mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse - Wikidata - awgldk - TriplyDB

mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

http://www.wikidata.org/entity/Q33819245

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Pharmacotherapy for stimulant-related disorders Modulation of the endocannabinoid system: vulnerability factor and new treatment target for stimulant addiction Aldehyde dehydrogenase inhibitors: a comprehensive review of the pharmacology, mechanism of action, substrate specificity, and clinical application Chronic inhibition of dopamine β-hydroxylase facilitates behavioral responses to cocaine in mice Methylsulfonylmethane: Applications and Safety of a Novel Dietary Supplement Pharmacological means of reducing human drug dependence: a selective and narrative review of the clinical literature.The interactome of the copper transporter ATP7A belongs to a network of neurodevelopmental and neurodegeneration factors.The Health Effect of Psychostimulants: A Literature Review Effects of pharmacologic dopamine β-hydroxylase inhibition on cocaine-induced reinstatement and dopamine neurochemistry in squirrel monkeys.The α1 Antagonist Doxazosin Alters the Behavioral Effects of Cocaine in Rats Disulfiram attenuates drug-primed reinstatement of cocaine seeking via inhibition of dopamine β-hydroxylase.Pharmacotherapeutics directed at deficiencies associated with cocaine dependence: focus on dopamine, norepinephrine and glutamate.Mechanism-based inactivation of human CYP2E1 by diethyldithocarbamate.Gender differences in clinical outcomes for cocaine dependence: randomized clinical trials of behavioral therapy and disulfiram.Chronic loss of noradrenergic tone produces β-arrestin2-mediated cocaine hypersensitivity and alters cellular D2 responses in the nucleus accumbens Disulfiram efficacy in the treatment of alcohol dependence: a meta-analysis.Long-term γ-hydroxybutyric acid (GHB) and disulfiram combination therapy in GHB treatment-resistant chronic alcoholics.The effects of post-extinction exercise on cocaine-primed and stress-induced reinstatement of cocaine seeking in rats Inhibition of aldehyde dehydrogenase-2 suppresses cocaine seeking by generating THP, a cocaine use-dependent inhibitor of dopamine synthesis.Assessment of safety, cardiovascular and subjective effects after intravenous cocaine and lofexidine.The role of interleukin-18 in glioblastoma pathology implies therapeutic potential of two old drugs-disulfiram and ritonavir.S-(N, N-diethylcarbamoyl)glutathione (carbamathione), a disulfiram metabolite and its effect on nucleus accumbens and prefrontal cortex dopamine, GABA, and glutamate: a microdialysis study.The impact of disulfiram treatment on the reinforcing effects of cocaine: a randomized clinical trial.Evaluation of the dopamine β-hydroxylase (DβH) inhibitor nepicastat in participants who meet criteria for cocaine use disorder.The selective dopamine β-hydroxylase inhibitor nepicastat attenuates multiple aspects of cocaine-seeking behavior The alpha-1 adrenergic antagonist doxazosin for treatment of cocaine dependence: A pilot study.The CYP2E1 inhibitor DDC up-regulates MMP-1 expression in hepatic stellate cells via an ERK1/2- and Akt-dependent mechanism.Pharmacogenetic randomized trial for cocaine abuse: disulfiram and α1A-adrenoceptor gene variation.Dopamine β-hydroxylase inhibitors enhance the discriminative stimulus effects of cocaine in rats.Disulfiram is a direct and potent inhibitor of human O6-methylguanine-DNA methyltransferase (MGMT) in brain tumor cells and mouse brain and markedly increases the alkylating DNA damage.Adrenaline rush: the role of adrenergic receptors in stimulant-induced behaviors Pharmacogenetics of alcohol, nicotine and drug addiction treatments.Current progress in pharmacologic treatment strategies for alcohol dependence.Emerging drugs for the treatment of cocaine use disorder: a review of neurobiological targets and pharmacotherapy.Norepinephrine regulates cocaine-primed reinstatement via α1-adrenergic receptors in the medial prefrontal cortex.Are Alcohol Anti-relapsing and Alcohol Withdrawal Drugs Useful in Cannabinoid Users?Review article: pharmacotherapy for alcohol dependence - the why, the what and the wherefore.Selective inhibition of dopamine-beta-hydroxylase enhances dopamine release from noradrenergic terminals in the medial prefrontal cortex.α1b-Adrenergic Receptor Localization and Relationship to the D1-Dopamine Receptor in the Rat Nucleus Accumbens.Effects of disulfiram on choice behavior in a rodent gambling task: association with catecholamine levels.

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mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

http://www.wikidata.org/entity/Q33819245

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

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mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

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mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

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mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

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Molecular Interventions: pharmacological perspectives from biology, chemistry and genomics

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mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

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P2093

David Weinshenker

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Meriem Gaval-Cruz

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P2860

Glutamatergic substrates of drug addiction and alcoholism

A quantitative-trait analysis of human plasma-dopamine beta-hydroxylase activity: evidence for a major functional polymorphism at the DBH locus

A haplotype at the DBH locus, associated with low plasma dopamine beta-hydroxylase activity, also associates with cocaine-induced paranoia

Disulfiram treatment for cocaine dependence in methadone-maintained opioid addicts

Stress-induced craving and stress response in cocaine dependent individuals

Alpha1b-adrenergic receptors control locomotor and rewarding effects of psychostimulants and opiates

Mice with chronic norepinephrine deficiency resemble amphetamine-sensitized animals

Noradrenaline is essential for mouse fetal development

Restoration of norepinephrine and reversal of phenotypes in mice lacking dopamine beta-hydroxylase

The neural basis of addiction: a pathology of motivation and choice

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175-187

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10.1124/MI.9.4.6

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19720750

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