Perforin and gamma interferon-mediated control of coronavirus central nervous system infection by CD8 T cells in the absence of CD4 T cells
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Regulatory T cells in arterivirus and coronavirus infections: do they protect against disease or enhance it?Gamma Interferon Plays a Crucial Early Antiviral Role in Protection against West Nile Virus InfectionMicroRNA-155 enhances T cell trafficking and antiviral effector function in a model of coronavirus-induced neurologic diseaseGamma interferon signaling in oligodendrocytes is critical for protection from neurotropic coronavirus infectionRegulatory T cells inhibit T cell proliferation and decrease demyelination in mice chronically infected with a coronavirusThe pathogenesis of murine coronavirus infection of the central nervous system.Important roles for gamma interferon and NKG2D in gammadelta T-cell-induced demyelination in T-cell receptor beta-deficient mice infected with a coronavirusTarget-dependent B7-H1 regulation contributes to clearance of central nervous system infection and dampens morbidity.TNF-alpha-dependent regulation of CXCR3 expression modulates neuronal survival during West Nile virus encephalitis.Immunopathogenesis of coronavirus infections: implications for SARS.Promoting remyelination: utilizing a viral model of demyelination to assess cell-based therapiesShifting hierarchies of interleukin-10-producing T cell populations in the central nervous system during acute and persistent viral encephalomyelitis.Virus-specific antibody, in the absence of T cells, mediates demyelination in mice infected with a neurotropic coronavirusInhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination.Expression of the mouse hepatitis virus receptor by central nervous system microgliaEnhanced antiviral T cell function in the absence of B7-H1 is insufficient to prevent persistence but exacerbates axonal bystander damage during viral encephalomyelitis.Virally activated CD8 T cells home to Mycobacterium bovis BCG-induced granulomas but enhance antimycobacterial protection only in immunodeficient mice.CD4 T cells promote CD8 T cell immunity at the priming and effector site during viral encephalitisImmune-mediated loss of transgene expression from virally transduced brain cells is irreversible, mediated by IFNγ, perforin, and TNFα, and due to the elimination of transduced cellsMyd88 Initiates Early Innate Immune Responses and Promotes CD4 T Cells during Coronavirus Encephalomyelitis.IFN-γ protects from lethal IL-17 mediated viral encephalomyelitis independent of neutrophils.The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.CD4 T cells contribute to virus control and pathology following central nervous system infection with neurotropic mouse hepatitis virus.Interleukin-10 is a critical regulator of white matter lesion containment following viral induced demyelinationAstrocyte-derived CXCL10 drives accumulation of antibody-secreting cells in the central nervous system during viral encephalomyelitis.Chemokines in and out of the central nervous system: much more than chemotaxis and inflammation.Memory CD4+ T-cell-mediated protection from lethal coronavirus encephalomyelitis.The great balancing act: regulation and fate of antiviral T-cell interactionsInterleukin-12 (IL-12), but not IL-23, deficiency ameliorates viral encephalitis without affecting viral control.IL-21 optimizes T cell and humoral responses in the central nervous system during viral encephalitis.Mouse hepatitis virus infection of the CNS: a model for defense, disease, and repair.Protective and pathologic roles of the immune response to mouse hepatitis virus type 1: implications for severe acute respiratory syndrome.The Biology of Persistent Infection: Inflammation and Demyelination following Murine Coronavirus Infection of the Central Nervous SystemT cell mediated suppression of neurotropic coronavirus replication in neural precursor cells.Viral-induced suppression of self-reactive T cells: Lessons from neurotropic coronavirus-induced demyelination.Role of the inflammasome-related cytokines Il-1 and Il-18 during infection with murine coronavirus.Interferon gamma modulation of disease manifestation and the local antibody response to alphavirus encephalomyelitis.Distinct CD4 T-cell effects on primary versus recall CD8 T-cell responses during viral encephalomyelitis.CD8 T cells require gamma interferon to clear borna disease virus from the brain and prevent immune system-mediated neuronal damage.Immune regulation of transgene expression in the brain: B cells regulate an early phase of elimination of transgene expression from adenoviral vectors.
P2860
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P2860
Perforin and gamma interferon-mediated control of coronavirus central nervous system infection by CD8 T cells in the absence of CD4 T cells
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Perforin and gamma interferon- ...... in the absence of CD4 T cells
@en
type
label
Perforin and gamma interferon- ...... in the absence of CD4 T cells
@en
prefLabel
Perforin and gamma interferon- ...... in the absence of CD4 T cells
@en
P2093
P2860
P1433
P1476
Perforin and gamma interferon- ...... in the absence of CD4 T cells
@en
P2093
Chandran Ramakrishna
David R Hinton
Konechi C Dowdell
Stephen A Stohlman
P2860
P304
P356
10.1128/JVI.78.4.1739-1750.2004
P407
P577
2004-02-01T00:00:00Z