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Coronavirus infection, ER stress, apoptosis and innate immunity

Coronavirus infection, ER stress, apoptosis and innate immunity

http://www.wikidata.org/entity/Q33767779

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Inflammatory and oxidative stress in rotavirus infection Molecular Mechanisms of White Spot Syndrome Virus Infection and Perspectives on Treatments Arms Race between Enveloped Viruses and the Host ERAD Machinery Bat Severe Acute Respiratory Syndrome-Like Coronavirus WIV1 Encodes an Extra Accessory Protein, ORFX, Involved in Modulation of the Host Immune Response.Viral and Cellular mRNA Translation in Coronavirus-Infected Cells.The endoplasmic reticulum stress sensor IRE1α protects cells from apoptosis induced by the coronavirus infectious bronchitis virus Astragalin inhibits autophagy-associated airway epithelial fibrosis.The NF-κB-dependent and -independent transcriptome and chromatin landscapes of human coronavirus 229E-infected cells.Comparative Analysis of Salivary Gland Proteomes of Two Glossina Species that Exhibit Differential Hytrosavirus Pathologies.Mitophagy in TGEV infection counteracts oxidative stress and apoptosis.Opportunistic intruders: how viruses orchestrate ER functions to infect cells.NS2A comprises a putative viroporin of Dengue virus 2.The unfolded protein response in virus infections.Human Coronaviruses: A Review of Virus-Host Interactions.Viroporins: Structures and Functions beyond Cell Membrane Permeabilization.Genotype 2 strains of porcine reproductive and respiratory syndrome virus dysregulate alveolar macrophage cytokine production via the unfolded protein response.Activation of the c-Jun NH2-terminal kinase pathway by coronavirus infectious bronchitis virus promotes apoptosis independently of c-Jun.Inhibition of cytosolic phospholipase A2α impairs an early step of coronavirus replication in cell culture.Encephalitis is mediated by ROP18 of Toxoplasma gondii, a severe pathogen in AIDS patients.The PERK Arm of the Unfolded Protein Response Negatively Regulates Transmissible Gastroenteritis Virus Replication by Suppressing Protein Translation and Promoting Type I IFN Production.Differential Regulation of Toll-Like Receptor-Mediated Cytokine Production by Unfolded Protein Response.

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Coronavirus infection, ER stress, apoptosis and innate immunity

http://www.wikidata.org/entity/Q33767779

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Coronavirus infection, ER stress, apoptosis and innate immunity

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Coronavirus infection, ER stress, apoptosis and innate immunity

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Coronavirus infection, ER stress, apoptosis and innate immunity

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Coronavirus infection, ER stress, apoptosis and innate immunity

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FMICB.2014.00296

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Frontiers in Microbiology

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Coronavirus infection, ER stress, apoptosis and innate immunity

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Ding X Liu

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To S Fung

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P2860

Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress

Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1

Activation of caspase-12, an endoplastic reticulum (ER) resident caspase, through tumor necrosis factor receptor-associated factor 2-dependent mechanism in response to the ER stress

XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor

Protein synthesis and endoplasmic reticulum stress can be modulated by the hepatitis C virus envelope protein E2 through the eukaryotic initiation factor 2alpha kinase PERK

Evidence that hepatitis C virus resistance to interferon is mediated through repression of the PKR protein kinase by the nonstructural 5A protein

A mammalian homologue of GCN2 protein kinase important for translational control by phosphorylation of eukaryotic initiation factor-2alpha

Endoplasmic reticulum stress-induced formation of transcription factor complex ERSF including NF-Y (CBF) and activating transcription factors 6alpha and 6beta that activates the mammalian unfolded protein response

Severe acute respiratory syndrome coronavirus phylogeny: toward consensus

Middle East respiratory syndrome coronavirus (MERS-CoV): announcement of the Coronavirus Study Group

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10.3389/FMICB.2014.00296

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5

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2014-06-17T00:00:00Z

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