Depletion of blood-borne macrophages does not reduce demyelination in mice infected with a neurotropic coronavirus.
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Regulatory T cells inhibit T cell proliferation and decrease demyelination in mice chronically infected with a coronavirusThe pathogenesis of murine coronavirus infection of the central nervous system.Coronavirus-induced demyelination occurs in the absence of inducible nitric oxide synthaseMacrophage infiltration, but not apoptosis, is correlated with immune-mediated demyelination following murine infection with a neurotropic coronavirus.Axonal damage is T cell mediated and occurs concomitantly with demyelination in mice infected with a neurotropic coronavirus.High-magnitude, virus-specific CD4 T-cell response in the central nervous system of coronavirus-infected mice.CD4 T-cell-mediated demyelination is increased in the absence of gamma interferon in mice infected with mouse hepatitis virus.Highly activated cytotoxic CD8 T cells express protective IL-10 at the peak of coronavirus-induced encephalitis.Analysis of the host transcriptome from demyelinating spinal cord of murine coronavirus-infected mice.Virally expressed interleukin-10 ameliorates acute encephalomyelitis and chronic demyelination in coronavirus-infected mice.Virus-induced demyelination in nude mice is mediated by gamma delta T cells.Self-reactive CD4(+) T cells activated during viral-induced demyelination do not prevent clinical recoveryThe spike glycoprotein of murine coronavirus MHV-JHM mediates receptor-independent infection and spread in the central nervous systems of Ceacam1a-/- MiceThe chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Interleukin-10 is a critical regulator of white matter lesion containment following viral induced demyelinationPerforin and gamma interferon-mediated control of coronavirus central nervous system infection by CD8 T cells in the absence of CD4 T cellsThe Biology of Persistent Infection: Inflammation and Demyelination following Murine Coronavirus Infection of the Central Nervous SystemImmune responses to non-tumor antigens in the central nervous system.Virus-induced inflammasome activation is suppressed by prostaglandin D2/DP1 signaling.Microglia are required for protection against lethal coronavirus encephalitis in mice.Distinct Gene Profiles of Bone Marrow-Derived Macrophages and Microglia During Neurotropic Coronavirus-Induced Demyelination.
P2860
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P2860
Depletion of blood-borne macrophages does not reduce demyelination in mice infected with a neurotropic coronavirus.
description
1999 nî lūn-bûn
@nan
1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@ast
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@en
type
label
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@ast
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@en
prefLabel
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@ast
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@en
P2093
P2860
P1433
P1476
Depletion of blood-borne macro ...... ith a neurotropic coronavirus.
@en
P2093
P2860
P304
P577
1999-08-01T00:00:00Z