Sprouty genes control diastema tooth development via bidirectional antagonism of epithelial-mesenchymal FGF signaling. - Wikidata - awgldk - TriplyDB

Sprouty genes control diastema tooth development via bidirectional antagonism of epithelial-mesenchymal FGF signaling.

Sprouty genes control diastema tooth development via bidirectional antagonism of epithelial-mesenchymal FGF signaling.

http://www.wikidata.org/entity/Q33762161

about

Stem cell sources for tooth regeneration: current status and future prospects Continuous tooth generation in mouse is induced by activated epithelial Wnt/beta-catenin signaling Test Tube Tooth: The Next Big Thing Hyperplasia of interstitial cells of cajal in sprouty homolog 4 deficient mice αE-catenin inhibits YAP/TAZ activity to regulate signalling centre formation during tooth development R-spondins/Lgrs expression in tooth development Primary cilia regulate Shh activity in the control of molar tooth number Enamel-free teeth: Tbx1 deletion affects amelogenesis in rodent incisors Control of mitotic spindle angle by the RAS-regulated ERK1/2 pathway determines lung tube shape Migration of Founder Epithelial Cells Drives Proper Molar Tooth Positioning and Morphogenesis Phenotypic and evolutionary implications of modulating the ERK-MAPK cascade using the dentition as a model Manipulation of Fgf and Bmp signaling in teleost fishes suggests potential pathways for the evolutionary origin of multicuspid teeth Three-dimensional analysis of the early development of the dentition From molecules to mastication: the development and evolution of teeth Wnt signaling in the murine diastema Regulation of tooth number by fine-tuning levels of receptor-tyrosine kinase signaling Modulation of Fgf3 dosage in mouse and men mirrors evolution of mammalian dentition Patterning by heritage in mouse molar row development Lrp4 modulates extracellular integration of cell signaling pathways in development.FGF signaling sustains the odontogenic fate of dental mesenchyme by suppressing β-catenin signaling Conditional expression of Spry1 in neural crest causes craniofacial and cardiac defects.The Interaction of Genetic Background and Mutational Effects in Regulation of Mouse Craniofacial Shape.Establishment of extracellular signal-regulated kinase 1/2 bistability and sustained activation through Sprouty 2 and its relevance for epithelial function.FGF signaling regulates the number of posterior taste papillae by controlling progenitor field size.Craniofacial and dental development in cardio-facio-cutaneous syndrome: the importance of Ras signaling homeostasis.Expression of microRNAs in the stem cell niche of the adult mouse incisor.Formation of oral and pharyngeal dentition in teleosts depends on differential recruitment of retinoic acid signaling Sprouty proteins inhibit receptor-mediated activation of phosphatidylinositol-specific phospholipase C.Sprouty genes function in suppression of prostate tumorigenesis microRNA miR-34a regulates cytodifferentiation and targets multi-signaling pathways in human dental papilla cells.Expression analysis of candidate genes regulating successional tooth formation in the human embryo FGFR2 in the dental epithelium is essential for development and maintenance of the maxillary cervical loop, a stem cell niche in mouse incisors Sprouty1 and Sprouty2 limit both the size of the otic placode and hindbrain Wnt8a by antagonizing FGF signaling Exogenous fibroblast growth factor 8 rescues development of mouse diastemal vestigial tooth ex vivo Sprouty is a cytoplasmic target of adenoviral E1A oncoproteins to regulate the receptor tyrosine kinase signalling pathway Interactions between BMP-7 and USAG-1 (uterine sensitization-associated gene-1) regulate supernumerary organ formations Spry1 and Spry2 are necessary for lens vesicle separation and corneal differentiation Molecular genetics of supernumerary tooth formation.Sprouty2 mediated tuning of signalling is essential for somite myogenesis.Sprouty gene dosage influences temporal-spatial dynamics of primary enamel knot formation

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P2860

Sprouty genes control diastema tooth development via bidirectional antagonism of epithelial-mesenchymal FGF signaling.

http://www.wikidata.org/entity/Q33762161

description

2006 nî lūn-bûn

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2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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type

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Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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J.DEVCEL.2006.05.014

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Developmental Cell

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Sprouty genes control diastema ...... ial-mesenchymal FGF signaling.

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Aapo Kangas

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Benjamin D Yu

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George Minowada

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Herve Lesot

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Jukka Jernvall

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Miroslav Peterka

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Ophir D Klein

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Renata Peterkova

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P2860

Gli2 and Gli3 localize to cilia and require the intraflagellar transport protein polaris for processing and function

Cellular signaling by fibroblast growth factor receptors

sprouty encodes a novel antagonist of FGF signaling that patterns apical branching of the Drosophila airways

FGF4, a direct target of LEF1 and Wnt signaling, can rescue the arrest of tooth organogenesis in Lef1(-/-) mice

Antagonistic interactions between FGF and BMP signaling pathways: a mechanism for positioning the sites of tooth formation

Sprouty1 is a critical regulator of GDNF/RET-mediated kidney induction

Conditional inactivation of FGF receptor 2 reveals an essential role for FGF signaling in the regulation of osteoblast function and bone growth

FGF10 maintains stem cell compartment in developing mouse incisors

Regulation of mammalian tooth cusp patterning by ectodin

Sprouty2, a mouse deafness gene, regulates cell fate decisions in the auditory sensory epithelium by antagonizing FGF signaling

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181-190

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Gail Roberta Martin

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6897019

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