Senescent cells as a source of inflammatory factors for tumor progression. - Wikidata - awgldk - TriplyDB

Senescent cells as a source of inflammatory factors for tumor progression.

Senescent cells as a source of inflammatory factors for tumor progression.

http://www.wikidata.org/entity/Q33833881

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Androgen deprivation-induced senescence promotes outgrowth of androgen-refractory prostate cancer cells Senescence in Human Mesenchymal Stem Cells: Functional Changes and Implications in Stem Cell-Based Therapy Evaluating biomarkers to model cancer risk post cosmic ray exposure The Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival?Role of Natural Radiosensitizers and Cancer Cell Radioresistance: An Update Therapeutic Implications for Overcoming Radiation Resistance in Cancer Therapy Parainflammation, chronic inflammation, and age-related macular degeneration The Impact of Immunosenescence on Pulmonary Disease The quiescent cellular state is Arf/p53-dependent and associated with H2AX downregulation and genome stability Transcriptional integration of mitochondrial biogenesis Therapeutic targeting of replicative immortality Modulation of inflammation by autophagy: Consequences for human disease Autophagy in thyroid cancer: present knowledge and future perspectives Race, Ethnicity, Psychosocial Factors, and Telomere Length in a Multicenter Setting Defining the toxicology of aging Telomeres and telomerase in head and neck squamous cell carcinoma: from pathogenesis to clinical implications.Changes in the expression of the Toll-like receptor system in the aging rat kidneys Subtelomeric CTCF and cohesin binding site organization using improved subtelomere assemblies and a novel annotation pipeline.Simple biologically informed inflammatory index of two serum cytokines predicts 10 year all-cause mortality in older adults.Oncogenic senescence: a multi-functional perspective.Host mediated inflammatory influence on glioblastoma multiforme recurrence following high-dose ionizing radiation.Gene polymorphisms of cellular senescence marker p21 and disease progression in non-alcohol-related fatty liver disease Deep sequencing reveals novel microRNAs and regulation of microRNA expression during cell senescence.Palbociclib: an evidence-based review of its potential in the treatment of breast cancer Exploiting tumor cell senescence in anticancer therapy Glucocorticoids suppress selected components of the senescence-associated secretory phenotype.Aging, cellular senescence, and cancer Long-distance inflammatory and genotoxic impact of cancer in vivo.Astrocyte senescence as a component of Alzheimer's disease.Tackling the tumor microenvironment: what challenge does it pose to anticancer therapies?Ageing induced vascular smooth muscle cell senescence in atherosclerosis.Oxidative stress damage as a detrimental factor in preterm birth pathology Simvastatin suppresses breast cancer cell proliferation induced by senescent cells Reciprocal interaction between carcinoma-associated fibroblasts and squamous carcinoma cells through interleukin-1α induces cancer progression PinX1 the tail on the chromosome Senescent cardiac fibroblast is critical for cardiac fibrosis after myocardial infarction.p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype.Epithelial cell senescence impairs repair process and exacerbates inflammation after airway injury.A p53/ARF-dependent anticancer barrier activates senescence and blocks tumorigenesis without impacting apoptosis Increased levels of circulating interleukin 6, interleukin 8, C-reactive protein, and risk of lung cancer

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Senescent cells as a source of inflammatory factors for tumor progression.

http://www.wikidata.org/entity/Q33833881

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2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Senescent cells as a source of inflammatory factors for tumor progression.

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Senescent cells as a source of inflammatory factors for tumor progression.

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type

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Senescent cells as a source of inflammatory factors for tumor progression.

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Senescent cells as a source of inflammatory factors for tumor progression.

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Senescent cells as a source of inflammatory factors for tumor progression.

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Senescent cells as a source of inflammatory factors for tumor progression.

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Cancer and Metastasis Reviews

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Senescent cells as a source of inflammatory factors for tumor progression.

@en

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Albert R Davalos

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Jean-Philippe Coppe

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Judith Campisi

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Pierre-Yves Desprez

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P2860

Oncogene-Induced Senescence: Putting the Brakes on Tumor Development

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

PRAK is essential for ras-induced senescence and tumor suppression

High mobility group protein-1 (HMG-1) is a unique activator of p53

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Senescence of activated stellate cells limits liver fibrosis

Remodeling of chromatin structure in senescent cells and its potential impact on tumor suppression and aging

MicroRNAs miR-146a/b negatively modulate the senescence-associated inflammatory mediators IL-6 and IL-8

NF-kappaB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses

Epithelial-mesenchymal transitions in tumour progression

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