Intrinsic polymerase activities of UmuD'(2)C and MucA'(2)B are responsible for their different mutagenic properties during bypass of a T-T cis-syn cyclobutane dimer.
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The aflatoxin B(1) formamidopyrimidine adduct plays a major role in causing the types of mutations observed in human hepatocellular carcinomaClusters of nucleotide substitutions and insertion/deletion mutations are associated with repeat sequencesEscherichia coli DNA polymerase III can replicate efficiently past a T-T cis-syn cyclobutane dimer if DNA polymerase V and the 3' to 5' exonuclease proofreading function encoded by dnaQ are inactivated.Translesion DNA Synthesis.A RecA protein surface required for activation of DNA polymerase V.
P2860
Intrinsic polymerase activities of UmuD'(2)C and MucA'(2)B are responsible for their different mutagenic properties during bypass of a T-T cis-syn cyclobutane dimer.
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2000 nî lūn-bûn
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Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@ast
Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@en
type
label
Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@ast
Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@en
prefLabel
Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@ast
Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@en
P2093
P2860
P1476
Intrinsic polymerase activitie ...... T-T cis-syn cyclobutane dimer.
@en
P2093
C W Lawrence
D Vandewiele
P I O'Grady
R Woodgate
P2860
P304
P356
10.1128/JB.182.8.2285-2291.2000
P407
P577
2000-04-01T00:00:00Z