C/EBPalpha is critical for the neonatal acute-phase response to inflammation - Wikidata - awgldk - TriplyDB

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

http://www.wikidata.org/entity/Q33781735

about

Chromium(VI) down-regulates heavy metal-induced metallothionein gene transcription by modifying transactivation potential of the key transcription factor, metal-responsive transcription factor 1 Hepatocytes: a key cell type for innate immunity Platelet-activating factor and endotoxin activate CCAAT/enhancer binding protein in rat small intestine Fetal liver development requires a paracrine action of oncostatin M through the gp130 signal transducer.Hepcidin regulation of ferroportin 1 expression in the liver and intestine of the rat Whole adult organism transcriptional profiling of acute metal exposures in male zebrafish Gene expression profiling of liver from dairy cows treated intra-mammary with lipopolysaccharide.C/EBPbeta, when expressed from the C/ebpalpha gene locus, can functionally replace C/EBPalpha in liver but not in adipose tissue Putting its fingers on stressful situations: the heavy metal-regulatory transcription factor MTF-1.LPS-induced inflammation in the chicken is associated with CCAAT/enhancer binding protein beta-mediated fat mass and obesity associated gene down-regulation in the liver but not hypothalamus.Mesenchymal-specific deletion of C/EBPβ suppresses pulmonary fibrosis.CCAAT/enhancer-binding protein δ is a critical mediator of lipopolysaccharide-induced acute lung injury.Immunogenomics reveal molecular circuits of diclofenac induced liver injury in mice Heat stress stimulates hepcidin mRNA expression and C/EBPα protein expression in aged rodent liver.Role of specific CCAAT/enhancer-binding protein isoforms in intestinal epithelial cells.Decreased brain damage and curtailed inflammation in transcription factor CCAAT/enhancer binding protein beta knockout mice following transient focal cerebral ischemia.Characterization of rat serum amyloid A4 (SAA4): a novel member of the SAA superfamily.Regulation of CCAAT/enhancer-binding protein (C/EBP) activator proteins by heterodimerization with C/EBPgamma (Ig/EBP).Hyperglycemia-induced production of acute phase reactants in adipose tissue.Disruption of hepatic C/EBPalpha results in impaired glucose tolerance and age-dependent hepatosteatosis.Generation of hepatocyte-like cells from in vitro transdifferentiated human fetal pancreas.Functional analysis of interleukin 6 response elements (IL-6REs) on the human gamma-fibrinogen promoter: binding of hepatic Stat3 correlates negatively with transactivation potential of type II IL-6REs.CCAAT enhancer-binding protein alpha is required for interleukin-6 receptor alpha signaling in newborn hepatocytes.

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C/EBPalpha is critical for the neonatal acute-phase response to inflammation

http://www.wikidata.org/entity/Q33781735

description

1998 nî lūn-bûn

@nan

1998 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

1998年の論文

@ja

1998年論文

@yue

1998年論文

@zh-hant

1998年論文

@zh-hk

1998年論文

@zh-mo

1998年論文

@zh-tw

1998年论文

@wuu

name

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@ast

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@en

type

label

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@ast

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@en

prefLabel

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@ast

C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@en

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Molecular and Cellular Biology

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C/EBPalpha is critical for the neonatal acute-phase response to inflammation

@en

P2093

B L Burgess-Beusse

http://www.w3.org/2001/XMLSchema#string

G J Darlington

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P2860

A nuclear factor for IL-6 expression (NF-IL6) is a member of a C/EBP family

NF-kappa B and C/EBP transcription factor families synergistically function in mouse serum amyloid A gene expression induced by inflammatory cytokines

A member of the C/EBP family, NF-IL6 beta, forms a heterodimer and transcriptionally synergizes with NF-IL6

Interaction and functional collaboration of p300 and C/EBPbeta

Characterization of tumor necrosis factor-deficient mice

Transcription factors Stat3 and Stat5b are present in rat liver nuclei late in an acute phase response and bind interleukin-6 response elements

The role of Stat and C/EBP transcription factors in the synergistic activation of rat serine protease inhibitor-3 gene by interleukin-6 and dexamethasone

CCAAT/enhancer-binding protein mRNA is translated into multiple proteins with different transcription activation potentials

Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8

Stat3: a STAT family member activated by tyrosine phosphorylation in response to epidermal growth factor and interleukin-6

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7269-7277

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scholarly article

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10.1128/MCB.18.12.7269

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12

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18

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1998-12-01T00:00:00Z

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9819413

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109308

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