Systems analysis of transcriptome and proteome in retinoic acid/arsenic trioxide-induced cell differentiation/apoptosis of promyelocytic leukemia.
about
Systems medicine: the future of medical genomics and healthcareFrom an old remedy to a magic bullet: molecular mechanisms underlying the therapeutic effects of arsenic in fighting leukemiaIntegrated network analysis of transcriptomic and proteomic data in psoriasisFolate system correlations in DNA microarray data.Transcriptomic response to differentiation induction.Acute promyelocytic leukemia: where did we start, where are we now, and the futureProtein transamidation by transglutaminase 2 in cells: a disputed Ca2+-dependent action of a multifunctional proteinA systems biology understanding of the synergistic effects of arsenic sulfide and Imatinib in BCR/ABL-associated leukemiaFactors determining sensitivity and resistance of tumor cells to arsenic trioxideInhibition of the interactions between eosinophil cationic protein and airway epithelial cells by traditional Chinese herbsA decision theory paradigm for evaluating identifier mapping and filtering methods using data integration.3Omics: a web-based systems biology tool for analysis, integration and visualization of human transcriptomic, proteomic and metabolomic data.Differentiation therapy of leukemia: 3 decades of development.Molecular mechanisms underlying the cholesterol-lowering effect of Ginkgo biloba extract in hepatocytes: a comparative study with lovastatinCuring all patients with acute promyelocytic leukemia: are we there yet?Arsenic trioxide cooperates with all trans retinoic acid to enhance mitogen-activated protein kinase activation and differentiation in PML-RARalpha negative human myeloblastic leukemia cells.Quantitative proteomic analysis reveals the perturbation of multiple cellular pathways in HL-60 cells induced by arsenite treatment.BIMEL is a key effector molecule in oxidative stress-mediated apoptosis in acute myeloid leukemia cells when combined with arsenic trioxide and buthionine sulfoximineImprinted genes and the environment: links to the toxic metals arsenic, cadmium, lead and mercury.Arsenic(III) species inhibit oxidative protein folding in vitro.ATO/ATRA/anthracycline-chemotherapy sequential consolidation achieves long-term efficacy in primary acute promyelocytic leukemia.Leukaemogenesis: more than mutant genesGene expression profile alone is inadequate in predicting complete response in multiple myeloma.Arsenite induces premature senescence via p53/p21 pathway as a result of DNA damage in human malignant glioblastoma cells.Arsenic trioxide, a potent inhibitor of NF-kappaB, abrogates allergen-induced airway hyperresponsiveness and inflammation.Retinoic acid regulation of eye and testis-specific transcripts within a complex locusRetinoid regulation of antiviral innate immunity in hepatocytes.Addition of Arsenic Trioxide into Induction Regimens Could Not Accelerate Recovery of Abnormality of Coagulation and Fibrinolysis in Patients with Acute Promyelocytic LeukemiaMutations of Epigenetic Modifier Genes as a Poor Prognostic Factor in Acute Promyelocytic Leukemia Under Treatment With All-Trans Retinoic Acid and Arsenic TrioxideA proteasome for all occasions.Acute promyelocytic leukemia: a model of molecular target based therapy.Transcriptional repression of CDKN2D by PML/RARα contributes to the altered proliferation and differentiation block of acute promyelocytic leukemia cells.Systematic identification of arsenic-binding proteins reveals that hexokinase-2 is inhibited by arsenic.Emerging new approaches for the treatment of acute promyelocytic leukemia.Proteasomal adaptation to environmental stress links resistance to proteotoxicity with longevity in Caenorhabditis elegansPerspectives of proteomics in acute myeloid leukemia.Treatment of acute promyelocytic leukaemia with all-trans retinoic acid and arsenic trioxide: a paradigm of synergistic molecular targeting therapy.From molecular interaction to acute promyelocytic leukemia: Calculating leukemogenesis and remission from endogenous molecular-cellular network.Metabolomics coupled with proteomics advancing drug discovery toward more agile development of targeted combination therapies.Transcriptomic Analysis Reveals Adaptive Responses of an Enterobacteriaceae Strain LSJC7 to Arsenic Exposure.
P2860
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P2860
Systems analysis of transcriptome and proteome in retinoic acid/arsenic trioxide-induced cell differentiation/apoptosis of promyelocytic leukemia.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@ast
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@en
type
label
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@ast
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@en
prefLabel
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@ast
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@en
P2093
P2860
P356
P1476
Systems analysis of transcript ...... sis of promyelocytic leukemia.
@en
P2093
Chun-Jun Zhao
Da-Kai Xiao
Eric Eveno
Hui-Yong Fan
Kan-Kan Wang
Pei-Zheng Zheng
Qing-Hua Zhang
Qiu-Hua Huang
P2860
P304
P356
10.1073/PNAS.0502825102
P407
P577
2005-05-13T00:00:00Z