Thioredoxin reductase mediates cell death effects of the combination of beta interferon and retinoic acid. - Wikidata - awgldk - TriplyDB

Thioredoxin reductase mediates cell death effects of the combination of beta interferon and retinoic acid.

Thioredoxin reductase mediates cell death effects of the combination of beta interferon and retinoic acid.

http://www.wikidata.org/entity/Q33781496

about

Redox regulation of cell signaling by selenocysteine in mammalian thioredoxin reductases Identification of GRIM-19, a novel cell death-regulatory gene induced by the interferon-beta and retinoic acid combination, using a genetic approach Identification and characterization of GRIM-1, a cell-death-associated gene product An alternative splicing variant of the selenoprotein thioredoxin reductase is a modulator of estrogen signaling The cell death regulator GRIM-19 is an inhibitor of signal transducer and activator of transcription 3 Thioredoxin reductase Viral interferon regulatory factor 1 of Kaposi's sarcoma-associated herpesvirus interacts with a cell death regulator, GRIM19, and inhibits interferon/retinoic acid-induced cell death.GRIM-19, a death-regulatory gene product, suppresses Stat3 activity via functional interaction GRIM-19, a cell death regulatory protein, is essential for assembly and function of mitochondrial complex I Identification and characterization of a cell cycle and apoptosis regulatory protein-1 as a novel mediator of apoptosis signaling by retinoid CD437 GRIM-19, a cell death regulatory gene product, is a subunit of bovine mitochondrial NADH:ubiquinone oxidoreductase (complex I)The mammalian cytosolic selenoenzyme thioredoxin reductase reduces ubiquinone. A novel mechanism for defense against oxidative stress Rapid induction of cell death by selenium-compromised thioredoxin reductase 1 but not by the fully active enzyme containing selenocysteine Mutation in NDUFA13/GRIM19 leads to early onset hypotonia, dyskinesia and sensorial deficiencies, and mitochondrial complex I instability.Cell death by SecTRAPs: thioredoxin reductase as a prooxidant killer of cells JS-K, a nitric oxide prodrug, has enhanced cytotoxicity in colon cancer cells with knockdown of thioredoxin reductase 1 News and views on thioredoxin reductases.GRIM-19 Expression and Function in Human Gliomas.Cytokine-induced tumor suppressors: a GRIM story GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses.Overexpression of nuclear apoptosis-inducing factor 1 altered the proteomic profile of human gastric cancer cell MKN45 and induced cell cycle arrest at G1/S phase.GRIM-1, a novel growth suppressor, inhibits rRNA maturation by suppressing small nucleolar RNAs Decreased expression of GRIM-19 by DNA hypermethylation promotes aerobic glycolysis and cell proliferation in head and neck squamous cell carcinoma.The viral interferon regulatory factors of KSHV: immunosuppressors or oncogenes?Tumor suppressive protein gene associated with retinoid-interferon-induced mortality (GRIM)-19 inhibits src-induced oncogenic transformation at multiple levels.Somatic and germline mutation in GRIM-19, a dual function gene involved in mitochondrial metabolism and cell death, is linked to mitochondrion-rich (Hurthle cell) tumours of the thyroid.GRIM-19 mutations fail to inhibit v-Src-induced oncogenesis.Regulation of snoRNAs in cancer: close encounters with interferon.Unlocking the potential of retinoic acid in anticancer therapy.Modulation of p53 dependent gene expression and cell death through thioredoxin-thioredoxin reductase by the Interferon-Retinoid combination.GRIM-19: A master regulator of cytokine induced tumor suppression, metastasis and energy metabolism.Ubiquitin B: an essential mediator of trichostatin A-induced tumor-selective killing in human cancer cells.Conversion of Fas-resistant to Fas-sensitive MCF-7 breast cancer cells by the synergistic interaction of interferon-gamma and all-trans retinoic acid.Overexpression of enzymatically active human cytosolic and mitochondrial thioredoxin reductase in HEK-293 cells. Effect on cell growth and differentiation.Regulation of interferon and retinoic acid-induced cell death activation through thioredoxin reductase.Thioredoxin participates in a cell death pathway induced by interferon and retinoid combination.Mutational analysis of human thioredoxin reductase 1. Effects on p53-mediated gene expression and interferon and retinoic acid-induced cell death.Downregulation of GRIM-19 promotes growth and migration of human glioma cells.Selenocysteine-containing proteins in mammals Selenium-containing enzymes in mammals: Chemical perspectives

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P2860

Thioredoxin reductase mediates cell death effects of the combination of beta interferon and retinoic acid.

http://www.wikidata.org/entity/Q33781496

description

1998 nî lūn-bûn

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1998 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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1998 թվականի նոյեմբերին հրատարակված գիտական հոդված

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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Thioredoxin reductase mediates ...... interferon and retinoic acid.

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Thioredoxin reductase mediates ...... interferon and retinoic acid.

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Thioredoxin reductase mediates ...... interferon and retinoic acid.

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Thioredoxin reductase mediates ...... interferon and retinoic acid.

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Thioredoxin reductase mediates ...... interferon and retinoic acid.

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Boyanapalli M

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Gutierrez PL

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Hassel BA

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Hofmann ER

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Jagus R

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Kalvakolanu DV

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Lindner DJ

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Weihua X

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P2860

Identification of a novel serine/threonine kinase and a novel 15-kD protein as potential mediators of the gamma interferon-induced cell death

Immunodeficiency and chronic myelogenous leukemia-like syndrome in mice with a targeted mutation of the ICSBP gene

A model for p53-induced apoptosis

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

Do all programmed cell deaths occur via apoptosis?

A dominant negative mutant of 2-5A-dependent RNase suppresses antiproliferative and antiviral effects of interferon

ATL-derived factor (ADF), an IL-2 receptor/Tac inducer homologous to thioredoxin; possible involvement of dithiol-reduction in the IL-2 receptor induction

Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

Thioredoxin deficiency in yeast prolongs S phase and shortens the G1 interval of the cell cycle.

Deletion of IRF-1, mapping to chromosome 5q31.1, in human leukemia and preleukemic myelodysplasia

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10.1128/MCB.18.11.6493

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11

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18

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109235

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