A null mutation in the UL36 gene of herpes simplex virus type 1 results in accumulation of unenveloped DNA-filled capsids in the cytoplasm of infected cells.
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Analysis of Rab GTPase-activating proteins indicates that Rab1a/b and Rab43 are important for herpes simplex virus 1 secondary envelopmentHigh-level expression of Marek's disease virus glycoprotein C is detrimental to virus growth in vitroDetermination of interactions between tegument proteins of herpes simplex virus type 1Proteolytic cleavage of VP1-2 is required for release of herpes simplex virus 1 DNA into the nucleusHSV-1 tegument protein and the development of its genome editing technologyTegument Assembly and Secondary Envelopment of AlphaherpesvirusesViral and host control of cytomegalovirus maturationRole of human cytomegalovirus tegument proteins in virion assemblyKaposi's sarcoma-associated herpesvirus ORF45 interacts with kinesin-2 transporting viral capsid-tegument complexes along microtubulesDynamic ubiquitination drives herpesvirus neuroinvasionStructure of the herpesvirus major capsid proteinCrystal Structure of the Herpesvirus Inner Tegument Protein UL37 Supports Its Essential Role in Control of Viral TraffickingThe herpesvirus VP1/2 protein is an effector of dynein-mediated capsid transport and neuroinvasion.Murine Cytomegalovirus Deubiquitinase Regulates Viral Chemokine Levels To Control Inflammation and PathogenesisImproper tagging of the non-essential small capsid protein VP26 impairs nuclear capsid egress of herpes simplex virus.Conserved Tryptophan Motifs in the Large Tegument Protein pUL36 Are Required for Efficient Secondary Envelopment of Herpes Simplex Virus Capsids.The UL36 tegument protein of herpes simplex virus 1 has a composite binding site at the capsid vertices.The C terminus of the large tegument protein pUL36 contains multiple capsid binding sites that function differently during assembly and cell entry of herpes simplex virusStructure and capsid association of the herpesvirus large tegument protein UL36.Localization of herpes simplex virus type 1 UL37 in the Golgi complex requires UL36 but not capsid structures.Allosteric signaling and a nuclear exit strategy: binding of UL25/UL17 heterodimers to DNA-Filled HSV-1 capsids.The pseudorabies virus VP1/2 tegument protein is required for intracellular capsid transportReconstitution of herpes simplex virus microtubule-dependent trafficking in vitro.The Herpesvirus capsid surface protein, VP26, and the majority of the tegument proteins are dispensable for capsid transport toward the nucleus.Identification of an essential domain in the herpesvirus VP1/2 tegument protein: the carboxy terminus directs incorporation into capsid assemblonsDystonin/BPAG1 promotes plus-end-directed transport of herpes simplex virus 1 capsids on microtubules during entry.Effects of charged cluster mutations on the function of herpes simplex virus type 1 UL34 protein.Identification and characterization of the pseudorabies virus tegument proteins UL46 and UL47: role for UL47 in virion morphogenesis in the cytoplasm.Identification of candidate gammaherpesvirus 68 genes required for virus replication by signature-tagged transposon mutagenesis.A herpesvirus encoded deubiquitinase is a novel neuroinvasive determinantRed-mediated transposition and final release of the mini-F vector of a cloned infectious herpesvirus genomeThe herpes simplex virus type 1 DNA packaging protein UL17 is a virion protein that is present in both the capsid and the tegument compartmentsNelfinavir inhibits maturation and export of herpes simplex virus 1.The major determinant for addition of tegument protein pUL48 (VP16) to capsids in herpes simplex virus type 1 is the presence of the major tegument protein pUL36 (VP1/2)Plus- and minus-end directed microtubule motors bind simultaneously to herpes simplex virus capsids using different inner tegument structuresPseudorabies virus UL37 gene product is involved in secondary envelopment.Virion association of IE62, the varicella-zoster virus (VZV) major transcriptional regulatory protein, requires expression of the VZV open reading frame 66 protein kinaseA null mutation in the gene encoding the herpes simplex virus type 1 UL37 polypeptide abrogates virus maturation.Cryo electron tomography of herpes simplex virus during axonal transport and secondary envelopment in primary neurons.Human cytomegalovirus UL47 tegument protein functions after entry and before immediate-early gene expression
P2860
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P2860
A null mutation in the UL36 gene of herpes simplex virus type 1 results in accumulation of unenveloped DNA-filled capsids in the cytoplasm of infected cells.
description
2000 nî lūn-bûn
@nan
2000 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի դեկտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
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2000年论文
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name
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@ast
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@en
type
label
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@ast
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@en
prefLabel
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@ast
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@en
P2860
P1433
P1476
A null mutation in the UL36 ge ...... e cytoplasm of infected cells.
@en
P2093
P2860
P304
11608-11618
P356
10.1128/JVI.74.24.11608-11618.2000
P577
2000-12-01T00:00:00Z