Genetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts. - Wikidata - awgldk - TriplyDB

Genetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts.

Genetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts.

http://www.wikidata.org/entity/Q33797150

about

Bcl10 can promote survival of antigen-stimulated B lymphocytes ZBTB2 increases PDK4 expression by transcriptional repression of RelA/p65 CARD11 mediates factor-specific activation of NF-kappaB by the T cell receptor complex.Impaired B cell development and function in the absence of IkappaBNS Rel/Nuclear factor-kappa B apoptosis pathways in human cervical cancer cells.NF-kB inhibitor blocks B cell development at two checkpoints NF-kappa B DNA-binding activity in embryos responding to a teratogen, cyclophosphamide NF-kappa B in rheumatoid arthritis: a pivotal regulator of inflammation, hyperplasia, and tissue destruction New insights into the role of nuclear factor-kB in cell growth regulation SIMPL is a tumor necrosis factor-specific regulator of nuclear factor-kappaB activity The Rel/NF-kappaB signal transduction pathway: introduction Unifying concepts in CD28, ICOS and CTLA4 co-receptor signalling Alpha 5 beta 1 integrin activates an NF-kappa B-dependent program of gene expression important for angiogenesis and inflammation.Physical and functional interaction of CARMA1 and CARMA3 with Ikappa kinase gamma-NFkappaB essential modulator Atypical forms of incontinentia pigmenti in male individuals result from mutations of a cytosine tract in exon 10 of NEMO (IKK-gamma)Nuclear factor kappaB-dependent gene expression profiling of Hodgkin's disease tumor cells, pathogenetic significance, and link to constitutive signal transducer and activator of transcription 5a activity Development of B cells and erythrocytes is specifically impaired by the drug celastrol in mice Kinase Inhibition Leads to Hormesis in a Dual Phosphorylation-Dephosphorylation Cycle Inactivation of BAD by IKK inhibits TNFα-induced apoptosis independently of NF-κB activation Dendrosomal nanocurcumin and p53 overexpression synergistically trigger apoptosis in glioblastoma cells Differential utilization of NF-kappaB RELA and RELB in response to extracellular versus intracellular polyIC stimulation in HT1080 cells.Single TNFalpha trimers mediating NF-kappaB activation: stochastic robustness of NF-kappaB signaling.Computational modeling with forward and reverse engineering links signaling network and genomic regulatory responses: NF-kappaB signaling-induced gene expression responses in inflammation Tissue-specific target analysis of disease-associated microRNAs in human signaling pathways.Selectivity of the NF-{kappa}B response.How NF-kappaB is activated: the role of the IkappaB kinase (IKK) complex.Control of development and immunity by rel transcription factors in Drosophila.Upregulation of toll-like receptor 2 gene expression in macrophage response to peptidoglycan and high concentration of lipopolysaccharide is involved in NF-kappa b activation.The combined absence of NF-kappa B1 and c-Rel reveals that overlapping roles for these transcription factors in the B cell lineage are restricted to the activation and function of mature cells.RelB is required for Peyer's patch development: differential regulation of p52-RelB by lymphotoxin and TNF NF-kappaB1 can inhibit v-Abl-induced lymphoid transformation by functioning as a negative regulator of cyclin D1 expression.Stochastic regulation in early immune response.Stimulus-specific induction of a novel nuclear factor-kappaB regulator, IkappaB-zeta, via Toll/Interleukin-1 receptor is mediated by mRNA stabilization.NF-kappa B defects in humans: the NEMO/incontinentia pigmenti connection.Coordinated binding of NF-kappaB family members in the response of human cells to lipopolysaccharide.Circuitry of nuclear factor kappaB signaling.The anti-apoptotic activities of Rel and RelA required during B-cell maturation involve the regulation of Bcl-2 expression.NF-kappaB activation by protein kinase C isoforms and B-cell function.Exogenous signal-independent nuclear IkappaB kinase activation triggered by Nkx3.2 enables constitutive nuclear degradation of IkappaB-alpha in chondrocytes.Potential role of nuclear factor κB in diabetic cardiomyopathy.

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P2860

Genetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts.

http://www.wikidata.org/entity/Q33797150

description

1999 nî lūn-bûn

@nan

1999 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի նոյեմբերին հրատարակված գիտական հոդված

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1999年の論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年论文

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name

Genetic approaches in mice to ...... on: transgenics and knockouts.

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Genetic approaches in mice to ...... on: transgenics and knockouts.

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Genetic approaches in mice to ...... on: transgenics and knockouts.

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Genetic approaches in mice to ...... on: transgenics and knockouts.

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M Grossmann

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R Grumont

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S Gerondakis

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T Pohl

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Y Nakamura

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P2860

Mutations in TWIST, a basic helix-loop-helix transcription factor, in Saethre-Chotzen syndrome

IEX-1L, an apoptosis inhibitor involved in NF-kappaB-mediated cell survival

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

A new member of the I kappaB protein family, I kappaB epsilon, inhibits RelA (p65)-mediated NF-kappaB transcription

Aberrant rel/nfkb genes and activity in human cancer

The candidate oncoprotein Bcl-3 is an antagonist of p50/NF-kappa B-mediated inhibition

The oncoprotein Bcl-3 directly transactivates through kappa B motifs via association with DNA-binding p50B homodimers

NF-kappa B RelA-deficient lymphocytes: normal development of T cells and B cells, impaired production of IgA and IgG1 and reduced proliferative responses

Immunological defects in mice with a targeted disruption in Bcl-3

Expression of relB is required for the development of thymic medulla and dendritic cells

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10.1038/SJ.ONC.1203236

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49

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18

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1047772247

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10602464

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