Pax5 loss imposes a reversible differentiation block in B-progenitor acute lymphoblastic leukemia.
about
limma powers differential expression analyses for RNA-sequencing and microarray studiesCLIP-GENE: a web service of the condition specific context-laid integrative analysis for gene prioritization in mouse TF knockout experiments.PAX5 is a tumor suppressor in mouse mutagenesis models of acute lymphoblastic leukemia.Ebf1 heterozygosity results in increased DNA damage in pro-B cells and their synergistic transformation by Pax5 haploinsufficiencyComparative genomics reveals multistep pathogenesis of E2A-PBX1 acute lymphoblastic leukemia.Monomethylarsonous acid (MMA+3) Inhibits IL-7 Signaling in Mouse Pre-B Cells.Synergism of FAK and tyrosine kinase inhibition in Ph+ B-ALL.Activated Notch counteracts Ikaros tumor suppression in mouse and human T-cell acute lymphoblastic leukemia.Conditional knockdown of BCL2A1 reveals rate-limiting roles in BCR-dependent B-cell survival.Long noncoding RNAs in B-cell development and activation.Lineage-Specific Genes Are Prominent DNA Damage Hotspots during Leukemic Transformation of B Cell Precursors.Mechanisms of pre-B-cell receptor checkpoint control and its oncogenic subversion in acute lymphoblastic leukemia.Genomics in acute lymphoblastic leukaemia: insights and treatment implications.Transcription factor networks in B-cell differentiation link development to acute lymphoid leukemia.Conserved IKAROS-regulated genes associated with B-progenitor acute lymphoblastic leukemia outcome.Differentiation of human neuroblastoma cells toward the osteogenic lineage by mTOR inhibitorQuantitative proteomic analysis reveals maturation as a mechanism underlying glucocorticoid resistance in B lineage ALL and re-sensitization by JNK inhibition.Molecular role of the PAX5-ETV6 oncoprotein in promoting B-cell acute lymphoblastic leukemia.Ikaros: Exploiting and targeting the hematopoietic stem cell niche in B-progenitor acute lymphoblastic leukemia.PhenomeExpress: a refined network analysis of expression datasets by inclusion of known disease phenotypes.Metabolic gatekeeper function of B-lymphoid transcription factorsThe role of the Janus-faced transcription factor PAX5-JAK2 in acute lymphoblastic leukemia.DNA Cross-Bridging Shapes a Single Nucleus from a Set of Mitotic ChromosomesTranscribing malignancy: transcription-associated genomic instability in cancer.It is a differentiation game: STAT5 in a new role.Activating PAX gene family paralogs to complement PAX5 leukemia driver mutationsChromatin-dependent allosteric regulation of DNMT3A activity by MeCP2
P2860
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P2860
Pax5 loss imposes a reversible differentiation block in B-progenitor acute lymphoblastic leukemia.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@ast
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@en
type
label
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@ast
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@en
prefLabel
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@ast
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@en
P2093
P2860
P50
P356
P1433
P1476
Pax5 loss imposes a reversible ...... acute lymphoblastic leukemia.
@en
P2093
Grace J Liu
Julian G Jude
Luisa Cimmino
Mark D McKenzie
Matthew T Witkowski
Mutlu Kartal-Kaess
Sarah A Best
P2860
P304
P356
10.1101/GAD.240416.114
P50
P577
2014-06-01T00:00:00Z