Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium. - Wikidata - awgldk - TriplyDB

Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.

Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.

http://www.wikidata.org/entity/Q33835721

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ZIP8 expression in human proximal tubule cells, human urothelial cells transformed by Cd+2 and As+3 and in specimens of normal human urothelium and urothelial cancer Association of global DNA methylation and global DNA hydroxymethylation with metals and other exposures in human blood DNA samples A panel study of occupational exposure to fine particulate matter and changes in DNA methylation over a single workday and years worked in boilermaker welders The control of histone methylation and gene expression by oxidative stress, hypoxia, and metals Elucidating the mechanisms of nickel compound uptake: a review of particulate and nano-nickel endocytosis and toxicity Disruptive chemicals, senescence and immortality Perturbations in nucleosome structure from heavy metal association Influence of toxicologically relevant metals on human epigenetic regulation Inhibition of 2-oxoglutarate dependent oxygenases Quantitative Mass Spectrometry Reveals Changes in Histone H2B Variants as Cells Undergo Inorganic Arsenic-Mediated Cellular Transformation Non-renal effects and the risk assessment of environmental cadmium exposure Global DNA methylation loss associated with mercury contamination and aging in the American alligator (Alligator mississippiensis)Environmental epigenetics and its implication on disease risk and health outcomes Toxicological responses of environmental mixtures: Environmental metal mixtures display synergistic induction of metal-responsive and oxidative stress genes in placental cells Cadmium induces carcinogenesis in BEAS-2B cells through ROS-dependent activation of PI3K/AKT/GSK-3β/β-catenin signaling The role of cadmium and nickel in estrogen receptor signaling and breast cancer: metalloestrogens or not?Inorganic Arsenic-induced cellular transformation is coupled with genome wide changes in chromatin structure, transcriptome and splicing patterns Cadmium induces histone H3 lysine methylation by inhibiting histone demethylase activity Differential DNA methylation in umbilical cord blood of infants exposed to mercury and arsenic in utero Bioavailability, intracellular mobilization of nickel, and HIF-1α activation in human lung epithelial cells exposed to metallic nickel and nickel oxide nanoparticles Assessment of infantile mineral imbalances in autism spectrum disorders (ASDs)Oxidative stress, epigenetics, and cancer stem cells in arsenic carcinogenesis and prevention Hydroxymethylation and metals: a potential epigenetic marker for effects of toxic exposures The preferential accumulation of heavy metals in different tissues following frequent respiratory exposure to PM2.5 in rats Maternal cadmium levels during pregnancy associated with lower birth weight in infants in a North Carolina cohort The content of cadmium, cobalt and nickel in laryngeal carcinoma Akt activation is responsible for enhanced migratory and invasive behavior of arsenic-transformed human bronchial epithelial cells Gene 33/Mig6 inhibits hexavalent chromium-induced DNA damage and cell transformation in human lung epithelial cells Heritability and preliminary genome-wide linkage analysis of arsenic metabolites in urine Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge ahead Differential methylation of the arsenic (III) methyltransferase promoter according to arsenic exposure Environmental chemical exposures and human epigenetics Arsenic induces polyadenylation of canonical histone mRNA by down-regulating stem-loop-binding protein gene expression Basic mechanics of DNA methylation and the unique landscape of the DNA methylome in metal-induced carcinogenesis Low levels of Cd induce persisting epigenetic modifications and acclimation mechanisms in the earthworm Lumbricus terrestris.Core-shell silica nanoparticles synthesized for quantitative study of DNA cleavage by laser-induced fluorescence microscopy.10th NTES Conference: Nickel and Arsenic Compounds Alter the Epigenome of Peripheral Blood Mononuclear Cells.Nickel and epigenetic gene silencing.Nucleosome structural studies.Short placental telomere was associated with cadmium pollution in an electronic waste recycling town in China.

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P2860

Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.

http://www.wikidata.org/entity/Q33835721

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2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009年の論文

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Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.

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Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.

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Epigenetics in metal carcinogenesis: nickel, arsenic, chromium and cadmium.

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Adriana Arita

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Max Costa

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P2860

Molecular mechanisms of arsenic carcinogenesis

The Polycomb group protein EZH2 directly controls DNA methylation

A silencing pathway to induce H3-K9 and H4-K20 trimethylation at constitutive heterochromatin

Chromate-induced epimutations in mammalian cells

Cockatiel-induced hypersensitivity pneumonitis.

Methylation of histone H3 lysine 9 creates a binding site for HP1 proteins

The language of covalent histone modifications

Loss of acetylation at Lys16 and trimethylation at Lys20 of histone H4 is a common hallmark of human cancer

Genetic and epigenetic mechanisms in metal carcinogenesis and cocarcinogenesis: nickel, arsenic, and chromium

Inhibition of core histones acetylation by carcinogenic nickel(II)

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10.1039/B903049B

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