Endoplasmic reticulum stress in disorders of myelinating cells.
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Metabolic acetate therapy improves phenotype in the tremor rat model of Canavan diseaseThe unfolded protein response in multiple sclerosisNeuronal endoplasmic reticulum stress in axon injury and neurodegenerationFunctional Role of the Disulfide Isomerase ERp57 in Axonal RegenerationN-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylationInhibition of GADD34, the stress-inducible regulatory subunit of the endoplasmic reticulum stress response, does not enhance functional recovery after spinal cord injuryDevelopmental splicing deregulation in leukodystrophies related to EIF2B mutationsNeuroimaging evidence of deficient axon myelination in Wolfram syndrome.Neural stem cell engraftment and myelination in the human brain.Early effects of a high-caloric diet and physical exercise on brain volumetry and behavior: a combined MRI and histology study in mice.Differential deployment of REST and CoREST promotes glial subtype specification and oligodendrocyte lineage maturation.Cytotoxic aggregation and amyloid formation by the myostatin precursor proteinPolymorphic cis- and trans-regulation of human gene expression.Expression profiles of endoplasmic reticulum stress-related molecules in demyelinating lesions and multiple sclerosis.Transgenic models for cytokine-induced neurological disease.Autophagy promotes oligodendrocyte survival and function following dysmyelination in a long-lived myelin mutantInterferon-γ inhibits central nervous system myelination through both STAT1-dependent and STAT1-independent pathwaysChemical stress induces the unfolded protein response in olfactory sensory neurons.Impaired eukaryotic translation initiation factor 2B activity specifically in oligodendrocytes reproduces the pathology of vanishing white matter disease in mice.Early brain vulnerability in Wolfram syndrome.NGLY1 mutation causes neuromotor impairment, intellectual disability, and neuropathy.Oligodendrocyte progenitors reversibly exit the cell cycle and give rise to astrocytes in response to interferon-γ.Pharmaceutical integrated stress response enhancement protects oligodendrocytes and provides a potential multiple sclerosis therapeuticImpaired eIF2B activity in ligodendrocytes contributes to VWMD pathogenesis.Transgenic neuronal overexpression reveals that stringently regulated p23 expression is critical for coordinated movement in miceDepletion of tissue factor suppresses hepatic metastasis and tumor growth in colorectal cancer via the downregulation of MMPs and the induction of autophagy and apoptosisModeling the natural history of Pelizaeus-Merzbacher disease.Making the White Matter Matters: Progress in Understanding Canavan's Disease and Therapeutic Interventions Through Eight Decades.cFLIP is critical for oligodendrocyte protection from inflammationParadoxical Sensitivity to an Integrated Stress Response Blocking Mutation in Vanishing White Matter Cells.ER Chaperone BiP/GRP78 Is Required for Myelinating Cell Survival and Provides Protection during Experimental Autoimmune Encephalomyelitis.Mutation of sec63 in zebrafish causes defects in myelinated axons and liver pathologyActivation of the unfolded protein response promotes axonal regeneration after peripheral nerve injuryAberrant Schwann cell lipid metabolism linked to mitochondrial deficits leads to axon degeneration and neuropathy.Resetting translational homeostasis restores myelination in Charcot-Marie-Tooth disease type 1B miceA modified UPR stress sensing system reveals a novel tissue distribution of IRE1/XBP1 activity during normal Drosophila development.Perk Ablation Ameliorates Myelination in S63del-Charcot-Marie-Tooth 1B Neuropathy.Oligodendrocyte-specific activation of PERK signaling protects mice against experimental autoimmune encephalomyelitis.rAAV Gene Therapy in a Canavan's Disease Mouse Model Reveals Immune Impairments and an Extended Pathology Beyond the Central Nervous System.Regulation of PERK-eIF2α signalling by tuberous sclerosis complex-1 controls homoeostasis and survival of myelinating oligodendrocytes
P2860
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P2860
Endoplasmic reticulum stress in disorders of myelinating cells.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մարտին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Endoplasmic reticulum stress in disorders of myelinating cells.
@ast
Endoplasmic reticulum stress in disorders of myelinating cells.
@en
Endoplasmic reticulum stress in disorders of myelinating cells.
@nl
type
label
Endoplasmic reticulum stress in disorders of myelinating cells.
@ast
Endoplasmic reticulum stress in disorders of myelinating cells.
@en
Endoplasmic reticulum stress in disorders of myelinating cells.
@nl
prefLabel
Endoplasmic reticulum stress in disorders of myelinating cells.
@ast
Endoplasmic reticulum stress in disorders of myelinating cells.
@en
Endoplasmic reticulum stress in disorders of myelinating cells.
@nl
P2860
P356
P1433
P1476
Endoplasmic reticulum stress in disorders of myelinating cells
@en
P2093
Brian Popko
Wensheng Lin
P2860
P2888
P304
P356
10.1038/NN.2273
P407
P577
2009-03-15T00:00:00Z