Macrophages produce TGF-beta-induced (beta-ig-h3) following ingestion of apoptotic cells and regulate MMP14 levels and collagen turnover in fibroblasts. - Wikidata - awgldk - TriplyDB

Macrophages produce TGF-beta-induced (beta-ig-h3) following ingestion of apoptotic cells and regulate MMP14 levels and collagen turnover in fibroblasts.

Macrophages produce TGF-beta-induced (beta-ig-h3) following ingestion of apoptotic cells and regulate MMP14 levels and collagen turnover in fibroblasts.

http://www.wikidata.org/entity/Q33760755

about

Prostate cancer and parasitism of the bone hematopoietic stem cell niche Macrophage TGF-β1 and the Proapoptotic Extracellular Matrix Protein BIGH3 Induce Renal Cell Apoptosis in Prediabetic and Diabetic Conditions In Silico and In Vivo Experiments Reveal M-CSF Injections Accelerate Regeneration Following Muscle Laceration.Gene expression of catabolic inflammatory cytokines peak before anabolic inflammatory cytokines after ACL injury in a preclinical model Inflammation blood and tissue factors of plaque growth in an experimental model evidenced by a systems approach Proteomic differences between male and female anterior cruciate ligament and patellar tendon.Temporal induction of immunoregulatory processes coincides with age-dependent resistance to viral-induced type 1 diabetes MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.Microarray-based transcriptional and epigenetic profiling of matrix metalloproteinases, collagens, and related genes in cancer.Interleukins 4 and 13 modulate gene expression and promote proliferation of primary human tenocytes.Blockade of transforming growth factor-beta1 accelerates lymphatic regeneration during wound repair.Endogenous Jmjd6 gene product is expressed at the cell surface and regulates phagocytosis in immature monocyte-like activated THP-1 cells.Local apoptosis promotes collagen production by monocyte-derived cells in transforming growth factor β1-induced lung fibrosis Epigenetic regulation of matrix metalloproteinases and their collagen substrates in cancer.Apoptosis modulation as a promising target for treatment of systemic sclerosis.MicroRNA-34a Negatively Regulates Efferocytosis by Tissue Macrophages in Part via SIRT1 M2 macrophage polarisation is associated with alveolar formation during postnatal lung development.Macrophage phenotypes during tissue repair.Elevated expression of NEU1 sialidase in idiopathic pulmonary fibrosis provokes pulmonary collagen deposition, lymphocytosis, and fibrosis.Dissection of the human multipotent adult progenitor cell secretome by proteomic analysis.Sjögren's syndrome-like ocular surface disease in thrombospondin-1 deficient mice.Transcriptomic meta-analysis of multiple sclerosis and its experimental models.Macrophages and CSF-1: implications for development and beyond Interleukin-33 potentiates bleomycin-induced lung injury.Inhibition of the αvβ6 integrin leads to limited alteration of TGF-α-induced pulmonary fibrosis.A novel multi-network approach reveals tissue-specific cellular modulators of fibrosis in systemic sclerosis.IFN-γ directly controls IL-33 protein level through a STAT1- and LMP2-dependent mechanism.Molecular basis of organ fibrosis: potential therapeutic approaches.Targeting vascular and leukocyte communication in angiogenesis, inflammation and fibrosis.Macrophage-Secreted TNFα and TGFβ1 Influence Migration Speed and Persistence of Cancer Cells in 3D Tissue Culture via Independent Pathways.The NEU1-selective sialidase inhibitor, C9-butyl-amide-DANA, blocks sialidase activity and NEU1-mediated bioactivities in human lung in vitro and murine lung in vivo.Gene expression profiling in the lungs of pigs with different susceptibilities to Glässer's disease.The effect of CSF-1 administration on lung maturation in a mouse model of neonatal hyperoxia exposure.The Ewing sarcoma secretome and its response to activation of Wnt/beta-catenin signaling.

P2860

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P2860

Macrophages produce TGF-beta-induced (beta-ig-h3) following ingestion of apoptotic cells and regulate MMP14 levels and collagen turnover in fibroblasts.

http://www.wikidata.org/entity/Q33760755

description

2008 nî lūn-bûn

@nan

2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

@ast

Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

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type

label

Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

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Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

@en

prefLabel

Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

@ast

Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

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JIMMUNOL.180.7.5036

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Journal of Immunology

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Macrophages produce TGF-beta-i ...... lagen turnover in fibroblasts.

@en

P2093

Irina G Luzina

http://www.w3.org/2001/XMLSchema#string

Kendrick Highsmith

http://www.w3.org/2001/XMLSchema#string

Kerill Pochetuhen

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Michael P Gillmeister

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Natalia Nacu

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Nevins W Todd

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Sergei P Atamas

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Virginia Lockatell

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Zachary A Cooper

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P2860

Induction of secondary apoptosis, inflammation, and lung fibrosis after intratracheal instillation of apoptotic cells in rats

A matrix metalloproteinase expressed on the surface of invasive tumour cells

Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

A critical role for the membrane-type 1 matrix metalloproteinase in collagen phagocytosis.

Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair.

Tumor necrosis factor-alpha and interferon-gamma suppress the activation of human type I collagen gene expression by transforming growth factor-beta 1. Evidence for two distinct mechanisms of inhibition at the transcriptional and posttranscriptional

Cytokine regulation of pulmonary fibrosis in scleroderma.

Lung inflammation and fibrosis: an alveolar macrophage-centered perspective from the 1970s to 1980s.

Expression of matrix metalloproteinases during rat skin wound healing: evidence that membrane type-1 matrix metalloproteinase is a stromal activator of pro-gelatinase A

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5036-5044

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10.4049/JIMMUNOL.180.7.5036

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7

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180

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5498691

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18354229

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apoptotic process

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2847349

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