Stimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infection
about
IFI16 is an innate immune sensor for intracellular DNAcPLA2 regulates the expression of type I interferons and intracellular immunity to Chlamydia trachomatisSTING: infection, inflammation and cancerSensors of Infection: Viral Nucleic Acid PRRs in FishInnate immune responses to Chlamydia pneumoniae infection: role of TLRs, NLRs, and the inflammasomeCytoplasmic DNA innate immune pathwaysSalmonella Suppresses the TRIF-Dependent Type I Interferon Response in Macrophages.Type I Interferon Induction by Neisseria gonorrhoeae: Dual Requirement of Cyclic GMP-AMP Synthase and Toll-like Receptor 4.Borrelia burgdorferi RNA induces type I and III interferons via Toll-like receptor 7 and contributes to production of NF-κB-dependent cytokinesTranscriptome analysis indicates an enhanced activation of adaptive and innate immunity by chlamydia-infected murine epithelial cells treated with interferon γ.Exosomes maintain cellular homeostasis by excreting harmful DNA from cells.Enhanced virulence of Chlamydia muridarum respiratory infections in the absence of TLR2 activation.MyD88 and STING signaling pathways are required for IRF3-mediated IFN-β induction in response to Brucella abortus infectionAbsent in melanoma 2 is required for innate immune recognition of Francisella tularensis.Rac1 regulates the NLRP3 inflammasome which mediates IL-1beta production in Chlamydophila pneumoniae infected human mononuclear cells.Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.5,6-Dimethylxanthenone-4-acetic acid (DMXAA) activates stimulator of interferon gene (STING)-dependent innate immune pathways and is regulated by mitochondrial membrane potentialSTING/MPYS mediates host defense against Listeria monocytogenes infection by regulating Ly6C(hi) monocyte migration.The DNA sensor, cyclic GMP-AMP synthase, is essential for induction of IFN-β during Chlamydia trachomatis infection.MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resolSignificant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infectionAnalyses of the pathways involved in early- and late-phase induction of IFN-beta during C. muridarum infection of oviduct epithelial cellsInterferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infectionVariants in toll-like receptor 1 and 4 genes are associated with Chlamydia trachomatis among women with pelvic inflammatory diseaseNOD1 in contrast to NOD2 functional polymorphism influence Chlamydia trachomatis infection and the risk of tubal factor infertility.The Chlamydia trachomatis Inclusion Membrane Protein CpoS Counteracts STING-Mediated Cellular Surveillance and Suicide Programs.Role of STAT1 in Chlamydia-Induced Type-1 Interferon Production in Oviduct Epithelial Cells.Detection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.CD11b/CD18 (Mac-1) is a novel surface receptor for extracellular double-stranded RNA to mediate cellular inflammatory responsesModeling the transcriptome of genital tract epithelial cells and macrophages in healthy mucosa versus mucosa inflamed by Chlamydia muridarum infection.Demonstration of innate immune responses in the thyroid gland: potential to sense danger and a possible trigger for autoimmune reactions.Chlamydial intracellular survival strategies.Refractoriness of interferon-beta signaling through NOD1 pathway in mouse respiratory epithelial cells using the anticancer xanthone compoundSTING-dependent recognition of cyclic di-AMP mediates type I interferon responses during Chlamydia trachomatis infection.Binding of bacterial secondary messenger molecule c di-GMP is a STING operationInduction of type I interferons by bacteria.The Emerging Roles of STING in Bacterial Infections.Sensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.The role of NOD1 and NOD2 in host defense against chlamydial infection.Induction and function of IFNβ during viral and bacterial infection.
P2860
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P2860
Stimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infection
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@ast
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@en
type
label
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@ast
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@en
prefLabel
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@ast
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@en
P2093
P2860
P356
P1476
Stimulator of IFN gene is crit ...... Chlamydia muridarum infection
@en
P2093
Daniel Prantner
Toni Darville
Uma M Nagarajan
P2860
P304
P356
10.4049/JIMMUNOL.0903704
P407
P577
2010-01-27T00:00:00Z