Expression profiles of endoplasmic reticulum stress-related molecules in demyelinating lesions and multiple sclerosis.
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Remyelination Therapy in Multiple SclerosisThe unfolded protein response in multiple sclerosisEndoplasmic reticulum stress in hepatic steatosis and inflammatory bowel diseasesFree cysteine modulates the conformation of human C/EBP homologous proteinThe Cannabinoid CB1/CB2 Agonist WIN55212.2 Promotes Oligodendrocyte Differentiation In Vitro and Neuroprotection During the Cuprizone-Induced Central Nervous System Demyelination.Endoplasmic reticulum stress and inflammation in the central nervous systemPathogenic implications of iron accumulation in multiple sclerosis.PERK-dependent activation of JAK1 and STAT3 contributes to endoplasmic reticulum stress-induced inflammation.Pharmaceutical integrated stress response enhancement protects oligodendrocytes and provides a potential multiple sclerosis therapeuticAspirin and multiple sclerosisER Chaperone BiP/GRP78 Is Required for Myelinating Cell Survival and Provides Protection during Experimental Autoimmune Encephalomyelitis.Cellular stress and innate inflammation in organ-specific autoimmunity: lessons learned from vitiligo.Harnessing the integrated stress response for the treatment of multiple sclerosis.The evidence for hypoperfusion as a factor in multiple sclerosis lesion development.Oligodendrocyte-specific activation of PERK signaling protects mice against experimental autoimmune encephalomyelitis.Calreticulin and other components of endoplasmic reticulum stress in rat and human inflammatory demyelination.PERK activation preserves the viability and function of remyelinating oligodendrocytes in immune-mediated demyelinating diseasesPotential For Cell-mediated Immune Responses In Mouse Models Of Pelizaeus-Merzbacher Disease.Endoplasmic reticulum stress and the unfolded protein response in disorders of myelinating gliaAttempts to Overcome Remyelination Failure: Toward Opening New Therapeutic Avenues for Multiple Sclerosis.Genetic inactivation of PERK signaling in mouse oligodendrocytes: normal developmental myelination with increased susceptibility to inflammatory demyelination.Endoplasmic Reticulum Malfunction in the Nervous System.Rab32 connects ER stress to mitochondrial defects in multiple sclerosis.Role of the unfolded protein response in the development of central nervous system.Manipulation of Oxygen and Endoplasmic Reticulum Stress Factors as Possible Interventions for Treatment of Multiple Sclerosis: Evidence for and Against.Increased expression of ER stress- and hypoxia-associated molecules in grey matter lesions in multiple sclerosis.
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P2860
Expression profiles of endoplasmic reticulum stress-related molecules in demyelinating lesions and multiple sclerosis.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Expression profiles of endopla ...... esions and multiple sclerosis.
@ast
Expression profiles of endopla ...... esions and multiple sclerosis.
@en
type
label
Expression profiles of endopla ...... esions and multiple sclerosis.
@ast
Expression profiles of endopla ...... esions and multiple sclerosis.
@en
prefLabel
Expression profiles of endopla ...... esions and multiple sclerosis.
@ast
Expression profiles of endopla ...... esions and multiple sclerosis.
@en
P2093
P2860
P356
P1476
Expression profiles of endopla ...... lesions and multiple sclerosis
@en
P2093
Aoife Ní Mháille
Michael Farrell
Paula Cunnea
Stephen McQuaid
P2860
P304
P356
10.1177/1352458511399114
P577
2011-03-07T00:00:00Z