The selective dopamine β-hydroxylase inhibitor nepicastat attenuates multiple aspects of cocaine-seeking behavior - Wikidata - awgldk - TriplyDB

The selective dopamine β-hydroxylase inhibitor nepicastat attenuates multiple aspects of cocaine-seeking behavior

The selective dopamine β-hydroxylase inhibitor nepicastat attenuates multiple aspects of cocaine-seeking behavior

http://www.wikidata.org/entity/Q36775245

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Elevated Norepinephrine may be a Unifying Etiological Factor in the Abuse of a Broad Range of Substances: Alcohol, Nicotine, Marijuana, Heroin, Cocaine, and Caffeine The Involvement of Norepinephrine in Behaviors Related to Psychostimulant Addiction Role of cues and contexts on drug-seeking behaviour The crystal structure of human dopamine β-hydroxylase at 2.9 Å resolution The form of a conditioned stimulus can influence the degree to which it acquires incentive motivational properties Stress-Induced Reinstatement of Drug Seeking: 20 Years of Progress.Effects of pharmacologic dopamine β-hydroxylase inhibition on cocaine-induced reinstatement and dopamine neurochemistry in squirrel monkeys.The galanin receptor agonist, galnon, attenuates cocaine-induced reinstatement and dopamine overflow in the frontal cortex.Chronic loss of noradrenergic tone produces β-arrestin2-mediated cocaine hypersensitivity and alters cellular D2 responses in the nucleus accumbens The effects of post-extinction exercise on cocaine-primed and stress-induced reinstatement of cocaine seeking in rats Assessment of safety, cardiovascular and subjective effects after intravenous cocaine and lofexidine.Evaluation of the dopamine β-hydroxylase (DβH) inhibitor nepicastat in participants who meet criteria for cocaine use disorder.Pain and suicidality: insights from reward and addiction neuroscience.Regulation of neurological and neuropsychiatric phenotypes by locus coeruleus-derived galanin.Specificity and impact of adrenergic projections to the midbrain dopamine system.Norepinephrine at the nexus of arousal, motivation and relapse Cue-evoked cocaine "craving": role of dopamine in the accumbens core.Dopamine β-hydroxylase inhibitors enhance the discriminative stimulus effects of cocaine in rats.Cocaine self-administration and extinction alter medullary noradrenergic and limbic forebrain cFos responses to acute, noncontingent cocaine injections in adult rats.The role of the neurokinin-1 receptor in stress-induced reinstatement of alcohol and cocaine seeking Adrenaline rush: the role of adrenergic receptors in stimulant-induced behaviors Drugs currently in Phase II clinical trials for cocaine addiction.Designing modulators of 5-hydroxytryptamine signaling to treat abuse disorders.Emerging drugs for the treatment of cocaine use disorder: a review of neurobiological targets and pharmacotherapy.Norepinephrine regulates cocaine-primed reinstatement via α1-adrenergic receptors in the medial prefrontal cortex.Regional influence of cocaine on evoked dopamine release in the nucleus accumbens core: A role for the caudal brainstem.Selective inhibition of dopamine-beta-hydroxylase enhances dopamine release from noradrenergic terminals in the medial prefrontal cortex.The α-1 adrenoceptor (ADRA1A) genotype moderates the magnitude of acute cocaine-induced subjective effects in cocaine-dependent individuals.α1b-Adrenergic Receptor Localization and Relationship to the D1-Dopamine Receptor in the Rat Nucleus Accumbens.Effects of disulfiram on choice behavior in a rodent gambling task: association with catecholamine levels.The dopamine β-hydroxylase inhibitor, nepicastat, reduces different alcohol-related behaviors in rats.Elevated dopamine in the medial prefrontal cortex suppresses cocaine seeking via D1 receptor overstimulation.Modeling cocaine relapse in rodents: Behavioral considerations and circuit mechanisms.Cocaine-like discriminative stimulus effects of "norepinephrine-preferring" monoamine releasers: time course and interaction studies in rhesus monkeys.Noradrenergic signaling in the VTA modulates cocaine craving.Adolescent cannabinoid exposure induces irritability-like behavior and cocaine cross-sensitization without affecting the escalation of cocaine self-administration in adulthood Anti-stress neuropharmacological mechanisms and targets for addiction treatment: A translational framework

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P2860

The selective dopamine β-hydroxylase inhibitor nepicastat attenuates multiple aspects of cocaine-seeking behavior

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David Weinshenker

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Jason P Schroeder

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S Alisha Epps

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Taylor W Grice

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P2860

Structural insight of dopamine β-hydroxylase, a drug target for complex traits, and functional significance of exonic single nucleotide polymorphisms

Catecholamine modulatory effects of nepicastat (RS-25560-197), a novel, potent and selective inhibitor of dopamine-beta-hydroxylase

The reinstatement model of drug relapse: history, methodology and major findings

α(2) Adrenergic and imidazoline receptor agonists prevent cue-induced cocaine seeking.

Imaging stress- and cue-induced drug and alcohol craving: association with relapse and clinical implications.

mechanisms of disulfiram-induced cocaine abstinence: antabuse and cocaine relapse

Anti-relapse medications: preclinical models for drug addiction treatment.

Disulfiram attenuates drug-primed reinstatement of cocaine seeking via inhibition of dopamine β-hydroxylase.

Randomized, double blind, placebo-controlled trial of disulfiram for the treatment of cocaine dependence in methadone-stabilized patients.

There and back again: a tale of norepinephrine and drug addiction.

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