The role of herpesvirus entry mediator as a negative regulator of T cell-mediated responses. - Wikidata - awgldk - TriplyDB

The role of herpesvirus entry mediator as a negative regulator of T cell-mediated responses.

The role of herpesvirus entry mediator as a negative regulator of T cell-mediated responses.

http://www.wikidata.org/entity/Q33778417

about

BTLA mediates inhibition of human tumor-specific CD8+ T cells that can be partially reversed by vaccination Beyond CTLA-4 and PD-1, the Generation Z of Negative Checkpoint Regulators Role of Tumor Necrosis Factor Superfamily in Neuroinflammation and Autoimmunity HVEM is a TNF Receptor with Multiple Regulatory Roles in the Mucosal Immune System BTLA interaction with HVEM expressed on CD8(+) T cells promotes survival and memory generation in response to a bacterial infection HVEM signalling promotes colitis Functional interaction between herpes simplex virus type 2 gD and HVEM transiently dampens local chemokine production after murine mucosal infection T follicular helper expansion and humoral-mediated rejection are independent of the HVEM/BTLA pathway B and T lymphocyte attenuator tempers early infection immunity Herpes virus entry mediator (HVEM) modulates proliferation and activation of regulatory T cells following HSV-1 infection Herpes simplex virus glycoprotein D interferes with binding of herpesvirus entry mediator to its ligands through downregulation and direct competition Targeting of B and T lymphocyte associated (BTLA) prevents graft-versus-host disease without global immunosuppression.Attenuating lymphocyte activity: the crystal structure of the BTLA-HVEM complex.Essential role of TNF family molecule LIGHT as a cytokine in the pathogenesis of hepatitis.LIGHT/TNFSR14 can regulate hepatic lipase expression by hepatocytes independent of T cells and Kupffer cells.Distinct expression and inhibitory function of B and T lymphocyte attenuator on human T cells.Regulation of inflammation, autoimmunity, and infection immunity by HVEM-BTLA signaling.Dichotomous regulation of GVHD through bidirectional functions of the BTLA-HVEM pathway.LIGHT regulates inflamed draining lymph node hypertrophy.Herpesvirus entry mediator (TNFRSF14) regulates the persistence of T helper memory cell populations.CD160 is essential for NK-mediated IFN-γ production Extracellular histones are mediators of death through TLR2 and TLR4 in mouse fatal liver injury Herpesvirus entry mediator and nectin-1 mediate herpes simplex virus 1 infection of the murine cornea.Global expression profiling of peripheral Qa-1-restricted CD8αα+TCRαβ+ regulatory T cells reveals innate-like features: implications for immune-regulatory repertoire Selective targeting of the LIGHT-HVEM costimulatory system for the treatment of graft-versus-host disease.Conserved Molecular Underpinnings and Characterization of a Role for Caveolin-1 in the Tumor Microenvironment of Mature T-Cell Lymphomas.NK-cell activation by LIGHT triggers tumor-specific CD8+ T-cell immunity to reject established tumors The Tumor Necrosis Factor Superfamily Molecule LIGHT Promotes Keratinocyte Activity and Skin Fibrosis.Herpes virus entry mediator licenses Listeria infection induced immunopathology through control of type I interferon.The role of coinhibitory signaling pathways in transplantation and tolerance Tumor necrosis factor superfamily 14 (LIGHT) controls thymic stromal lymphopoietin to drive pulmonary fibrosis.Role of Nectin-1 and Herpesvirus Entry Mediator as Cellular Receptors for Herpes Simplex Virus 1 on Primary Murine Dermal Fibroblasts.The Expression of BTLA Was Increased and the Expression of HVEM and LIGHT Were Decreased in the T Cells of Patients with Rheumatoid Arthritis [corrected]The signaling networks of the herpesvirus entry mediator (TNFRSF14) in immune regulation Glycoprotein D-independent spread of pseudorabies virus infection in cultured peripheral nervous system neurons in a compartmented system.Alternative entry receptors for herpes simplex virus and their roles in disease Herpesvirus entry mediator on radiation-resistant cell lineages promotes ocular herpes simplex virus 1 pathogenesis in an entry-independent manner.Immunoregulation by tumor necrosis factor superfamily member LIGHT Immunotherapies: the blockade of inhibitory signals.Herpesvirus entry mediator is a serotype specific determinant of pathogenesis in ocular herpes.

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The role of herpesvirus entry mediator as a negative regulator of T cell-mediated responses.

http://www.wikidata.org/entity/Q33778417

description

2005 nî lūn-bûn

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2005 թուականի Մարտին հրատարակուած գիտական յօդուած

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2005 թվականի մարտին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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Journal of Clinical Investigation

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The role of herpesvirus entry ...... of T cell-mediated responses.

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P2093

Daniel Degrandi

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James Lo

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Jing Wang

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Karin Mink

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Klaus Pfeffer

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Robert A Anders

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Sarah Blink

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Sumit K Subudhi

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Xiaojuan Liu

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Yang Wang

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P2860

B7-H1, a third member of the B7 family, co-stimulates T-cell proliferation and interleukin-10 secretion

The lymphotoxin-beta receptor is necessary and sufficient for LIGHT-mediated apoptosis of tumor cells

B7-H3: a costimulatory molecule for T cell activation and IFN-gamma production

LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator

CD28/B7 system of T cell costimulation

Jak2 deficiency defines an essential developmental checkpoint in definitive hematopoiesis

Herpesvirus entry mediator, a member of the tumor necrosis factor receptor (TNFR) family, interacts with members of the TNFR-associated factor family and activates the transcription factors NF-kappaB and AP-1

LIGHT, a novel ligand for lymphotoxin beta receptor and TR2/HVEM induces apoptosis and suppresses in vivo tumor formation via gene transfer

The regulation of T cell homeostasis and autoimmunity by T cell-derived LIGHT

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

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711-717

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10.1172/JCI200522982

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3

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115

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2005-03-01T00:00:00Z

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15696194

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546456

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