A single nucleotide polymorphism in the Bax gene promoter affects transcription and influences retinal ganglion cell death
about
Silencing of Fem1cR3 gene expression in the DBA/2J mouse precedes retinal ganglion cell death and is associated with histone deacetylase activityThe apoptotic response in HCT116BAX-/- cancer cells becomes rapidly saturated with increasing expression of a GFP-BAX fusion proteinVariations in the rheostat model of apoptosis: what studies of retinal ganglion cell death tell us about the functions of the Bcl2 family proteins.Histone deacetylase 3 (HDAC3) plays an important role in retinal ganglion cell death after acute optic nerve injury.Pou4f1 and pou4f2 are dispensable for the long-term survival of adult retinal ganglion cells in mice.Assessment of epigenetic contributions to sexually-dimorphic Kiss1 expression in the anteroventral periventricular nucleus of miceThe Bcl-2 family member BIM has multiple glaucoma-relevant functions in DBA/2J miceAnalysis of functional polymorphisms in apoptosis-related genes in primary open angle glaucomaNuclear atrophy of retinal ganglion cells precedes the bax-dependent stage of apoptosis.Retinal glial responses to optic nerve crush are attenuated in Bax-deficient mice and modulated by purinergic signaling pathways.Role of HDACs in optic nerve damage-induced nuclear atrophy of retinal ganglion cells.The cell and molecular biology of glaucoma: mechanisms of retinal ganglion cell death.Genetic determinants of neuronal vulnerability to apoptosis.Glaucoma and optic nerve repair.Genomic Control of Retinal Cell Number: Challenges, Protocol, and Results.BAX to basics: How the BCL2 gene family controls the death of retinal ganglion cells.JUN is important for ocular hypertension-induced retinal ganglion cell degeneration.Together JUN and DDIT3 (CHOP) control retinal ganglion cell death after axonal injury.JUN regulates early transcriptional responses to axonal injury in retinal ganglion cells.AAV2-Mediated Transduction of the Mouse Retina After Optic Nerve Injury.Meta-analysis of transcriptomic changes in optic nerve injury and neurodegenerative models reveals a fundamental response to injury throughout the central nervous system.Targeting HDAC3 Activity with RGFP966 Protects Against Retinal Ganglion Cell Nuclear Atrophy and Apoptosis After Optic Nerve Injury.
P2860
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P2860
A single nucleotide polymorphism in the Bax gene promoter affects transcription and influences retinal ganglion cell death
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
A single nucleotide polymorphi ...... es retinal ganglion cell death
@ast
A single nucleotide polymorphi ...... es retinal ganglion cell death
@en
type
label
A single nucleotide polymorphi ...... es retinal ganglion cell death
@ast
A single nucleotide polymorphi ...... es retinal ganglion cell death
@en
prefLabel
A single nucleotide polymorphi ...... es retinal ganglion cell death
@ast
A single nucleotide polymorphi ...... es retinal ganglion cell death
@en
P2093
P2860
P356
P1433
P1476
A single nucleotide polymorphi ...... es retinal ganglion cell death
@en
P2093
Robert W Nickells
Sheila J Semaan
P2860
P304
P356
10.1042/AN20100003
P577
2010-03-31T00:00:00Z