA) regulation via systematic novelty exposure: the influence of maternal HPA function.Impaired conditioned fear response and startle reactivity in epinephrine-deficient mice Effects of chronic plus acute prolonged stress on measures of coping style, anxiety, and evoked HPA-axis reactivity.Post-exposure sleep deprivation facilitates correctly timed interactions between glucocorticoid and adrenergic systems, which attenuate traumatic stress responses.The impact of anxiety upon cognition: perspectives from human threat of shock studies.The basolateral amygdala modulates specific sensory memory representations in the cerebral cortex.Memory modulation.Angst and the amygdala Pharmacotherapy of PTSD: premises, principles, and priorities Emotion-induced retrograde amnesia varies as a function of noradrenergic-glucocorticoid activity Emotional memory in schizophrenia.Corticotropin-releasing factor in the basolateral amygdala enhances memory consolidation via an interaction with the beta-adrenoceptor-cAMP pathway: dependence on glucocorticoid receptor activation.Cortisol's effects on hippocampal activation in depressed patients are related to alterations in memory formation Bipolar and major depressive disorder: neuroimaging the developmental-degenerative divide.Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat.Glucocorticoid effects on memory consolidation depend on functional interactions between the medial prefrontal cortex and basolateral amygdala.Noradrenergic activation of the basolateral amygdala modulates the consolidation of object-in-context recognition memory Pilot study of the effect of lipophilic vs. hydrophilic beta-adrenergic blockers being taken at time of intracardiac defibrillator discharge on subsequent PTSD symptoms.Membrane-associated glucocorticoid activity is necessary for modulation of long-term memory via chromatin modification.Forebrain glucocorticoid receptor overexpression increases environmental reactivity and produces a stress-induced spatial discrimination deficit Glucocorticoids in the prefrontal cortex enhance memory consolidation and impair working memory by a common neural mechanism Stress-induced enhancement of mouse amygdalar synaptic plasticity depends on glucocorticoid and ß-adrenergic activity.Emotional memory formation under lower versus higher stress conditions Noradrenergic actions in the basolateral complex of the amygdala modulate Arc expression in hippocampal synapses and consolidation of aversive and non-aversive memory.The basolateral amygdala regulates adaptation to stress via β-adrenergic receptor-mediated reductions in phosphorylated extracellular signal-regulated kinase.Effects of pre-encoding stress on brain correlates associated with the long-term memory for emotional scenes

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Glucocorticoid enhancement of memory requires arousal-induced noradrenergic activation in the basolateral amygdala

http://www.wikidata.org/entity/Q34598037

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2006 nî lūn-bûn

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2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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Glucocorticoid enhancement of ...... on in the basolateral amygdala

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Proceedings of the National Academy of Sciences of the United States of America

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Glucocorticoid enhancement of ...... on in the basolateral amygdala

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Benno Roozendaal

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Eddy A Van der Zee

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James L McGaugh

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Shoki Okuda

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P2860

TYROSINE HYDROXYLASE. THE INITIAL STEP IN NOREPINEPHRINE BIOSYNTHESIS

Cognitive neuroscience of emotional memory.

Enhanced memory for emotional material following stress-level cortisol treatment in humans.

Glucocorticoid enhancement of memory storage involves noradrenergic activation in the basolateral amygdala

1999 Curt P. Richter award. Glucocorticoids and the regulation of memory consolidation.

Both protein kinase A and mitogen-activated protein kinase are required in the amygdala for the macromolecular synthesis-dependent late phase of long-term potentiation.

Long-term memory is facilitated by cAMP response element-binding protein overexpression in the amygdala.

Stress in the brain.

The neurobiology of stress: from serendipity to clinical relevance.

Role of adrenal stress hormones in forming lasting memories in the brain.

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