Tumorigenic 3T3 cells maintain an alkaline intracellular pH under physiological conditions - Wikidata - awgldk - TriplyDB

Tumorigenic 3T3 cells maintain an alkaline intracellular pH under physiological conditions

Tumorigenic 3T3 cells maintain an alkaline intracellular pH under physiological conditions

http://www.wikidata.org/entity/Q33811440

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A calcineurin homologous protein inhibits GTPase-stimulated Na-H exchange Rho GTPases, oxidation, and cell redox control pH-Responsive, posttranslational regulation of the Trk1 potassium transporter by the type 1-related Ppz1 phosphatase.Hypoxia, cancer metabolism and the therapeutic benefit of targeting lactate/H(+) symporters Vacuolar H(+)-ATPase mutants transform cells and define a binding site for the papillomavirus E5 oncoprotein Lactate and malignant tumors: a therapeutic target at the end stage of glycolysis Expression of calcineurin B homologous protein 2 protects serum deprivation-induced cell death by serum-independent activation of Na+/H+ exchanger Attenuation of mouse melanoma by A/C magnetic field after delivery of bi-magnetic nanoparticles by neural progenitor cells.Microenvironmental acidosis in carcinogenesis and metastases: new strategies in prevention and therapy.The two-pore domain potassium channel KCNK5: induction by estrogen receptor alpha and role in proliferation of breast cancer cells.Live-cell imaging of tumor proteolysis: impact of cellular and non-cellular microenvironment Microenvironmental and cellular consequences of altered blood flow in tumours.pH, Lactate, and Hypoxia: Reciprocity in Regulating High-Affinity Monocarboxylate Transporter Expression in Glioblastoma Cation-coupled bicarbonate transporters The Role of Monocarboxylate Transporters and Their Chaperone CD147 in Lactate Efflux Inhibition and the Anticancer Effects of Terminalia chebula in Neuroblastoma Cell Line N2-A.Na-H exchange acts downstream of RhoA to regulate integrin-induced cell adhesion and spreading.Why are epididymal tumours so rare?Tumour hypoxia induces a metabolic shift causing acidosis: a common feature in cancer.Intracellular pH and the control of multidrug resistance.Inhibition of activator protein 1 activity and neoplastic transformation by aspirin.The chemistry, physiology and pathology of pH in cancer.BAX inhibitor-1 enhances cancer metastasis by altering glucose metabolism and activating the sodium-hydrogen exchanger: the alteration of mitochondrial function.Systematic identification of genes involved in metabolic acid stress resistance in yeast and their potential as cancer targets.Extracellular and Luminal pH Regulation by Vacuolar H+-ATPase Isoform Expression and Targeting to the Plasma Membrane and Endosomes.Repeated H2 O2 exposure drives cell cycle progression in an in vitro model of ulcerative colitis.Dual role of CO2/HCO3(-) buffer in the regulation of intracellular pH of three-dimensional tumor growths.Spectral imaging microscopy demonstrates cytoplasmic pH oscillations in glial cells.Contributions of cell metabolism and H+ diffusion to the acidic pH of tumors.Abnormal glycosylation and altered Golgi structure in colorectal cancer: dependence on intra-Golgi pH.Separation of metabolic supply and demand: aerobic glycolysis as a normal physiological response to fluctuating energetic demands in the membrane.Altered proton extrusion in cells adapted to growth at low extracellular pH.Galpha12 and Galpha13 regulate extracellular signal-regulated kinase and c-Jun kinase pathways by different mechanisms in COS-7 cells.Naturally occurring deuterium is essential for the normal growth rate of cells.Effect of bafilomycin A1, a specific inhibitor of vacuolar (V-type) proton ATPases, on the capacitation of rabbit spermatozoa.A plasma membrane V-type H(+)-ATPase may contribute to elevated intracellular pH (pHin) in some human tumor cells.Characterization of a temperature-sensitive yeast vacuolar ATPase mutant with defects in actin distribution and bud morphology.Genetic alterations leading to increases in internal potassium concentrations are detrimental for DNA integrity in Saccharomyces cerevisiae.Expression and functional role of vacuolar H(+)-ATPase in human hepatocellular carcinoma.Alkaline stress-induced autophagy is mediated by mTORC1 inactivation.Assessing Na+/H+ exchange and cell effector functionality in metastatic breast cancer.

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Tumorigenic 3T3 cells maintain an alkaline intracellular pH under physiological conditions

http://www.wikidata.org/entity/Q33811440

description

1990 nî lūn-bûn

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1990 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1990 թվականի հոտեմբերին հրատարակված գիտական հոդված

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1990年の論文

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1990年学术文章

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1990年学术文章

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1990年学术文章

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1990年学术文章

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1990年学术文章

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1990年學術文章

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Tumorigenic 3T3 cells maintain ...... under physiological conditions

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Tumorigenic 3T3 cells maintain ...... under physiological conditions

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PNAS.87.19.7414

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Tumorigenic 3T3 cells maintain ...... under physiological conditions

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P2093

G M Martinez

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R J Gillies

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R Martinez-Zaguilan

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R Perona

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R Serrano

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P2860

Intracellular pH.

Osteoclastic bone resorption by a polarized vacuolar proton pump.

Fluorescence probe measurement of the intralysosomal pH in living cells and the perturbation of pH by various agents.

Intracellular pH is increased after transformation of Chinese hamster embryo fibroblasts.

Na+/H+ antiport in Swiss 3T3 cells: mitogenic stimulation leads to cytoplasmic alkalinization

Na+/H+ exchange and growth factor-induced cytosolic pH changes. Role in cellular proliferation.

Fluorescent indicators of ion concentrations.

Effects of growth factors on intracellular pH regulation.

Intracellular pH regulation in epithelial cells.

Transformation and pH homeostasis of fibroblasts expressing yeast H(+)-ATPase containing site-directed mutations.

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7414-7418

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scholarly article

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10.1073/PNAS.87.19.7414

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19

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2170979

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54757

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